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Refined by: Journal Title: Scientific Reports remove subject: Ventricle remove
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1
Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration
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Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration

Scientific reports, 2019-02, Vol.9 (1), p.2002, Article 2002 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-37862-3 ;PMID: 30765730

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2
Disorganization of intercalated discs in dilated cardiomyopathy
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Disorganization of intercalated discs in dilated cardiomyopathy

Scientific reports, 2021-06, Vol.11 (1), p.11852-11852, Article 11852 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-90502-1 ;PMID: 34088908

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3
lncRNA H19/miR-675 axis regulates cardiomyocyte apoptosis by targeting VDAC1 in diabetic cardiomyopathy
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lncRNA H19/miR-675 axis regulates cardiomyocyte apoptosis by targeting VDAC1 in diabetic cardiomyopathy

Scientific reports, 2016-10, Vol.6 (1), p.36340-36340, Article 36340 [Peer Reviewed Journal]

Copyright Nature Publishing Group Oct 2016 ;Copyright © 2016, The Author(s) 2016 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep36340 ;PMID: 27796346

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4
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling
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Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling

Scientific reports, 2018-01, Vol.8 (1), p.1998-11, Article 1998 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-20114-9 ;PMID: 29386531

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5
Subcutaneous Ehrlich Ascites Carcinoma mice model for studying cancer-induced cardiomyopathy
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Article
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Subcutaneous Ehrlich Ascites Carcinoma mice model for studying cancer-induced cardiomyopathy

Scientific reports, 2018-04, Vol.8 (1), p.5599-11, Article 5599 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-23669-9 ;PMID: 29618792

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6
Computational analysis of ventricular mechanics in hypertrophic cardiomyopathy patients
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Computational analysis of ventricular mechanics in hypertrophic cardiomyopathy patients

Scientific reports, 2023-01, Vol.13 (1), p.958-958, Article 958 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-28037-w ;PMID: 36653468

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7
The estimated glomerular filtration rate predicts pacemaker-induced cardiomyopathy
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The estimated glomerular filtration rate predicts pacemaker-induced cardiomyopathy

Scientific reports, 2023-10, Vol.13 (1), p.16514-16514, Article 16514 [Peer Reviewed Journal]

The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Springer Nature Limited 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-43953-7 ;PMID: 37783787

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8
Survival and prognostic factors in hypertrophic cardiomyopathy: a meta-analysis
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Survival and prognostic factors in hypertrophic cardiomyopathy: a meta-analysis

Scientific reports, 2017-09, Vol.7 (1), p.11957-10, Article 11957 [Peer Reviewed Journal]

2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-12289-4 ;PMID: 28931939

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9
MiR-320a as a Potential Novel Circulating Biomarker of Arrhythmogenic CardioMyopathy
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MiR-320a as a Potential Novel Circulating Biomarker of Arrhythmogenic CardioMyopathy

Scientific reports, 2017-07, Vol.7 (1), p.4802-10, Article 4802 [Peer Reviewed Journal]

Copyright Nature Publishing Group Jul 2017 ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-05001-z ;PMID: 28684747

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10
Arrhythmogenic Left Ventricular Cardiomyopathy: A Clinical and CMR Study
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Article
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Arrhythmogenic Left Ventricular Cardiomyopathy: A Clinical and CMR Study

Scientific reports, 2020-01, Vol.10 (1), p.533-533, Article 533 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-57203-2 ;PMID: 31953454

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11
Blockade of L-type Ca2+ channel attenuates doxorubicin-induced cardiomyopathy via suppression of CaMKII-NF-κB pathway
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Article
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Blockade of L-type Ca2+ channel attenuates doxorubicin-induced cardiomyopathy via suppression of CaMKII-NF-κB pathway

Scientific reports, 2019-07, Vol.9 (1), p.1-14, Article 9850 [Peer Reviewed Journal]

2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-46367-6 ;PMID: 31285514

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12
Left ventricular global function index by magnetic resonance imaging - a novel marker for differentiating cardiac amyloidosis from hypertrophic cardiomyopathy
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Article
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Left ventricular global function index by magnetic resonance imaging - a novel marker for differentiating cardiac amyloidosis from hypertrophic cardiomyopathy

Scientific reports, 2020-03, Vol.10 (1), p.4707-4707, Article 4707 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-020-61608-9 ;PMID: 32170130

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13
Comparison of the antiremodeling effects of losartan and mirabegron in a rat model of uremic cardiomyopathy
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Comparison of the antiremodeling effects of losartan and mirabegron in a rat model of uremic cardiomyopathy

Scientific reports, 2021-09, Vol.11 (1), p.17495-17495, Article 17495 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-96815-5 ;PMID: 34471171

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14
A pilot study for risk stratification of ventricular tachyarrhythmia in hypertrophic cardiomyopathy with routine echocardiography parameters
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A pilot study for risk stratification of ventricular tachyarrhythmia in hypertrophic cardiomyopathy with routine echocardiography parameters

Scientific reports, 2024-02, Vol.14 (1), p.3799-3799 [Peer Reviewed Journal]

2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-024-54153-2 ;PMID: 38360886

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15
Identification of key immune-related genes in dilated cardiomyopathy using bioinformatics analysis
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Identification of key immune-related genes in dilated cardiomyopathy using bioinformatics analysis

Scientific reports, 2023-02, Vol.13 (1), p.1820-1820, Article 1820 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. corrected publication 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-26277-w ;PMID: 36725968

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16
Exploring potential therapeutic agents for lipopolysaccharide-induced septic cardiomyopathy based on transcriptomics using bioinformatics
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Article
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Exploring potential therapeutic agents for lipopolysaccharide-induced septic cardiomyopathy based on transcriptomics using bioinformatics

Scientific reports, 2023-11, Vol.13 (1), p.20589-20589, Article 20589 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-47699-0 ;PMID: 37996554

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17
Mesenchymal stromal cells inhibit NLRP3 inflammasome activation in a model of Coxsackievirus B3-induced inflammatory cardiomyopathy
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Article
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Mesenchymal stromal cells inhibit NLRP3 inflammasome activation in a model of Coxsackievirus B3-induced inflammatory cardiomyopathy

Scientific reports, 2018-02, Vol.8 (1), p.2820-16, Article 2820 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-20686-6 ;PMID: 29434214

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18
Altered intercellular communication and extracellular matrix signaling as a potential disease mechanism in human hypertrophic cardiomyopathy
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Altered intercellular communication and extracellular matrix signaling as a potential disease mechanism in human hypertrophic cardiomyopathy

Scientific reports, 2022-03, Vol.12 (1), p.5211-5211, Article 5211 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-08561-x ;PMID: 35338173

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19
Potent Paracrine Effects of human induced Pluripotent Stem Cell-derived Mesenchymal Stem Cells Attenuate Doxorubicin-induced Cardiomyopathy
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Article
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Potent Paracrine Effects of human induced Pluripotent Stem Cell-derived Mesenchymal Stem Cells Attenuate Doxorubicin-induced Cardiomyopathy

Scientific reports, 2015-06, Vol.5 (1), p.11235-11235, Article 11235 [Peer Reviewed Journal]

Copyright Nature Publishing Group Jun 2015 ;Copyright © 2015, Macmillan Publishers Limited 2015 Macmillan Publishers Limited ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep11235 ;PMID: 26057572

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20
The kisspeptin-1 receptor antagonist peptide-234 aggravates uremic cardiomyopathy in a rat model
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Article
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The kisspeptin-1 receptor antagonist peptide-234 aggravates uremic cardiomyopathy in a rat model

Scientific reports, 2023-08, Vol.13 (1), p.14046-14046, Article 14046 [Peer Reviewed Journal]

Springer Nature Limited 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Springer Nature Limited 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-41037-0 ;PMID: 37640761

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