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1
Alterations in cardiac DNA methylation in human dilated cardiomyopathy
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Alterations in cardiac DNA methylation in human dilated cardiomyopathy

EMBO molecular medicine, 2013-03, Vol.5 (3), p.413-429 [Peer Reviewed Journal]

Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO ;Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO. ;2013. This work is published under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO 2013 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201201553 ;PMID: 23341106

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2
Antisense‐mediated exon skipping: a therapeutic strategy for titin‐based dilated cardiomyopathy
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Antisense‐mediated exon skipping: a therapeutic strategy for titin‐based dilated cardiomyopathy

EMBO molecular medicine, 2015-05, Vol.7 (5), p.562-576 [Peer Reviewed Journal]

2015 The Authors. Published under the terms of the CC BY 4.0 license ;2015 The Authors. Published under the terms of the CC BY 4.0 license. ;COPYRIGHT 2015 John Wiley & Sons, Inc. ;2015. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 The Authors. Published under the terms of the CC BY 4.0 license 2015 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201505047 ;PMID: 25759365

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3
Disease modeling of a mutation in α‐actinin 2 guides clinical therapy in hypertrophic cardiomyopathy
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Disease modeling of a mutation in α‐actinin 2 guides clinical therapy in hypertrophic cardiomyopathy

EMBO molecular medicine, 2019-12, Vol.11 (12), p.e11115-n/a [Peer Reviewed Journal]

2019 The Authors. Published under the terms of the CC BY 4.0 license ;2019 The Authors. Published under the terms of the CC BY 4.0 license. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201911115 ;PMID: 31680489

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4
Rescue of cardiomyopathy through U7snRNA‐mediated exon skipping in Mybpc3‐targeted knock‐in mice
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Rescue of cardiomyopathy through U7snRNA‐mediated exon skipping in Mybpc3‐targeted knock‐in mice

EMBO molecular medicine, 2013-07, Vol.5 (7), p.1128-1145 [Peer Reviewed Journal]

Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO ;2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO. ;Attribution ;2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO 2013 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201202168 ;PMID: 23716398

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5
Genomic structural variations lead to dysregulation of important coding and non‐coding RNA species in dilated cardiomyopathy
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Genomic structural variations lead to dysregulation of important coding and non‐coding RNA species in dilated cardiomyopathy

EMBO molecular medicine, 2018-01, Vol.10 (1), p.107-120 [Peer Reviewed Journal]

2017 The Authors. Published under the terms of the CC BY 4.0 license ;2017 The Authors. Published under the terms of the CC BY 4.0 license. ;2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201707838 ;PMID: 29138229

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6
Metabolic crosstalk between the heart and liver impacts familial hypertrophic cardiomyopathy
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Metabolic crosstalk between the heart and liver impacts familial hypertrophic cardiomyopathy

EMBO molecular medicine, 2014-04, Vol.6 (4), p.482-495 [Peer Reviewed Journal]

2014 The Authors. ;2014. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. 2014 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201302852 ;PMID: 24567073

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7
PRAS40 prevents development of diabetic cardiomyopathy and improves hepatic insulin sensitivity in obesity
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PRAS40 prevents development of diabetic cardiomyopathy and improves hepatic insulin sensitivity in obesity

EMBO molecular medicine, 2014-01, Vol.6 (1), p.57-65 [Peer Reviewed Journal]

2013 The Authors. ;2014. This work is published under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. 2014 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201303183 ;PMID: 24408966

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8
Alternative oxidase‐mediated respiration prevents lethal mitochondrial cardiomyopathy
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Alternative oxidase‐mediated respiration prevents lethal mitochondrial cardiomyopathy

EMBO molecular medicine, 2019-01, Vol.11 (1), p.n/a [Peer Reviewed Journal]

2018 The Authors. Published under the terms of the CC BY 4.0 license ;2018 The Authors. Published under the terms of the CC BY 4.0 license. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201809456 ;PMID: 30530468

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9
Inhibition of PIKfyve prevents myocardial apoptosis and hypertrophy through activation of SIRT3 in obese mice
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Inhibition of PIKfyve prevents myocardial apoptosis and hypertrophy through activation of SIRT3 in obese mice

EMBO molecular medicine, 2017-06, Vol.9 (6), p.770-785 [Peer Reviewed Journal]

2017 The Authors. Published under the terms of the CC BY 4.0 license ;2017 The Authors. Published under the terms of the CC BY 4.0 license. ;COPYRIGHT 2017 John Wiley & Sons, Inc. ;2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201607096 ;PMID: 28396567

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10
Mitochondrial dynamism and heart disease: changing shape and shaping change
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Mitochondrial dynamism and heart disease: changing shape and shaping change

EMBO molecular medicine, 2015-07, Vol.7 (7), p.865-877 [Peer Reviewed Journal]

2015 The Author. Published under the terms of the CC BY 4.0 license ;2015 The Author. Published under the terms of the CC BY 4.0 license. ;2015. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 The Author. Published under the terms of the CC BY 4.0 license 2015 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201404575 ;PMID: 25861797

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11
CUEDC2 modulates cardiomyocyte oxidative capacity by regulating GPX1 stability
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CUEDC2 modulates cardiomyocyte oxidative capacity by regulating GPX1 stability

EMBO molecular medicine, 2016-07, Vol.8 (7), p.813-829 [Peer Reviewed Journal]

2016 The Authors. Published under the terms of the CC BY 4.0 license ;2016 The Authors. Published under the terms of the CC BY 4.0 license. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201506010 ;PMID: 27286733

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12
Sequence variation in PPP1R13L results in a novel form of cardio‐cutaneous syndrome
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Sequence variation in PPP1R13L results in a novel form of cardio‐cutaneous syndrome

EMBO molecular medicine, 2017-03, Vol.9 (3), p.319-336 [Peer Reviewed Journal]

2017 The Authors. Published under the terms of the CC BY 4.0 license ;2017 The Authors. Published under the terms of the CC BY 4.0 license. ;2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201606523 ;PMID: 28069640

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13
Lysosomal dysfunction and impaired autophagy underlie the pathogenesis of amyloidogenic light chain‐mediated cardiotoxicity
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Lysosomal dysfunction and impaired autophagy underlie the pathogenesis of amyloidogenic light chain‐mediated cardiotoxicity

EMBO molecular medicine, 2014-11, Vol.6 (11), p.1493-1507 [Peer Reviewed Journal]

2014 The Authors. Published under the terms of the CC BY 4.0 license ;2014 The Authors. Published under the terms of the CC BY 4.0 license. ;2014. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. Published under the terms of the CC BY 4.0 license 2014 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201404190 ;PMID: 25319546

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14
VEGF‐B‐induced vascular growth leads to metabolic reprogramming and ischemia resistance in the heart
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VEGF‐B‐induced vascular growth leads to metabolic reprogramming and ischemia resistance in the heart

EMBO molecular medicine, 2014-03, Vol.6 (3), p.307-321 [Peer Reviewed Journal]

2014 The Authors. ;COPYRIGHT 2014 John Wiley & Sons, Inc. ;2014. This work is published under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. 2014 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201303147 ;PMID: 24448490

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15
CRTH2 promotes endoplasmic reticulum stress‐induced cardiomyocyte apoptosis through m‐calpain
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CRTH2 promotes endoplasmic reticulum stress‐induced cardiomyocyte apoptosis through m‐calpain

EMBO molecular medicine, 2018-03, Vol.10 (3), p.n/a [Peer Reviewed Journal]

2018 The Authors. Published under the terms of the CC BY 4.0 license ;2018 The Authors. Published under the terms of the CC BY 4.0 license. ;2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201708237 ;PMID: 29335338

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16
The heart‐liver metabolic axis: defective communication exacerbates disease
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The heart‐liver metabolic axis: defective communication exacerbates disease

EMBO molecular medicine, 2014-04, Vol.6 (4), p.436-438 [Peer Reviewed Journal]

2014 The Authors. Published under the terms of the CC BY license. ;2014. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. Published under the terms of the CC BY license. 2014 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201303800 ;PMID: 24623378

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17
FAT FLUX: enzymes, regulators, and pathophysiology of intracellular lipolysis
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FAT FLUX: enzymes, regulators, and pathophysiology of intracellular lipolysis

EMBO molecular medicine, 2015-04, Vol.7 (4), p.359-362 [Peer Reviewed Journal]

2015 The Author. Published under the terms of the CC BY 4.0 license ;2015. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 The Author. Published under the terms of the CC BY 4.0 license 2015 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201404846 ;PMID: 25604059

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18
TIP30 counteracts cardiac hypertrophy and failure by inhibiting translational elongation
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TIP30 counteracts cardiac hypertrophy and failure by inhibiting translational elongation

EMBO molecular medicine, 2019-10, Vol.11 (10), p.e10018-n/a [Peer Reviewed Journal]

2019 The Authors. Published under the terms of the CC BY 4.0 license ;2019 The Authors. Published under the terms of the CC BY 4.0 license. ;COPYRIGHT 2019 John Wiley & Sons, Inc. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201810018 ;PMID: 31468715

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19
Magnitude of the Potential Screening Gap for Fabry Disease in Manitoba: A Population-Based Retrospective Cohort Study
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Magnitude of the Potential Screening Gap for Fabry Disease in Manitoba: A Population-Based Retrospective Cohort Study

Canadian journal of kidney health and disease, 2023-01, Vol.10, p.20543581231162218-20543581231162218 [Peer Reviewed Journal]

The Author(s) 2023 ;The Author(s) 2023. ;The Author(s) 2023. This work is licensed under the Creative Commons Attribution – Non-Commercial License https://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 2023 Canadian Society of Nephrology, unless otherwise noted. Manuscript content on this site is licensed under Creative Commons Licenses ;ISSN: 2054-3581 ;EISSN: 2054-3581 ;DOI: 10.1177/20543581231162218 ;PMID: 36970566

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20
In search of the next super models
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In search of the next super models

EMBO molecular medicine, 2019-12, Vol.11 (12), p.e11502-n/a [Peer Reviewed Journal]

2019 The Authors. Published under the terms of the CC BY 4.0 license ;2019 The Authors. Published under the terms of the CC BY 4.0 license. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;ISSN: 1757-4684 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201911502 ;PMID: 31736275

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