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1
T4 ERK5 degradation: a turning point from compensated metabolic cardiomyopathy to heart failure
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Conference Proceeding
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T4 ERK5 degradation: a turning point from compensated metabolic cardiomyopathy to heart failure

Heart (British Cardiac Society), 2018, Vol.104 (Suppl 3), p.A2 [Peer Reviewed Journal]

2018, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/heartjnl-2018-BSCR.4

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2
P12 A novel model of cardiomyopathy reveals a tissue specific role for the complex i assembly factor ecsit
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P12 A novel model of cardiomyopathy reveals a tissue specific role for the complex i assembly factor ecsit

Heart (British Cardiac Society), 2018, Vol.104 (Suppl 3), p.A6 [Peer Reviewed Journal]

2018, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/heartjnl-2018-BSCR.17

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3
Evaluation of arrhythmias using an insertable cardiac monitor in patients with hypertrophic cardiomyopathy
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Evaluation of arrhythmias using an insertable cardiac monitor in patients with hypertrophic cardiomyopathy

EUROPEAN HEART JOURNAL, 2019, Vol.40, p.4169 [Peer Reviewed Journal]

ISSN: 1522-9645 ;ISSN: 0195-668X ;EISSN: 1522-9645 ;DOI: 10.1093/eurheartj/ehz746.1163

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4
Pathophysiology measured with positron emission tomography is equally impaired in hypertrophic and non-hypertrophic regions in hypertrophic cardiomyopathy
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Conference Proceeding
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Pathophysiology measured with positron emission tomography is equally impaired in hypertrophic and non-hypertrophic regions in hypertrophic cardiomyopathy

EUROPEAN HEART JOURNAL, 2019, Vol.40, p.1983 [Peer Reviewed Journal]

ISSN: 1522-9645 ;ISSN: 0195-668X ;EISSN: 1522-9645 ;DOI: 10.1093/eurheartj/ehz745.0237

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5
Abstract 796: Reproductive hormones modulate cardioprotection from doxorubicin-induced cardiomyopathy in female spontaneously hypertensive rats
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Abstract 796: Reproductive hormones modulate cardioprotection from doxorubicin-induced cardiomyopathy in female spontaneously hypertensive rats

Cancer research (Chicago, Ill.), 2017, Vol.77 (13_Supplement), p.796-796 [Peer Reviewed Journal]

ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/1538-7445.AM2017-796

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6
FAMILIAL RISK OF DILATED AND HYPERTROPHIC CARDIOMYOPATHY: A NATIONAL FAMILY STUDY IN SWEDEN
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FAMILIAL RISK OF DILATED AND HYPERTROPHIC CARDIOMYOPATHY: A NATIONAL FAMILY STUDY IN SWEDEN

EUROPEAN HEART JOURNAL SUPPLEMENTS, 2022, Vol.24 [Peer Reviewed Journal]

ISSN: 1520-765X ;EISSN: 1554-2815 ;DOI: 10.1093/eurheartjsupp/suac121.672

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7
Outcomes and phenotypic expression of rare variants in hypertrophic cardiomyopathy genes in over 200,000 adults
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Outcomes and phenotypic expression of rare variants in hypertrophic cardiomyopathy genes in over 200,000 adults

The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. This article is published and distributed under the terms of the Oxford University Press, Standard Journals Publication Model (https://academic.oup.com/journals/pages/open_access/funder_policies/chorus/standard_publication_model) ;ISSN: 0195-668X ;EISSN: 1522-9645 ;DOI: 10.1093/eurheartj/ehab724.1731

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8
Abstract 185: Genome Editing of Isogenic Human Induced Pluripotent Stem Cells Allows for Functional and Transcriptomic Insights Into Hereditary Dilated Cardiomyopathy Caused by Phospholamban Mutations
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Abstract 185: Genome Editing of Isogenic Human Induced Pluripotent Stem Cells Allows for Functional and Transcriptomic Insights Into Hereditary Dilated Cardiomyopathy Caused by Phospholamban Mutations

CIRCULATION RESEARCH, 2017, Vol.121 (suppl_1) [Peer Reviewed Journal]

ISSN: 0009-7330 ;EISSN: 1524-4571 ;DOI: 10.1161/res.121.suppl_1.185

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9
Transient vs. persistent improved ejection fraction in non-ischaemic dilated cardiomyopathy
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Conference Proceeding
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Transient vs. persistent improved ejection fraction in non-ischaemic dilated cardiomyopathy

EUROPEAN HEART JOURNAL SUPPLEMENTS, 2021, Vol.23 (G), p.G116 [Peer Reviewed Journal]

ISSN: 1520-765X ;EISSN: 1554-2815 ;DOI: 10.1093/eurheartj/suab139.027

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10
BEYOND 15MM: TOWARDS A NEW PERSONALISED, AI MEASURED DEFINITION OF HYPERTROPHIC CARDIOMYOPATHY USING AN AGE, SEX, AND BODY SURFACE AREA-ADJUSTED APPROACH
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Conference Proceeding
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BEYOND 15MM: TOWARDS A NEW PERSONALISED, AI MEASURED DEFINITION OF HYPERTROPHIC CARDIOMYOPATHY USING AN AGE, SEX, AND BODY SURFACE AREA-ADJUSTED APPROACH

HEART, 2023, Vol.109 (SUPPL_1), p.A24 [Peer Reviewed Journal]

ISSN: 1355-6037 ;EISSN: 1468-201X

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11
Impaired myocardial perfusion reserve is associated with adverse cardiovascular events in patients with dilated cardiomyopathy
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Impaired myocardial perfusion reserve is associated with adverse cardiovascular events in patients with dilated cardiomyopathy

2019 by American Heart Association, Inc. ;ISSN: 0009-7322 ;EISSN: 1524-4539

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12
P52 Two pathologies one heart: life-threatening lupus myocarditis in a patient with pre-existing dilated cardiomyopathy
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P52 Two pathologies one heart: life-threatening lupus myocarditis in a patient with pre-existing dilated cardiomyopathy

The Author(s) 2020. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For permissions, please email: journals.permissions@oup.com ;ISSN: 1462-0324 ;EISSN: 1462-0332 ;DOI: 10.1093/rheumatology/keaa111.051

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13
An altered nucleotide metabolism as a novel mechanism leading to Huntington disease related cardiomyopathy
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An altered nucleotide metabolism as a novel mechanism leading to Huntington disease related cardiomyopathy

2019 Springer Nature B.V. The final publication is available at Springer via https://dx.doi.org/10.1007/s11302-018-9637-0 2019-12-01 ;ISSN: 1573-9538 ;EISSN: 1573-9546

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14
An altered nucleotide metabolism as a novel mechanism leading to Huntington disease related cardiomyopathy
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An altered nucleotide metabolism as a novel mechanism leading to Huntington disease related cardiomyopathy

2019 Springer Nature B.V. The final publication is available at Springer via https://dx.doi.org/10.1007/s11302-018-9637-0 ;ISSN: 1573-9538 ;EISSN: 1573-9546

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15
An altered metabolism of nucleotides leads to huntington’s disease related cardiomyopathy
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An altered metabolism of nucleotides leads to huntington’s disease related cardiomyopathy

2018, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 1468-330X ;EISSN: 1468-330X

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16
Titin Truncating Variants Predict Life-threatening Arrhythmias in Patients With Dilated Cardiomyopathy
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Titin Truncating Variants Predict Life-threatening Arrhythmias in Patients With Dilated Cardiomyopathy

2017 American Heart Association, Inc. ;ISSN: 0009-7322 ;EISSN: 1524-4539

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17
Phospholamban antisense oligonucleotides drive the reversal of cardiac dysfunction and multiple heart failure parameters during murine dilated cardiomyopathy
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Phospholamban antisense oligonucleotides drive the reversal of cardiac dysfunction and multiple heart failure parameters during murine dilated cardiomyopathy

EUROPEAN HEART JOURNAL, 2019, Vol.40, p.3950 [Peer Reviewed Journal]

ISSN: 0195-668X ;EISSN: 1522-9645

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18
Risk of sudden cardiac death in new york heart association class 1 patients with dilated cardiomyopathy: a competing risk analysis
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Conference Proceeding
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Risk of sudden cardiac death in new york heart association class 1 patients with dilated cardiomyopathy: a competing risk analysis

EUROPEAN HEART JOURNAL SUPPLEMENTS, 2019, Vol.21 (J), p.J135 [Peer Reviewed Journal]

ISSN: 1520-765X ;EISSN: 1554-2815

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19
New variant in CMYA5 gene is associated with early-onset restrictive cardiomyopathy and autism-spectrum disorder
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Conference Proceeding
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New variant in CMYA5 gene is associated with early-onset restrictive cardiomyopathy and autism-spectrum disorder

CARDIOVASCULAR RESEARCH, 2018, Vol.114, p.S19 [Peer Reviewed Journal]

ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvy060.036

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20
A cardiomyopathy of unusual origin
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A cardiomyopathy of unusual origin

Revue medicale suisse, 2016, Vol.12 (502), p.148-151 [Peer Reviewed Journal]

ISSN: 1660-9379 ;PMID: 26946792

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