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1
ACVIM consensus statement guidelines for the classification, diagnosis, and management of cardiomyopathies in cats
ACVIM consensus statement guidelines for the classification, diagnosis, and management of cardiomyopathies in cats
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ACVIM consensus statement guidelines for the classification, diagnosis, and management of cardiomyopathies in cats

Journal of veterinary internal medicine, 2020-05, Vol.34 (3), p.1062-1077 [Peer Reviewed Journal]

2020 The Authors. published by Wiley Periodicals, Inc. on behalf of the American College of Veterinary Internal Medicine. ;2020 The Authors. Journal of Veterinary Internal Medicine published by Wiley Periodicals, Inc. on behalf of the American College of Veterinary Internal Medicine. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0891-6640 ;ISSN: 1939-1676 ;EISSN: 1939-1676 ;DOI: 10.1111/jvim.15745 ;PMID: 32243654

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2
Speckle tracking echocardiographic deformation indices in Chagas and idiopathic dilated cardiomyopathy: Incremental prognostic value of longitudinal strain
Speckle tracking echocardiographic deformation indices in Chagas and idiopathic dilated cardiomyopathy: Incremental prognostic value of longitudinal strain
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Speckle tracking echocardiographic deformation indices in Chagas and idiopathic dilated cardiomyopathy: Incremental prognostic value of longitudinal strain

PloS one, 2019-08, Vol.14 (8), p.e0221028-e0221028 [Peer Reviewed Journal]

COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Santos Junior et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Santos Junior et al 2019 Santos Junior et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0221028 ;PMID: 31437176

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3
Alcohol use disorders and the heart
Alcohol use disorders and the heart
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Alcohol use disorders and the heart

Addiction (Abingdon, England), 2019-09, Vol.114 (9), p.1670-1678 [Peer Reviewed Journal]

2019 The Authors. published by John Wiley & Sons Ltd on behalf of Society for the Study of Addiction ;2019 The Authors. Addiction published by John Wiley & Sons Ltd on behalf of Society for the Study of Addiction. ;2019 Society for the Study of Addiction ;ISSN: 0965-2140 ;EISSN: 1360-0443 ;DOI: 10.1111/add.14703 ;PMID: 31309639

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4
Effects of troponin T cardiomyopathy mutations on the calcium sensitivity of the regulated thin filament and the actomyosin cross-bridge kinetics of human β-cardiac myosin
Effects of troponin T cardiomyopathy mutations on the calcium sensitivity of the regulated thin filament and the actomyosin cross-bridge kinetics of human β-cardiac myosin
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Effects of troponin T cardiomyopathy mutations on the calcium sensitivity of the regulated thin filament and the actomyosin cross-bridge kinetics of human β-cardiac myosin

PloS one, 2013-12, Vol.8 (12), p.e83403-e83403 [Peer Reviewed Journal]

2013 Sommese et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Sommese et al 2013 Sommese et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0083403 ;PMID: 24367593

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5
Accurate Classification of Non-ischemic Cardiomyopathy
Accurate Classification of Non-ischemic Cardiomyopathy
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Accurate Classification of Non-ischemic Cardiomyopathy

Current cardiology reports, 2023-10, Vol.25 (10), p.1299-1317 [Peer Reviewed Journal]

The Author(s) 2023 ;2023. The Author(s). ;ISSN: 1523-3782 ;EISSN: 1534-3170 ;DOI: 10.1007/s11886-023-01944-0 ;PMID: 37721634

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6
The clinical profile, genetic basis and survival of childhood cardiomyopathy: a single-center retrospective study
The clinical profile, genetic basis and survival of childhood cardiomyopathy: a single-center retrospective study
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The clinical profile, genetic basis and survival of childhood cardiomyopathy: a single-center retrospective study

European journal of pediatrics, 2024-03, Vol.183 (3), p.1389-1401 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1432-1076 ;ISSN: 0340-6199 ;EISSN: 1432-1076 ;DOI: 10.1007/s00431-023-05358-6 ;PMID: 38165464

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7
Cardiac magnetic resonance and galectin-3 level as predictors of prognostic outcomes for non-ischemic cardiomyopathy patients
Cardiac magnetic resonance and galectin-3 level as predictors of prognostic outcomes for non-ischemic cardiomyopathy patients
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Cardiac magnetic resonance and galectin-3 level as predictors of prognostic outcomes for non-ischemic cardiomyopathy patients

International Journal of Cardiovascular Imaging, 2016-12, Vol.32 (12), p.1725-1733 [Peer Reviewed Journal]

Springer Science+Business Media Dordrecht 2016 ;ISSN: 1569-5794 ;EISSN: 1573-0743 ;EISSN: 1875-8312 ;DOI: 10.1007/s10554-016-0958-1 ;PMID: 27566192 ;CODEN: IJCIBI

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8
Molecular mechanisms of diabetic cardiomyopathy
Molecular mechanisms of diabetic cardiomyopathy
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Molecular mechanisms of diabetic cardiomyopathy

Diabetologia, 2014-04, Vol.57 (4), p.660-671 [Peer Reviewed Journal]

Springer-Verlag Berlin Heidelberg 2014 ;2015 INIST-CNRS ;ISSN: 0012-186X ;EISSN: 1432-0428 ;DOI: 10.1007/s00125-014-3171-6 ;PMID: 24477973

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9
Diabetic cardiomyopathy: molecular mechanisms, detrimental effects of conventional treatment, and beneficial effects of natural therapy
Diabetic cardiomyopathy: molecular mechanisms, detrimental effects of conventional treatment, and beneficial effects of natural therapy
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Diabetic cardiomyopathy: molecular mechanisms, detrimental effects of conventional treatment, and beneficial effects of natural therapy

Heart failure reviews, 2019-03, Vol.24 (2), p.279-299 [Peer Reviewed Journal]

Springer Science+Business Media, LLC, part of Springer Nature 2018 ;Heart Failure Reviews is a copyright of Springer, (2018). All Rights Reserved. ;ISSN: 1382-4147 ;EISSN: 1573-7322 ;DOI: 10.1007/s10741-018-9749-1 ;PMID: 30349977

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10
Moving Beyond the Sarcomere to Explain Heterogeneity in Hypertrophic Cardiomyopathy: JACC Review Topic of the Week
Moving Beyond the Sarcomere to Explain Heterogeneity in Hypertrophic Cardiomyopathy: JACC Review Topic of the Week
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Moving Beyond the Sarcomere to Explain Heterogeneity in Hypertrophic Cardiomyopathy: JACC Review Topic of the Week

Journal of the American College of Cardiology, 2019-04, Vol.73 (15), p.1978-1986 [Peer Reviewed Journal]

Copyright © 2019 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;2019. American College of Cardiology Foundation ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2019.01.061 ;PMID: 31000001

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11
Sexual dimorphisms of mRNA and miRNA in human/murine heart disease
Sexual dimorphisms of mRNA and miRNA in human/murine heart disease
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Sexual dimorphisms of mRNA and miRNA in human/murine heart disease

PloS one, 2017-07, Vol.12 (7), p.e0177988-e0177988 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Tsuji et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Tsuji et al 2017 Tsuji et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0177988 ;PMID: 28704447

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12
Plasminogen activator inhibitor-1 reduces cardiac fibrosis and promotes M2 macrophage polarization in inflammatory cardiomyopathy
Plasminogen activator inhibitor-1 reduces cardiac fibrosis and promotes M2 macrophage polarization in inflammatory cardiomyopathy
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Plasminogen activator inhibitor-1 reduces cardiac fibrosis and promotes M2 macrophage polarization in inflammatory cardiomyopathy

Basic research in cardiology, 2021-12, Vol.116 (1), p.1-1, Article 1 [Peer Reviewed Journal]

The Author(s) 2021 ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0300-8428 ;EISSN: 1435-1803 ;DOI: 10.1007/s00395-020-00840-w ;PMID: 33432417

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13
The administration of high-mobility group box 1 fragment prevents deterioration of cardiac performance by enhancement of bone marrow mesenchymal stem cell homing in the delta-sarcoglycan-deficient hamster
The administration of high-mobility group box 1 fragment prevents deterioration of cardiac performance by enhancement of bone marrow mesenchymal stem cell homing in the delta-sarcoglycan-deficient hamster
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The administration of high-mobility group box 1 fragment prevents deterioration of cardiac performance by enhancement of bone marrow mesenchymal stem cell homing in the delta-sarcoglycan-deficient hamster

PloS one, 2018-12, Vol.13 (12), p.e0202838-e0202838 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Kido et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Kido et al 2018 Kido et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0202838 ;PMID: 30517097

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14
Cirrhotic Cardiomyopathy
Cirrhotic Cardiomyopathy
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Cirrhotic Cardiomyopathy

Current gastroenterology reports, 2020-09, Vol.22 (9), p.45-45, Article 45 [Peer Reviewed Journal]

Springer Science+Business Media, LLC, part of Springer Nature 2020 ;Springer Science+Business Media, LLC, part of Springer Nature 2020. ;ISSN: 1522-8037 ;EISSN: 1534-312X ;DOI: 10.1007/s11894-020-00783-1 ;PMID: 32651721

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15
Enhanced heart failure, mortality and renin activation in female mice with experimental dilated cardiomyopathy
Enhanced heart failure, mortality and renin activation in female mice with experimental dilated cardiomyopathy
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Enhanced heart failure, mortality and renin activation in female mice with experimental dilated cardiomyopathy

PloS one, 2017-12, Vol.12 (12), p.e0189315-e0189315 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Tripathi et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Tripathi et al 2017 Tripathi et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0189315 ;PMID: 29240788

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16
SGLT-2 Inhibition with Dapagliflozin Reduces the Activation of the Nlrp3/ASC Inflammasome and Attenuates the Development of Diabetic Cardiomyopathy in Mice with Type 2 Diabetes. Further Augmentation of the Effects with Saxagliptin, a DPP4 Inhibitor
SGLT-2 Inhibition with Dapagliflozin Reduces the Activation of the Nlrp3/ASC Inflammasome and Attenuates the Development of Diabetic Cardiomyopathy in Mice with Type 2 Diabetes. Further Augmentation of the Effects with Saxagliptin, a DPP4 Inhibitor
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SGLT-2 Inhibition with Dapagliflozin Reduces the Activation of the Nlrp3/ASC Inflammasome and Attenuates the Development of Diabetic Cardiomyopathy in Mice with Type 2 Diabetes. Further Augmentation of the Effects with Saxagliptin, a DPP4 Inhibitor

Cardiovascular drugs and therapy, 2017-04, Vol.31 (2), p.119-132 [Peer Reviewed Journal]

Springer Science+Business Media New York 2017 ;Cardiovascular Drugs and Therapy is a copyright of Springer, 2017. ;ISSN: 0920-3206 ;EISSN: 1573-7241 ;DOI: 10.1007/s10557-017-6725-2 ;PMID: 28447181

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17
Dilated cardiomyopathy in the era of precision medicine: latest concepts and developments
Dilated cardiomyopathy in the era of precision medicine: latest concepts and developments
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Dilated cardiomyopathy in the era of precision medicine: latest concepts and developments

Heart failure reviews, 2022-07, Vol.27 (4), p.1173-1191 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature 2021 ;2021. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature. ;The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature 2021. ;ISSN: 1382-4147 ;EISSN: 1573-7322 ;DOI: 10.1007/s10741-021-10139-0 ;PMID: 34263412

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18
Gap junction protein beta 4 plays an important role in cardiac function in humans, rodents, and zebrafish
Gap junction protein beta 4 plays an important role in cardiac function in humans, rodents, and zebrafish
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Gap junction protein beta 4 plays an important role in cardiac function in humans, rodents, and zebrafish

PloS one, 2020-10, Vol.15 (10), p.e0240129 [Peer Reviewed Journal]

COPYRIGHT 2020 Public Library of Science ;COPYRIGHT 2020 Public Library of Science ;2020 Okamoto et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Okamoto et al 2020 Okamoto et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0240129 ;PMID: 33048975

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19
Exome-wide association study reveals novel susceptibility genes to sporadic dilated cardiomyopathy
Exome-wide association study reveals novel susceptibility genes to sporadic dilated cardiomyopathy
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Exome-wide association study reveals novel susceptibility genes to sporadic dilated cardiomyopathy

PloS one, 2017-03, Vol.12 (3), p.e0172995 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;This is an open access article, free of all copyright, and may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose. The work is made available under the Creative Commons CC0 public domain dedication: https://creativecommons.org/publicdomain/zero/1.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0172995 ;PMID: 28296976

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20
A Murine Hypertrophic Cardiomyopathy Model: The DBA/2J Strain
A Murine Hypertrophic Cardiomyopathy Model: The DBA/2J Strain
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A Murine Hypertrophic Cardiomyopathy Model: The DBA/2J Strain

PloS one, 2015-08, Vol.10 (8), p.e0133132-e0133132 [Peer Reviewed Journal]

COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Zhao et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Zhao et al 2015 Zhao et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0133132 ;PMID: 26241864

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