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1
CRIMSON: An open-source software framework for cardiovascular integrated modelling and simulation
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CRIMSON: An open-source software framework for cardiovascular integrated modelling and simulation

PLoS computational biology, 2021-05, Vol.17 (5), p.e1008881-e1008881 [Peer Reviewed Journal]

COPYRIGHT 2021 Public Library of Science ;2021 Arthurs et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 Arthurs et al 2021 Arthurs et al ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1008881 ;PMID: 33970900

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2
Predictive model identifies key network regulators of cardiomyocyte mechano-signaling
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Predictive model identifies key network regulators of cardiomyocyte mechano-signaling

PLoS computational biology, 2017-11, Vol.13 (11), p.e1005854-e1005854 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Tan PM, Buchholz KS, Omens JH, McCulloch AD, Saucerman JJ (2017) Predictive model identifies key network regulators of cardiomyocyte mechano-signaling. PLoS Comput Biol 13(11): e1005854. https://doi.org/10.1371/journal.pcbi.1005854 ;2017 Tan et al 2017 Tan et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Tan PM, Buchholz KS, Omens JH, McCulloch AD, Saucerman JJ (2017) Predictive model identifies key network regulators of cardiomyocyte mechano-signaling. PLoS Comput Biol 13(11): e1005854. https://doi.org/10.1371/journal.pcbi.1005854 ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1005854 ;PMID: 29131824

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3
A method to quantify mechanobiologic forces during zebrafish cardiac development using 4-D light sheet imaging and computational modeling
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A method to quantify mechanobiologic forces during zebrafish cardiac development using 4-D light sheet imaging and computational modeling

PLoS computational biology, 2017-10, Vol.13 (10), p.e1005828-e1005828 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Vedula V, Lee J, Xu H, Kuo C-CJ, Hsiai TK, Marsden AL (2017) A method to quantify mechanobiologic forces during zebrafish cardiac development using 4-D light sheet imaging and computational modeling. PLoS Comput Biol13(10): e1005828. https://doi.org/10.1371/journal.pcbi.1005828 ;2017 Vedula et al 2017 Vedula et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Vedula V, Lee J, Xu H, Kuo C-CJ, Hsiai TK, Marsden AL (2017) A method to quantify mechanobiologic forces during zebrafish cardiac development using 4-D light sheet imaging and computational modeling. PLoS Comput Biol13(10): e1005828. https://doi.org/10.1371/journal.pcbi.1005828 ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1005828 ;PMID: 29084212

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4
Self-organization of conducting pathways explains electrical wave propagation in cardiac tissues with high fraction of non-conducting cells
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Self-organization of conducting pathways explains electrical wave propagation in cardiac tissues with high fraction of non-conducting cells

PLoS computational biology, 2019-03, Vol.15 (3), p.e1006597-e1006597 [Peer Reviewed Journal]

COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Kudryashova et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Kudryashova et al 2019 Kudryashova et al ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1006597 ;PMID: 30883540

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5
Measurable health effects associated with the daylight saving time shift
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Measurable health effects associated with the daylight saving time shift

PLoS computational biology, 2020-06, Vol.16 (6), p.e1007927-e1007927 [Peer Reviewed Journal]

COPYRIGHT 2020 Public Library of Science ;COPYRIGHT 2020 Public Library of Science ;2020 Zhang et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Zhang et al 2020 Zhang et al ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1007927 ;PMID: 32511231

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6
Use of Angiotensin-Converting Enzyme Inhibitors and Angiotensin II Receptor Blockers During the COVID-19 Pandemic: A Modeling Analysis
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Use of Angiotensin-Converting Enzyme Inhibitors and Angiotensin II Receptor Blockers During the COVID-19 Pandemic: A Modeling Analysis

PLoS computational biology, 2020-10, Vol.16 (10), p.e1008235-e1008235 [Peer Reviewed Journal]

COPYRIGHT 2020 Public Library of Science ;COPYRIGHT 2020 Public Library of Science ;2020 Sadria, Layton. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Sadria, Layton 2020 Sadria, Layton ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1008235 ;PMID: 33031368

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7
Allosteric modulation of cardiac myosin dynamics by omecamtiv mecarbil
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Allosteric modulation of cardiac myosin dynamics by omecamtiv mecarbil

PLoS computational biology, 2017-11, Vol.13 (11), p.e1005826-e1005826 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Hashem S, Tiberti M, Fornili A (2017) Allosteric modulation of cardiac myosin dynamics by omecamtiv mecarbil. PLoS Comput Biol 13(11): e1005826. https://doi.org/10.1371/journal.pcbi.1005826 ;2017 Hashem et al 2017 Hashem et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Hashem S, Tiberti M, Fornili A (2017) Allosteric modulation of cardiac myosin dynamics by omecamtiv mecarbil. PLoS Comput Biol 13(11): e1005826. https://doi.org/10.1371/journal.pcbi.1005826 ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1005826 ;PMID: 29108014

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8
Estimating the probabilities of rare arrhythmic events in multiscale computational models of cardiac cells and tissue
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Estimating the probabilities of rare arrhythmic events in multiscale computational models of cardiac cells and tissue

PLoS computational biology, 2017-11, Vol.13 (11), p.e1005783-e1005783 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Walker MA, Gurev V, Rice JJ, Greenstein JL, Winslow RL (2017) Estimating the probabilities of rare arrhythmic events in multiscale computational models of cardiac cells and tissue. PLoS Comput Biol 13(11): e1005783. https://doi.org/10.1371/journal.pcbi.1005783 ;2017 Walker et al 2017 Walker et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Walker MA, Gurev V, Rice JJ, Greenstein JL, Winslow RL (2017) Estimating the probabilities of rare arrhythmic events in multiscale computational models of cardiac cells and tissue. PLoS Comput Biol 13(11): e1005783. https://doi.org/10.1371/journal.pcbi.1005783 ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1005783 ;PMID: 29145393

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9
Stabilization of diastolic calcium signal via calcium pump regulation of complex local calcium releases and transient decay in a computational model of cardiac pacemaker cell with individual release channels
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Stabilization of diastolic calcium signal via calcium pump regulation of complex local calcium releases and transient decay in a computational model of cardiac pacemaker cell with individual release channels

PLoS computational biology, 2017-08, Vol.13 (8), p.e1005675-e1005675 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Maltsev AV, Maltsev VA, Stern MD (2017) Stabilization of diastolic calcium signal via calcium pump regulation of complex local calcium releases and transient decay in a computational model of cardiac pacemaker cell with individual release channels. PLoS Comput Biol 13(8): e1005675. https://doi.org/10.1371/journal.pcbi.1005675 ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Maltsev AV, Maltsev VA, Stern MD (2017) Stabilization of diastolic calcium signal via calcium pump regulation of complex local calcium releases and transient decay in a computational model of cardiac pacemaker cell with individual release channels. PLoS Comput Biol 13(8): e1005675. https://doi.org/10.1371/journal.pcbi.1005675 ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1005675 ;PMID: 28792496

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10
In silico study of multicellular automaticity of heterogeneous cardiac cell monolayers: Effects of automaticity strength and structural linear anisotropy
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In silico study of multicellular automaticity of heterogeneous cardiac cell monolayers: Effects of automaticity strength and structural linear anisotropy

PLoS computational biology, 2018-03, Vol.14 (3), p.e1005978-e1005978 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Duverger JE, Jacquemet V, Vinet A, Comtois P (2018) In silico study of multicellular automaticity of heterogeneous cardiac cell monolayers: Effects of automaticity strength and structural linear anisotropy. PLoS Comput Biol 14(3): e1005978. https://doi.org/10.1371/journal.pcbi.1005978 ;2018 Duverger et al 2018 Duverger et al ;2018 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Duverger JE, Jacquemet V, Vinet A, Comtois P (2018) In silico study of multicellular automaticity of heterogeneous cardiac cell monolayers: Effects of automaticity strength and structural linear anisotropy. PLoS Comput Biol 14(3): e1005978. https://doi.org/10.1371/journal.pcbi.1005978 ;ISSN: 1553-7358 ;ISSN: 1553-734X ;EISSN: 1553-7358 ;DOI: 10.1371/journal.pcbi.1005978 ;PMID: 29529023

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