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1
Alterations in cardiac DNA methylation in human dilated cardiomyopathy
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Alterations in cardiac DNA methylation in human dilated cardiomyopathy

EMBO molecular medicine, 2013-03, Vol.5 (3), p.413-429 [Peer Reviewed Journal]

Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO ;Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO. ;2013. This work is published under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO 2013 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201201553 ;PMID: 23341106

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2
Muscle wasting in young patients with dilated cardiomyopathy
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Muscle wasting in young patients with dilated cardiomyopathy

Journal of cachexia, sarcopenia and muscle, 2017-08, Vol.8 (4), p.542-548 [Peer Reviewed Journal]

2017 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of the Society on Sarcopenia, Cachexia and Wasting Disorders ;2017 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of the Society on Sarcopenia, Cachexia and Wasting Disorders. ;2017. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.12193 ;PMID: 28251827

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3
Antisense‐mediated exon skipping: a therapeutic strategy for titin‐based dilated cardiomyopathy
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Antisense‐mediated exon skipping: a therapeutic strategy for titin‐based dilated cardiomyopathy

EMBO molecular medicine, 2015-05, Vol.7 (5), p.562-576 [Peer Reviewed Journal]

2015 The Authors. Published under the terms of the CC BY 4.0 license ;2015 The Authors. Published under the terms of the CC BY 4.0 license. ;COPYRIGHT 2015 John Wiley & Sons, Inc. ;2015. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 The Authors. Published under the terms of the CC BY 4.0 license 2015 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201505047 ;PMID: 25759365

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4
Disease modeling of a mutation in α‐actinin 2 guides clinical therapy in hypertrophic cardiomyopathy
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Disease modeling of a mutation in α‐actinin 2 guides clinical therapy in hypertrophic cardiomyopathy

EMBO molecular medicine, 2019-12, Vol.11 (12), p.e11115-n/a [Peer Reviewed Journal]

2019 The Authors. Published under the terms of the CC BY 4.0 license ;2019 The Authors. Published under the terms of the CC BY 4.0 license. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201911115 ;PMID: 31680489

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5
Rescue of cardiomyopathy through U7snRNA‐mediated exon skipping in Mybpc3‐targeted knock‐in mice
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Rescue of cardiomyopathy through U7snRNA‐mediated exon skipping in Mybpc3‐targeted knock‐in mice

EMBO molecular medicine, 2013-07, Vol.5 (7), p.1128-1145 [Peer Reviewed Journal]

Copyright © 2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO ;2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO. ;Attribution ;2013 The Authors. Published by John Wiley and Sons, Ltd on behalf of EMBO 2013 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201202168 ;PMID: 23716398

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6
Megestrol acetate improves cardiac function in a model of cancer cachexia‐induced cardiomyopathy by autophagic modulation
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Megestrol acetate improves cardiac function in a model of cancer cachexia‐induced cardiomyopathy by autophagic modulation

Journal of cachexia, sarcopenia and muscle, 2016-12, Vol.7 (5), p.555-566 [Peer Reviewed Journal]

2016 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of the Society of Sarcopenia, Cachexia and Wasting Disorders ;2016. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.12116 ;PMID: 27239419

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7
Reduction in mitochondrial iron alleviates cardiac damage during injury
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Reduction in mitochondrial iron alleviates cardiac damage during injury

EMBO molecular medicine, 2016-03, Vol.8 (3), p.247-267 [Peer Reviewed Journal]

2016 The Authors. Published under the terms of the CC BY 4.0 license ;2016 The Authors. Published under the terms of the CC BY 4.0 license. ;2016. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201505748 ;PMID: 26896449

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8
Incidence of Stress Cardiomyopathy During the Coronavirus Disease 2019 Pandemic
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Incidence of Stress Cardiomyopathy During the Coronavirus Disease 2019 Pandemic

JAMA Network Open, 2020-07, Vol.3 (7), p.e2014780-e2014780 [Peer Reviewed Journal]

2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright 2020 Jabri A et al. . ;ISSN: 2574-3805 ;EISSN: 2574-3805 ;DOI: 10.1001/jamanetworkopen.2020.14780 ;PMID: 32644140

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9
Risk of Chronic Cardiomyopathy Among Patients With the Acute Phase or Indeterminate Form of Chagas Disease: A Systematic Review and Meta-analysis
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Risk of Chronic Cardiomyopathy Among Patients With the Acute Phase or Indeterminate Form of Chagas Disease: A Systematic Review and Meta-analysis

JAMA Network Open, 2020-08, Vol.3 (8), p.e2015072-e2015072 [Peer Reviewed Journal]

2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright 2020 Chadalawada S et al. . ;ISSN: 2574-3805 ;EISSN: 2574-3805 ;DOI: 10.1001/jamanetworkopen.2020.15072 ;PMID: 32865573

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10
Standard Operation Procedure of Percutaneous Endomyocardial Biopsy in Peking Union Medical College Hospital
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Standard Operation Procedure of Percutaneous Endomyocardial Biopsy in Peking Union Medical College Hospital

Cardiology discovery, 2021-09, Vol.1 (3), p.148-153 [Peer Reviewed Journal]

ISSN: 2693-8499 ;EISSN: 2693-8499 ;DOI: 10.1097/CD9.0000000000000023

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11
Inhibition of the NLRP3/IL‐1β axis protects against sepsis‐induced cardiomyopathy
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Inhibition of the NLRP3/IL‐1β axis protects against sepsis‐induced cardiomyopathy

Journal of cachexia, sarcopenia and muscle, 2021-12, Vol.12 (6), p.1653-1668 [Peer Reviewed Journal]

2021 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of Society on Sarcopenia, Cachexia and Wasting Disorders. ;2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.12763 ;PMID: 34472725

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12
Genomic structural variations lead to dysregulation of important coding and non‐coding RNA species in dilated cardiomyopathy
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Genomic structural variations lead to dysregulation of important coding and non‐coding RNA species in dilated cardiomyopathy

EMBO molecular medicine, 2018-01, Vol.10 (1), p.107-120 [Peer Reviewed Journal]

2017 The Authors. Published under the terms of the CC BY 4.0 license ;2017 The Authors. Published under the terms of the CC BY 4.0 license. ;2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201707838 ;PMID: 29138229

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13
Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathy
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Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathy

Journal of cachexia, sarcopenia and muscle, 2024-04, Vol.15 (2), p.536-551 [Peer Reviewed Journal]

2024 The Authors. published by Wiley Periodicals LLC. ;2024 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by Wiley Periodicals LLC. ;2024. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.13411 ;PMID: 38221511

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14
Metabolic crosstalk between the heart and liver impacts familial hypertrophic cardiomyopathy
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Metabolic crosstalk between the heart and liver impacts familial hypertrophic cardiomyopathy

EMBO molecular medicine, 2014-04, Vol.6 (4), p.482-495 [Peer Reviewed Journal]

2014 The Authors. ;2014. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. 2014 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201302852 ;PMID: 24567073

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15
The lncRNA HOTAIR attenuates pyroptosis of diabetic cardiomyocytes by recruiting FUS to regulate SIRT3 expression
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The lncRNA HOTAIR attenuates pyroptosis of diabetic cardiomyocytes by recruiting FUS to regulate SIRT3 expression

The Kaohsiung journal of medical sciences, 2023-05, Vol.39 (5), p.458-467 [Peer Reviewed Journal]

2023 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Kaohsiung Medical University. ;2023 The Authors. The Kaohsiung Journal of Medical Sciences published by John Wiley & Sons Australia, Ltd on behalf of Kaohsiung Medical University. ;COPYRIGHT 2023 John Wiley & Sons, Inc. ;2023. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1607-551X ;EISSN: 2410-8650 ;DOI: 10.1002/kjm2.12676 ;PMID: 37073806

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16
Receptor interacting protein kinase‐3 mediates both myopathy and cardiomyopathy in preclinical animal models of Duchenne muscular dystrophy
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Receptor interacting protein kinase‐3 mediates both myopathy and cardiomyopathy in preclinical animal models of Duchenne muscular dystrophy

Journal of cachexia, sarcopenia and muscle, 2023-12, Vol.14 (6), p.2520-2531 [Peer Reviewed Journal]

2023 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of Society on Sarcopenia, Cachexia and Wasting Disorders. ;2023. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.13265 ;PMID: 37909859

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17
Impairment of the ER/mitochondria compartment in human cardiomyocytes with PLN p.Arg14del mutation
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Impairment of the ER/mitochondria compartment in human cardiomyocytes with PLN p.Arg14del mutation

EMBO molecular medicine, 2021-06, Vol.13 (6), p.e13074-n/a [Peer Reviewed Journal]

2021 The Authors. Published under the terms of the CC BY 4.0 license ;2021 The Authors. Published under the terms of the CC BY 4.0 license. ;2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.202013074 ;PMID: 33998164

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18
Association of Titin-Truncating Genetic Variants With Life-threatening Cardiac Arrhythmias in Patients With Dilated Cardiomyopathy and Implanted Defibrillators
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Association of Titin-Truncating Genetic Variants With Life-threatening Cardiac Arrhythmias in Patients With Dilated Cardiomyopathy and Implanted Defibrillators

JAMA Network Open, 2019-06, Vol.2 (6), p.e196520-e196520 [Peer Reviewed Journal]

2019. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright 2019 Corden B et al. . ;ISSN: 2574-3805 ;EISSN: 2574-3805 ;DOI: 10.1001/jamanetworkopen.2019.6520 ;PMID: 31251381

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19
Cardiac magnetic resonance features and outcomes of patients with non-compaction cardiomyopathy – A retrospective follow-up from Pakistan
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Cardiac magnetic resonance features and outcomes of patients with non-compaction cardiomyopathy – A retrospective follow-up from Pakistan

Annals of medicine and surgery, 2022-08, Vol.80, p.103962-103962, Article 103962 [Peer Reviewed Journal]

2022 ;2022 Published by Elsevier Ltd on behalf of IJS Publishing Group Ltd. 2022 ;ISSN: 2049-0801 ;EISSN: 2049-0801 ;DOI: 10.1016/j.amsu.2022.103962 ;PMID: 35846858

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20
Oxidized LDL‐dependent pathway as new pathogenic trigger in arrhythmogenic cardiomyopathy
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Oxidized LDL‐dependent pathway as new pathogenic trigger in arrhythmogenic cardiomyopathy

EMBO molecular medicine, 2021-09, Vol.13 (9), p.e14365-n/a [Peer Reviewed Journal]

2021 The Authors. Published under the terms of the CC BY 4.0 license ;2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.202114365 ;PMID: 34337880

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