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1
Folic acid reduces doxorubicin‐induced cardiomyopathy by modulating endothelial nitric oxide synthase
Folic acid reduces doxorubicin‐induced cardiomyopathy by modulating endothelial nitric oxide synthase
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Folic acid reduces doxorubicin‐induced cardiomyopathy by modulating endothelial nitric oxide synthase

Journal of cellular and molecular medicine, 2017-12, Vol.21 (12), p.3277-3287 [Peer Reviewed Journal]

2017 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.13231 ;PMID: 28608983

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2
Pre-eclampsia and peripartum cardiomyopathy in molar pregnancy: clinical implication for maternally imprinted genes
Pre-eclampsia and peripartum cardiomyopathy in molar pregnancy: clinical implication for maternally imprinted genes
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Pre-eclampsia and peripartum cardiomyopathy in molar pregnancy: clinical implication for maternally imprinted genes

Ultrasound in obstetrics & gynecology, 2004-04, Vol.23 (4), p.398 [Peer Reviewed Journal]

Copyright 2004 ISUOG. ;ISSN: 0960-7692 ;EISSN: 1469-0705 ;DOI: 10.1002/uog.1015 ;PMID: 15065193

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3
Hereditary transthyretin amyloidosis in mainland China: a unicentric retrospective study
Hereditary transthyretin amyloidosis in mainland China: a unicentric retrospective study
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Hereditary transthyretin amyloidosis in mainland China: a unicentric retrospective study

Annals of clinical and translational neurology, 2021-04, Vol.8 (4), p.831-841 [Peer Reviewed Journal]

2021 The Authors. published by Wiley Periodicals LLC on behalf of American Neurological Association ;2021 The Authors. Annals of Clinical and Translational Neurology published by Wiley Periodicals LLC on behalf of American Neurological Association. ;2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2328-9503 ;EISSN: 2328-9503 ;DOI: 10.1002/acn3.51328 ;PMID: 33739616

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4
Hypertrophic cardiomyopathy: New approaches and a time to reappraise older approaches
Hypertrophic cardiomyopathy: New approaches and a time to reappraise older approaches
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Hypertrophic cardiomyopathy: New approaches and a time to reappraise older approaches

The Journal of thoracic and cardiovascular surgery, 2016-10, Vol.152 (4), p.983 [Peer Reviewed Journal]

EISSN: 1097-685X ;DOI: 10.1016/j.jtcvs.2016.06.034 ;PMID: 27474178

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5
ALMS1-deficient fibroblasts over-express extra-cellular matrix components, display cell cycle delay and are resistant to apoptosis
ALMS1-deficient fibroblasts over-express extra-cellular matrix components, display cell cycle delay and are resistant to apoptosis
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ALMS1-deficient fibroblasts over-express extra-cellular matrix components, display cell cycle delay and are resistant to apoptosis

PloS one, 2011-04, Vol.6 (4), p.e19081-e19081 [Peer Reviewed Journal]

COPYRIGHT 2011 Public Library of Science ;COPYRIGHT 2011 Public Library of Science ;2011 Zulato et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Zulato et al. 2011 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0019081 ;PMID: 21541333

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6
Diabetes modulation of the myocardial infarction-acute kidney injury axis
Diabetes modulation of the myocardial infarction-acute kidney injury axis
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Diabetes modulation of the myocardial infarction-acute kidney injury axis

American journal of physiology. Heart and circulatory physiology, 2022-03, Vol.322 (3), p.H394-H405 [Peer Reviewed Journal]

Copyright American Physiological Society Mar 2022 ;ISSN: 0363-6135 ;EISSN: 1522-1539 ;DOI: 10.1152/ajpheart.00639.2021 ;PMID: 35089809

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7
Disarray of sarcomeric alpha-actinin in cardiomyocytes infected by Trypanosoma cruzi
Disarray of sarcomeric alpha-actinin in cardiomyocytes infected by Trypanosoma cruzi
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Disarray of sarcomeric alpha-actinin in cardiomyocytes infected by Trypanosoma cruzi

Parasitology, 2006-08, Vol.133 (2), p.171-178 [Peer Reviewed Journal]

2006 Cambridge University Press ;2006 INIST-CNRS ;Copyright Cambridge University Press Aug 2006 ;ISSN: 0031-1820 ;EISSN: 1469-8161 ;DOI: 10.1017/S0031182006000011 ;PMID: 16650336 ;CODEN: PARAAE

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8
Cardiac Involvement of Female Carrier of Duchenne Muscular Dystrophy
Cardiac Involvement of Female Carrier of Duchenne Muscular Dystrophy
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Cardiac Involvement of Female Carrier of Duchenne Muscular Dystrophy

Internal Medicine, 2000, Vol.39(1), pp.2-3 [Peer Reviewed Journal]

The Japanese Society of Internal Medicine ;ISSN: 0918-2918 ;EISSN: 1349-7235 ;DOI: 10.2169/internalmedicine.39.2 ;PMID: 10674837

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