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1
Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration
Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration
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Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration

Scientific reports, 2019-02, Vol.9 (1), p.2002, Article 2002 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-37862-3 ;PMID: 30765730

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2
Disorganization of intercalated discs in dilated cardiomyopathy
Disorganization of intercalated discs in dilated cardiomyopathy
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Disorganization of intercalated discs in dilated cardiomyopathy

Scientific reports, 2021-06, Vol.11 (1), p.11852-11852, Article 11852 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-90502-1 ;PMID: 34088908

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3
lncRNA H19/miR-675 axis regulates cardiomyocyte apoptosis by targeting VDAC1 in diabetic cardiomyopathy
lncRNA H19/miR-675 axis regulates cardiomyocyte apoptosis by targeting VDAC1 in diabetic cardiomyopathy
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lncRNA H19/miR-675 axis regulates cardiomyocyte apoptosis by targeting VDAC1 in diabetic cardiomyopathy

Scientific reports, 2016-10, Vol.6 (1), p.36340-36340, Article 36340 [Peer Reviewed Journal]

Copyright Nature Publishing Group Oct 2016 ;Copyright © 2016, The Author(s) 2016 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep36340 ;PMID: 27796346

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4
Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy
Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy
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Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy

Scientific reports, 2022-12, Vol.12 (1), p.22501-22501, Article 22501 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-26889-2 ;PMID: 36577774

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5
Circulating miR-19b and miR-181b are potential biomarkers for diabetic cardiomyopathy
Circulating miR-19b and miR-181b are potential biomarkers for diabetic cardiomyopathy
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Circulating miR-19b and miR-181b are potential biomarkers for diabetic cardiomyopathy

Scientific reports, 2017-10, Vol.7 (1), p.13514-11, Article 13514 [Peer Reviewed Journal]

2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-13875-2 ;PMID: 29044172

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6
The phospholamban p.(Arg14del) pathogenic variant leads to cardiomyopathy with heart failure and is unreponsive to standard heart failure therapy
The phospholamban p.(Arg14del) pathogenic variant leads to cardiomyopathy with heart failure and is unreponsive to standard heart failure therapy
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The phospholamban p.(Arg14del) pathogenic variant leads to cardiomyopathy with heart failure and is unreponsive to standard heart failure therapy

Scientific reports, 2020-06, Vol.10 (1), p.9819-9819 [Peer Reviewed Journal]

The Author(s) 2020 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-020-66656-9 ;PMID: 32555305

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7
Exploring TTN variants as genetic insights into cardiomyopathy pathogenesis and potential emerging clues to molecular mechanisms in cardiomyopathies
Exploring TTN variants as genetic insights into cardiomyopathy pathogenesis and potential emerging clues to molecular mechanisms in cardiomyopathies
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Exploring TTN variants as genetic insights into cardiomyopathy pathogenesis and potential emerging clues to molecular mechanisms in cardiomyopathies

Scientific reports, 2024-03, Vol.14 (1), p.5313-5313 [Peer Reviewed Journal]

2024. The Author(s). ;The Author(s) 2024 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-024-56154-7 ;PMID: 38438525

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8
Subcutaneous Ehrlich Ascites Carcinoma mice model for studying cancer-induced cardiomyopathy
Subcutaneous Ehrlich Ascites Carcinoma mice model for studying cancer-induced cardiomyopathy
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Subcutaneous Ehrlich Ascites Carcinoma mice model for studying cancer-induced cardiomyopathy

Scientific reports, 2018-04, Vol.8 (1), p.5599-11, Article 5599 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-23669-9 ;PMID: 29618792

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9
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling
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Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling

Scientific reports, 2018-01, Vol.8 (1), p.1998-11, Article 1998 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-20114-9 ;PMID: 29386531

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10
Sex differences in the prognosis of patients with hypertrophic cardiomyopathy
Sex differences in the prognosis of patients with hypertrophic cardiomyopathy
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Sex differences in the prognosis of patients with hypertrophic cardiomyopathy

Scientific reports, 2021-03, Vol.11 (1), p.4854-4854, Article 4854 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-84335-1 ;PMID: 33649405

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11
The embryological basis of subclinical hypertrophic cardiomyopathy
The embryological basis of subclinical hypertrophic cardiomyopathy
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The embryological basis of subclinical hypertrophic cardiomyopathy

Scientific reports, 2016-06, Vol.6 (1), p.27714, Article 27714 [Peer Reviewed Journal]

Copyright Nature Publishing Group Jun 2016 ;Copyright © 2016, Macmillan Publishers Limited 2016 Macmillan Publishers Limited ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep27714 ;PMID: 27323879

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12
Differences in molecular phenotype in mouse and human hypertrophic cardiomyopathy
Differences in molecular phenotype in mouse and human hypertrophic cardiomyopathy
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Differences in molecular phenotype in mouse and human hypertrophic cardiomyopathy

Scientific reports, 2021-06, Vol.11 (1), p.13163-13163, Article 13163 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-89451-6 ;PMID: 34162896

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13
Integrative network analysis interweaves the missing links in cardiomyopathy diseasome
Integrative network analysis interweaves the missing links in cardiomyopathy diseasome
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Integrative network analysis interweaves the missing links in cardiomyopathy diseasome

Scientific reports, 2022-11, Vol.12 (1), p.19670-19670, Article 19670 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-24246-x ;PMID: 36385157

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14
Survival and prognostic factors in hypertrophic cardiomyopathy: a meta-analysis
Survival and prognostic factors in hypertrophic cardiomyopathy: a meta-analysis
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Survival and prognostic factors in hypertrophic cardiomyopathy: a meta-analysis

Scientific reports, 2017-09, Vol.7 (1), p.11957-10, Article 11957 [Peer Reviewed Journal]

2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-12289-4 ;PMID: 28931939

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15
Myricitrin Alleviates Oxidative Stress-induced Inflammation and Apoptosis and Protects Mice against Diabetic Cardiomyopathy
Myricitrin Alleviates Oxidative Stress-induced Inflammation and Apoptosis and Protects Mice against Diabetic Cardiomyopathy
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Myricitrin Alleviates Oxidative Stress-induced Inflammation and Apoptosis and Protects Mice against Diabetic Cardiomyopathy

Scientific reports, 2017-03, Vol.7 (1), p.44239-44239, Article 44239 [Peer Reviewed Journal]

Copyright Nature Publishing Group Mar 2017 ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep44239 ;PMID: 28287141

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16
Genetic Basis and Genotype-Phenotype Correlations in Han Chinese Patients with Idiopathic Dilated Cardiomyopathy
Genetic Basis and Genotype-Phenotype Correlations in Han Chinese Patients with Idiopathic Dilated Cardiomyopathy
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Genetic Basis and Genotype-Phenotype Correlations in Han Chinese Patients with Idiopathic Dilated Cardiomyopathy

Scientific reports, 2020-02, Vol.10 (1), p.2226-2226, Article 2226 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-020-58984-7 ;PMID: 32041989

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17
Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy
Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy
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Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy

Scientific reports, 2019-12, Vol.9 (1), p.20350-8, Article 20350 [Peer Reviewed Journal]

2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-56617-2 ;PMID: 31889077

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18
Computational analysis of ventricular mechanics in hypertrophic cardiomyopathy patients
Computational analysis of ventricular mechanics in hypertrophic cardiomyopathy patients
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Computational analysis of ventricular mechanics in hypertrophic cardiomyopathy patients

Scientific reports, 2023-01, Vol.13 (1), p.958-958, Article 958 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-28037-w ;PMID: 36653468

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19
MiR-320a as a Potential Novel Circulating Biomarker of Arrhythmogenic CardioMyopathy
MiR-320a as a Potential Novel Circulating Biomarker of Arrhythmogenic CardioMyopathy
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MiR-320a as a Potential Novel Circulating Biomarker of Arrhythmogenic CardioMyopathy

Scientific reports, 2017-07, Vol.7 (1), p.4802-10, Article 4802 [Peer Reviewed Journal]

Copyright Nature Publishing Group Jul 2017 ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-05001-z ;PMID: 28684747

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20
The estimated glomerular filtration rate predicts pacemaker-induced cardiomyopathy
The estimated glomerular filtration rate predicts pacemaker-induced cardiomyopathy
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The estimated glomerular filtration rate predicts pacemaker-induced cardiomyopathy

Scientific reports, 2023-10, Vol.13 (1), p.16514-16514, Article 16514 [Peer Reviewed Journal]

Springer Nature Limited 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-43953-7 ;PMID: 37783787

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