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1
BMAL1 regulates mitochondrial fission and mitophagy through mitochondrial protein BNIP3 and is critical in the development of dilated cardiomyopathy
BMAL1 regulates mitochondrial fission and mitophagy through mitochondrial protein BNIP3 and is critical in the development of dilated cardiomyopathy
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BMAL1 regulates mitochondrial fission and mitophagy through mitochondrial protein BNIP3 and is critical in the development of dilated cardiomyopathy

Protein & cell, 2020-09, Vol.11 (9), p.661-679 [Peer Reviewed Journal]

Copyright reserved, 2020, The Author(s) 2020 ;The Author(s) 2020 ;The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1674-800X ;EISSN: 1674-8018 ;DOI: 10.1007/s13238-020-00713-x ;PMID: 32277346

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2
Isogenic human pluripotent stem cell disease models reveal ABRA deficiency underlies cTnT mutation-induced familial dilated cardiomyopathy
Isogenic human pluripotent stem cell disease models reveal ABRA deficiency underlies cTnT mutation-induced familial dilated cardiomyopathy
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Isogenic human pluripotent stem cell disease models reveal ABRA deficiency underlies cTnT mutation-induced familial dilated cardiomyopathy

Protein & cell, 2022-01, Vol.13 (1), p.65-71 [Peer Reviewed Journal]

Copyright reserved, 2021, The Author(s) ;The Author(s) 2021 ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1674-800X ;EISSN: 1674-8018 ;DOI: 10.1007/s13238-021-00843-w ;PMID: 33884582

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3
RNA binding protein 24 deletion disrupts global alternative splicing and causes dilated cardiomyopathy
RNA binding protein 24 deletion disrupts global alternative splicing and causes dilated cardiomyopathy
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RNA binding protein 24 deletion disrupts global alternative splicing and causes dilated cardiomyopathy

Protein & cell, 2019-06, Vol.10 (6), p.405-416 [Peer Reviewed Journal]

Copyright reserved, 2018, The Author(s) ;The Author(s) 2018 ;Protein & Cell is a copyright of Springer, (2018). All Rights Reserved. © 2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1674-800X ;EISSN: 1674-8018 ;DOI: 10.1007/s13238-018-0578-8 ;PMID: 30267374

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4
Screening for mutations in human cardiomyopathy- is RBM24 a new but rare disease gene?
Screening for mutations in human cardiomyopathy- is RBM24 a new but rare disease gene?
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Screening for mutations in human cardiomyopathy- is RBM24 a new but rare disease gene?

Protein & cell, 2019-06, Vol.10 (6), p.393-394 [Peer Reviewed Journal]

Copyright reserved, 2018, The Author(s) ;The Author(s) 2018 ;Protein & Cell is a copyright of Springer, (2018). All Rights Reserved. © 2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1674-800X ;EISSN: 1674-8018 ;DOI: 10.1007/s13238-018-0590-z ;PMID: 30421357

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5
Propofol and salvianolic acid A synergistically attenuated cardiac ischemia-reperfusion injury in diabetic mice via modulating the CD36/AMPK pathway
Propofol and salvianolic acid A synergistically attenuated cardiac ischemia-reperfusion injury in diabetic mice via modulating the CD36/AMPK pathway
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Propofol and salvianolic acid A synergistically attenuated cardiac ischemia-reperfusion injury in diabetic mice via modulating the CD36/AMPK pathway

Burns and trauma, 2024-01, Vol.12, p.tkad055-tkad055 [Peer Reviewed Journal]

The Author(s) 2024. Published by Oxford University Press. ;The Author(s) 2024. Published by Oxford University Press. 2024 ;ISSN: 2321-3868 ;EISSN: 2321-3876 ;DOI: 10.1093/burnst/tkad055 ;PMID: 38601971

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6
HSP70 alleviates sepsis-induced cardiomyopathy by attenuating mitochondrial dysfunction-initiated NLRP3 inflammasome-mediated pyroptosis in cardiomyocytes
HSP70 alleviates sepsis-induced cardiomyopathy by attenuating mitochondrial dysfunction-initiated NLRP3 inflammasome-mediated pyroptosis in cardiomyocytes
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HSP70 alleviates sepsis-induced cardiomyopathy by attenuating mitochondrial dysfunction-initiated NLRP3 inflammasome-mediated pyroptosis in cardiomyocytes

Burns and trauma, 2022-01, Vol.10, p.tkac043-tkac043 [Peer Reviewed Journal]

ISSN: 2321-3876 ;ISSN: 2321-3868 ;EISSN: 2321-3876 ;DOI: 10.1093/burnst/tkac043

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7
TMEM43-S358L mutation enhances NF-κB-TGFβ signal cascade in arrhythmogenic right ventricular dysplasia/cardiomyopathy
TMEM43-S358L mutation enhances NF-κB-TGFβ signal cascade in arrhythmogenic right ventricular dysplasia/cardiomyopathy
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TMEM43-S358L mutation enhances NF-κB-TGFβ signal cascade in arrhythmogenic right ventricular dysplasia/cardiomyopathy

Protein & cell, 2019-02, Vol.10 (2), p.104-119 [Peer Reviewed Journal]

The Author(s) 2018 ;Protein & Cell is a copyright of Springer, (2018). All Rights Reserved. © 2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1674-800X ;EISSN: 1674-8018 ;DOI: 10.1007/s13238-018-0563-2 ;PMID: 29980933

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8
Cardiac proteomics reveals the potential mechanism of microtubule associated protein 4 phosphorylation-induced mitochondrial dysfunction
Cardiac proteomics reveals the potential mechanism of microtubule associated protein 4 phosphorylation-induced mitochondrial dysfunction
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Cardiac proteomics reveals the potential mechanism of microtubule associated protein 4 phosphorylation-induced mitochondrial dysfunction

Burns and trauma, 2019, Vol.7 (1), p.8-8 [Peer Reviewed Journal]

Copyright © 2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2321-3868 ;ISSN: 2321-3876 ;EISSN: 2321-3876 ;DOI: 10.1186/s41038-019-0146-3 ;PMID: 30906793

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9
Secondary hemochromatosis as a result of acute transfusion-induced iron overload in a burn patient
Secondary hemochromatosis as a result of acute transfusion-induced iron overload in a burn patient
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Secondary hemochromatosis as a result of acute transfusion-induced iron overload in a burn patient

Burns and trauma, 2016-12, Vol.4, p.10 [Peer Reviewed Journal]

Copyright BioMed Central 2016 ;The Author(s) 2016 ;ISSN: 2321-3868 ;ISSN: 2321-3876 ;EISSN: 2321-3876 ;DOI: 10.1186/s41038-016-0034-z ;PMID: 27574680

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