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1
Correction: SVR12 rates higher than 99% after sofosbuvir/velpatasvir combination in HCV infected patients with F0-F1 fibrosis stage: A real world experience
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Correction: SVR12 rates higher than 99% after sofosbuvir/velpatasvir combination in HCV infected patients with F0-F1 fibrosis stage: A real world experience

PloS one, 2019-09, Vol.14 (9), p.e0223287-e0223287 [Peer Reviewed Journal]

COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Mangia et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Mangia et al 2019 Mangia et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0223287 ;PMID: 31553772

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2
Correction to: Effects of phosphodiesterase 4 inhibition on bleomycin-induced pulmonary fibrosis in mice
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Correction to: Effects of phosphodiesterase 4 inhibition on bleomycin-induced pulmonary fibrosis in mice

BMC pulmonary medicine, 2022-03, Vol.22 (1), p.113-113, Article 113 [Peer Reviewed Journal]

COPYRIGHT 2022 BioMed Central Ltd. ;2022. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 1471-2466 ;EISSN: 1471-2466 ;DOI: 10.1186/s12890-022-01876-5 ;PMID: 35351085

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3
Short telomeres are a risk factor for idiopathic pulmonary fibrosis
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Short telomeres are a risk factor for idiopathic pulmonary fibrosis

Proceedings of the National Academy of Sciences - PNAS, 2008-09, Vol.105 (35), p.13051-13056 [Peer Reviewed Journal]

Copyright 2008 The National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Sep 2, 2008 ;2008 by The National Academy of Sciences of the USA ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.0804280105 ;PMID: 18753630

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4
Low-Dose of Intrapulmonary Pirfenidone Improves Human Transforming Growth Factorbeta;1-Driven Lung Fibrosis
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Low-Dose of Intrapulmonary Pirfenidone Improves Human Transforming Growth Factorbeta;1-Driven Lung Fibrosis

Frontiers in pharmacology, 2020-11 [Peer Reviewed Journal]

COPYRIGHT 2020 Frontiers Research Foundation ;ISSN: 1663-9812 ;EISSN: 1663-9812

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5
Emerging role of HMGB1 in fibrotic diseases
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Emerging role of HMGB1 in fibrotic diseases

Journal of cellular and molecular medicine, 2014-12, Vol.18 (12), p.2331-2339 [Peer Reviewed Journal]

2014 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;Copyright © 2014 John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine ;2014 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. 2014 ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.12419 ;PMID: 25284457

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6
Cystic fibrosis: a clinical view
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Cystic fibrosis: a clinical view

Cellular and molecular life sciences : CMLS, 2017-01, Vol.74 (1), p.129-140 [Peer Reviewed Journal]

Springer International Publishing 2016 ;Cellular and Molecular Life Sciences is a copyright of Springer, 2017. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-016-2393-9 ;PMID: 27709245

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7
Efficacy and Safety of Ivacaftor in Patients Aged 6 to 11 Years with Cystic Fibrosis with a G551D Mutation
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Efficacy and Safety of Ivacaftor in Patients Aged 6 to 11 Years with Cystic Fibrosis with a G551D Mutation

American journal of respiratory and critical care medicine, 2013-06, Vol.187 (11), p.1219-1225 [Peer Reviewed Journal]

2014 INIST-CNRS ;Copyright American Thoracic Society Jun 1, 2013 ;Copyright © 2013 by the American Thoracic Society 2013 ;ISSN: 1073-449X ;EISSN: 1535-4970 ;DOI: 10.1164/rccm.201301-0153OC ;PMID: 23590265

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8
Type V collagen induced tolerance suppresses collagen deposition, TGF-β and associated transcripts in pulmonary fibrosis
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Type V collagen induced tolerance suppresses collagen deposition, TGF-β and associated transcripts in pulmonary fibrosis

PloS one, 2013-10, Vol.8 (10), p.e76451 [Peer Reviewed Journal]

2013 Vittal et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Vittal et al 2013 Vittal et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0076451 ;PMID: 24204629

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9
Pulmonary fibrosis 4 months after COVID-19 is associated with severity of illness and blood leucocyte telomere length
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Pulmonary fibrosis 4 months after COVID-19 is associated with severity of illness and blood leucocyte telomere length

Thorax, 2021-12, Vol.76 (12), p.1242-1245 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2021. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. ;2021 Author(s) (or their employer(s)) 2021. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. http://creativecommons.org/licenses/by-nc/4.0/ This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ . Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Author(s) (or their employer(s)) 2021. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. 2021 ;ISSN: 0040-6376 ;EISSN: 1468-3296 ;DOI: 10.1136/thoraxjnl-2021-217031 ;PMID: 33927016

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10
Pulmonary fibrosis: patterns and perpetrators
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Pulmonary fibrosis: patterns and perpetrators

The Journal of clinical investigation, 2012-08, Vol.122 (8), p.2756-2762 [Peer Reviewed Journal]

COPYRIGHT 2012 American Society for Clinical Investigation ;COPYRIGHT 2012 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Aug 2012 ;Copyright © 2012, American Society for Clinical Investigation 2012 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/jci60323 ;PMID: 22850886

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11
Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44
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Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44

The Journal of experimental medicine, 2011-07, Vol.208 (7), p.1459-1471 [Peer Reviewed Journal]

2011 Li et al. 2011 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20102510 ;PMID: 21708929

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12
Idiopathic Pulmonary Fibrosis (an Update) and Progressive Pulmonary Fibrosis in Adults: An Official ATS/ERS/JRS/ALAT Clinical Practice Guideline
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Idiopathic Pulmonary Fibrosis (an Update) and Progressive Pulmonary Fibrosis in Adults: An Official ATS/ERS/JRS/ALAT Clinical Practice Guideline

American journal of respiratory and critical care medicine, 2022-05, Vol.205 (9), p.e18-e47 [Peer Reviewed Journal]

Copyright American Thoracic Society May 1, 2022 ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 1073-449X ;EISSN: 1535-4970 ;DOI: 10.1164/rccm.202202-0399st ;PMID: 35486072

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13
Capitalizing on the heterogeneous effects of CFTR nonsense and frameshift variants to inform therapeutic strategy for cystic fibrosis
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Capitalizing on the heterogeneous effects of CFTR nonsense and frameshift variants to inform therapeutic strategy for cystic fibrosis

PLoS genetics, 2018-11, Vol.14 (11), p.e1007723-e1007723 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Sharma et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Sharma et al 2018 Sharma et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1007723 ;PMID: 30444886

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14
A Systematic Review of the Clinical Efficacy and Safety of CFTR Modulators in Cystic Fibrosis
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A Systematic Review of the Clinical Efficacy and Safety of CFTR Modulators in Cystic Fibrosis

Scientific reports, 2019-05, Vol.9 (1), p.7234, Article 7234 [Peer Reviewed Journal]

The Author(s) 2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-43652-2 ;PMID: 31076617

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15
Mucin 5B promoter polymorphism is associated with idiopathic pulmonary fibrosis but not with development of lung fibrosis in systemic sclerosis or sarcoidosis
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Mucin 5B promoter polymorphism is associated with idiopathic pulmonary fibrosis but not with development of lung fibrosis in systemic sclerosis or sarcoidosis

Thorax, 2013-05, Vol.68 (5), p.436-441 [Peer Reviewed Journal]

Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;Copyright: 2013 Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 0040-6376 ;EISSN: 1468-3296 ;DOI: 10.1136/thoraxjnl-2012-201786 ;PMID: 23321605 ;CODEN: THORA7

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16
Blockade of CCL24 with a monoclonal antibody ameliorates experimental dermal and pulmonary fibrosis
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Article
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Blockade of CCL24 with a monoclonal antibody ameliorates experimental dermal and pulmonary fibrosis

Annals of the rheumatic diseases, 2019-09, Vol.78 (9), p.1260-1268 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. ;2019 Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ . Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. 2019 ;ISSN: 0003-4967 ;EISSN: 1468-2060 ;DOI: 10.1136/annrheumdis-2019-215119 ;PMID: 31129606

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17
Inhalation of lung spheroid cell secretome and exosomes promotes lung repair in pulmonary fibrosis
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Inhalation of lung spheroid cell secretome and exosomes promotes lung repair in pulmonary fibrosis

Nature communications, 2020-02, Vol.11 (1), p.1064-14, Article 1064 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-14344-7 ;PMID: 32111836

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18
Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3
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Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3

American journal of respiratory and critical care medicine, 2012-03, Vol.185 (5), p.537-546 [Peer Reviewed Journal]

2015 INIST-CNRS ;Copyright © 2012 by the American Thoracic Society 2012 ;ISSN: 1073-449X ;EISSN: 1535-4970 ;DOI: 10.1164/rccm.201106-0965oc ;PMID: 22095546

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19
Restoring Cystic Fibrosis Transmembrane Conductance Regulator Function Reduces Airway Bacteria and Inflammation in People with Cystic Fibrosis and Chronic Lung Infections
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Restoring Cystic Fibrosis Transmembrane Conductance Regulator Function Reduces Airway Bacteria and Inflammation in People with Cystic Fibrosis and Chronic Lung Infections

American journal of respiratory and critical care medicine, 2017-06, Vol.195 (12), p.1617-1628 [Peer Reviewed Journal]

Copyright American Thoracic Society Jun 15, 2017 ;Copyright © 2017 by the American Thoracic Society 2017 ;ISSN: 1073-449X ;EISSN: 1535-4970 ;DOI: 10.1164/rccm.201609-1954OC ;PMID: 28222269

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20
Pharmacological chaperones improve intra-domain stability and inter-domain assembly via distinct binding sites to rescue misfolded CFTR
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Pharmacological chaperones improve intra-domain stability and inter-domain assembly via distinct binding sites to rescue misfolded CFTR

Cellular and molecular life sciences : CMLS, 2021-12, Vol.78 (23), p.7813-7829 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 ;2021. The Author(s), under exclusive licence to Springer Nature Switzerland AG. ;The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 1420-682X ;ISSN: 1420-9071 ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-021-03994-5 ;PMID: 34714360

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