skip to main content
Language:
Search Limited to: Search Limited to: Resource type Show Results with: Show Results with: Search type Index

Results 1 - 20 of 358  for All Library Resources

Results 1 2 3 4 5 next page
Refined by: Database: Nature Open Access remove subject: Heart remove
Result Number Material Type Add to My Shelf Action Record Details and Options
1
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling
Material Type:
Article
Add to My Research

Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling

Scientific reports, 2018-01, Vol.8 (1), p.1998-11, Article 1998 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-20114-9 ;PMID: 29386531

Full text available

2
Silencing long non-coding RNA Kcnq1ot1 alleviates pyroptosis and fibrosis in diabetic cardiomyopathy
Material Type:
Article
Add to My Research

Silencing long non-coding RNA Kcnq1ot1 alleviates pyroptosis and fibrosis in diabetic cardiomyopathy

Cell death & disease, 2018-09, Vol.9 (10), p.1000-13, Article 1000 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-018-1029-4 ;PMID: 30250027

Full text available

3
Analysis of cardiac magnetic resonance imaging in 36,000 individuals yields genetic insights into dilated cardiomyopathy
Material Type:
Article
Add to My Research

Analysis of cardiac magnetic resonance imaging in 36,000 individuals yields genetic insights into dilated cardiomyopathy

Nature communications, 2020-05, Vol.11 (1), p.2254-2254, Article 2254 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-15823-7 ;PMID: 32382064

Full text available

4
Intergenerational inheritance of high fat diet-induced cardiac lipotoxicity in Drosophila
Material Type:
Article
Add to My Research

Intergenerational inheritance of high fat diet-induced cardiac lipotoxicity in Drosophila

Nature communications, 2019-01, Vol.10 (1), p.193-14, Article 193 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-08128-3 ;PMID: 30643137

Full text available

5
Biallelic loss of LDB3 leads to a lethal pediatric dilated cardiomyopathy
Material Type:
Article
Add to My Research

Biallelic loss of LDB3 leads to a lethal pediatric dilated cardiomyopathy

European journal of human genetics : EJHG, 2023-01, Vol.31 (1), p.97-104 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 1018-4813 ;EISSN: 1476-5438 ;DOI: 10.1038/s41431-022-01204-9 ;PMID: 36253531

Full text available

6
High glucose induces Drp1-mediated mitochondrial fission via the Orai1 calcium channel to participate in diabetic cardiomyocyte hypertrophy
Material Type:
Article
Add to My Research

High glucose induces Drp1-mediated mitochondrial fission via the Orai1 calcium channel to participate in diabetic cardiomyocyte hypertrophy

Cell death & disease, 2021-02, Vol.12 (2), p.216-216, Article 216 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-021-03502-4 ;PMID: 33637715

Full text available

7
Correction of human phospholamban R14del mutation associated with cardiomyopathy using targeted nucleases and combination therapy
Material Type:
Article
Add to My Research

Correction of human phospholamban R14del mutation associated with cardiomyopathy using targeted nucleases and combination therapy

Nature communications, 2015-04, Vol.6 (1), p.6955, Article 6955 [Peer Reviewed Journal]

Copyright Nature Publishing Group Apr 2015 ;Attribution ;Copyright © 2015, Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. 2015 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms7955 ;PMID: 25923014

Full text available

8
Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes
Material Type:
Article
Add to My Research

Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes

Scientific reports, 2017-11, Vol.7 (1), p.14829-11, Article 14829 [Peer Reviewed Journal]

Scientific Reports is a copyright of Springer, 2017. ;2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-13675-8 ;PMID: 29093449

Full text available

9
Identification of key immune-related genes in dilated cardiomyopathy using bioinformatics analysis
Material Type:
Article
Add to My Research

Identification of key immune-related genes in dilated cardiomyopathy using bioinformatics analysis

Scientific reports, 2023-02, Vol.13 (1), p.1820-1820, Article 1820 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. corrected publication 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-26277-w ;PMID: 36725968

Full text available

10
lncRNA MIAT functions as a competing endogenous RNA to upregulate DAPK2 by sponging miR-22-3p in diabetic cardiomyopathy
Material Type:
Article
Add to My Research

lncRNA MIAT functions as a competing endogenous RNA to upregulate DAPK2 by sponging miR-22-3p in diabetic cardiomyopathy

Cell death & disease, 2017-07, Vol.8 (7), p.e2929-e2929 [Peer Reviewed Journal]

Copyright Nature Publishing Group Jul 2017 ;Copyright © 2017 The Author(s) 2017 The Author(s) ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/cddis.2017.321 ;PMID: 28703801

Full text available

11
Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy
Material Type:
Article
Add to My Research

Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy

Scientific reports, 2022-12, Vol.12 (1), p.22501-22501, Article 22501 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-26889-2 ;PMID: 36577774

Full text available

12
The mechanosensitive Piezo1 channel mediates heart mechano-chemo transduction
Material Type:
Article
Add to My Research

The mechanosensitive Piezo1 channel mediates heart mechano-chemo transduction

Nature communications, 2021-02, Vol.12 (1), p.869-14, Article 869 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-21178-4 ;PMID: 33558521

Full text available

13
Retrospective analysis of clinical phenotype and prognosis of hypertrophic cardiomyopathy complicated with hypertension
Material Type:
Article
Add to My Research

Retrospective analysis of clinical phenotype and prognosis of hypertrophic cardiomyopathy complicated with hypertension

Scientific reports, 2020-01, Vol.10 (1), p.349-349, Article 349 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-57230-z ;PMID: 31941943

Full text available

14
NDUFAB1 confers cardio-protection by enhancing mitochondrial bioenergetics through coordination of respiratory complex and supercomplex assembly
Material Type:
Article
Add to My Research

NDUFAB1 confers cardio-protection by enhancing mitochondrial bioenergetics through coordination of respiratory complex and supercomplex assembly

Cell research, 2019-09, Vol.29 (9), p.754-766 [Peer Reviewed Journal]

2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 1001-0602 ;EISSN: 1748-7838 ;DOI: 10.1038/s41422-019-0208-x ;PMID: 31366990

Full text available

15
Metabolic stress-induced cardiomyopathy is caused by mitochondrial dysfunction due to attenuated Erk5 signaling
Material Type:
Article
Add to My Research

Metabolic stress-induced cardiomyopathy is caused by mitochondrial dysfunction due to attenuated Erk5 signaling

Nature communications, 2017-09, Vol.8 (1), p.494-16, Article 494 [Peer Reviewed Journal]

2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2017 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-017-00664-8 ;PMID: 28887535

Full text available

16
Cytoskeletal disarray increases arrhythmogenic vulnerability during sympathetic stimulation in a model of hypertrophic cardiomyopathy
Material Type:
Article
Add to My Research

Cytoskeletal disarray increases arrhythmogenic vulnerability during sympathetic stimulation in a model of hypertrophic cardiomyopathy

Scientific reports, 2023-07, Vol.13 (1), p.11296-11296, Article 11296 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-38296-2 ;PMID: 37438479

Full text available

17
Caveolinopathies: from the biology of caveolin-3 to human diseases
Material Type:
Article
Add to My Research

Caveolinopathies: from the biology of caveolin-3 to human diseases

European journal of human genetics : EJHG, 2010-02, Vol.18 (2), p.137-145 [Peer Reviewed Journal]

2015 INIST-CNRS ;Copyright Nature Publishing Group Feb 2010 ;Copyright © 2010 Macmillan Publishers Limited 2010 Macmillan Publishers Limited ;ISSN: 1018-4813 ;EISSN: 1476-5438 ;DOI: 10.1038/ejhg.2009.103 ;PMID: 19584897

Full text available

18
Thrombospondin-3 augments injury-induced cardiomyopathy by intracellular integrin inhibition and sarcolemmal instability
Material Type:
Article
Add to My Research

Thrombospondin-3 augments injury-induced cardiomyopathy by intracellular integrin inhibition and sarcolemmal instability

Nature communications, 2019-01, Vol.10 (1), p.76-76, Article 76 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-08026-8 ;PMID: 30622267

Full text available

19
Individualized interactomes for network-based precision medicine in hypertrophic cardiomyopathy with implications for other clinical pathophenotypes
Material Type:
Article
Add to My Research

Individualized interactomes for network-based precision medicine in hypertrophic cardiomyopathy with implications for other clinical pathophenotypes

Nature communications, 2021-02, Vol.12 (1), p.873-873, Article 873 [Peer Reviewed Journal]

This is a U.S. government work and not under copyright protection in the U.S.; foreign copyright protection may apply 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;This is a U.S. government work and not under copyright protection in the U.S.; foreign copyright protection may apply 2021. corrected publication 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;This is a U.S. government work and not under copyright protection in the U.S.; foreign copyright protection may apply 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-21146-y ;PMID: 33558530

Full text available

20
Restoring the impaired cardiac calcium homeostasis and cardiac function in iron overload rats by the combined deferiprone and N-acetyl cysteine
Material Type:
Article
Add to My Research

Restoring the impaired cardiac calcium homeostasis and cardiac function in iron overload rats by the combined deferiprone and N-acetyl cysteine

Scientific reports, 2017-03, Vol.7 (1), p.44460-44460, Article 44460 [Peer Reviewed Journal]

Copyright Nature Publishing Group Mar 2017 ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep44460 ;PMID: 28287621

Full text available

Results 1 - 20 of 358  for All Library Resources

Results 1 2 3 4 5 next page

Personalize your results

  1. Edit

Refine Search Results

Expand My Results

  1.   

Refine My Results

Creation Date 

From To
  1. Before 1997  (8)
  2. 1997 To 2003  (29)
  3. 2004 To 2010  (43)
  4. 2011 To 2018  (132)
  5. After 2018  (147)
  6. More options open sub menu

Language 

  1. Japanese  (60)
  2. Norwegian  (1)
  3. More options open sub menu

Searching Remote Databases, Please Wait