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1
Prevention of exercised induced cardiomyopathy following Pip-PMO treatment in dystrophic mdx mice
Prevention of exercised induced cardiomyopathy following Pip-PMO treatment in dystrophic mdx mice
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Prevention of exercised induced cardiomyopathy following Pip-PMO treatment in dystrophic mdx mice

Scientific reports, 2015-03, Vol.5 (1), p.8986-8986, Article 8986 [Peer Reviewed Journal]

Copyright Nature Publishing Group Mar 2015 ;Copyright © 2015, Macmillan Publishers Limited. All rights reserved 2015 Macmillan Publishers Limited. All rights reserved ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep08986 ;PMID: 25758104

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2
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling
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Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling

Scientific reports, 2018-01, Vol.8 (1), p.1998-11, Article 1998 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-20114-9 ;PMID: 29386531

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3
Homogeneous 2D and 3D alignment of cardiomyocyte in dilated cardiomyopathy revealed by intravital heart imaging
Homogeneous 2D and 3D alignment of cardiomyocyte in dilated cardiomyopathy revealed by intravital heart imaging
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Homogeneous 2D and 3D alignment of cardiomyocyte in dilated cardiomyopathy revealed by intravital heart imaging

Scientific reports, 2021-07, Vol.11 (1), p.14698-14698, Article 14698 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-94100-z ;PMID: 34282197

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4
Investigation of diets associated with dilated cardiomyopathy in dogs using foodomics analysis
Investigation of diets associated with dilated cardiomyopathy in dogs using foodomics analysis
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Investigation of diets associated with dilated cardiomyopathy in dogs using foodomics analysis

Scientific reports, 2021-08, Vol.11 (1), p.15881-12, Article 15881 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-94464-2 ;PMID: 34354102

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5
Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy
Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy
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Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy

Scientific reports, 2018-01, Vol.8 (1), p.724-9, Article 724 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-19138-4 ;PMID: 29335596

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6
Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy
Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy
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Serum circular RNAs act as blood-based biomarkers for hypertrophic obstructive cardiomyopathy

Scientific reports, 2019-12, Vol.9 (1), p.20350-8, Article 20350 [Peer Reviewed Journal]

2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-56617-2 ;PMID: 31889077

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7
Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes
Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes
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Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes

Scientific reports, 2017-11, Vol.7 (1), p.14829-11, Article 14829 [Peer Reviewed Journal]

Scientific Reports is a copyright of Springer, 2017. ;2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-13675-8 ;PMID: 29093449

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8
Mechanistic clues to the protective effect of chrysin against doxorubicin-induced cardiomyopathy: Plausible roles of p53, MAPK and AKT pathways
Mechanistic clues to the protective effect of chrysin against doxorubicin-induced cardiomyopathy: Plausible roles of p53, MAPK and AKT pathways
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Mechanistic clues to the protective effect of chrysin against doxorubicin-induced cardiomyopathy: Plausible roles of p53, MAPK and AKT pathways

Scientific reports, 2017-07, Vol.7 (1), p.4795-13, Article 4795 [Peer Reviewed Journal]

Copyright Nature Publishing Group Jul 2017 ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-05005-9 ;PMID: 28684738

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9
Prevalence, functional characteristics, and clinical significance of right ventricular involvement in patients with hypertrophic cardiomyopathy
Prevalence, functional characteristics, and clinical significance of right ventricular involvement in patients with hypertrophic cardiomyopathy
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Prevalence, functional characteristics, and clinical significance of right ventricular involvement in patients with hypertrophic cardiomyopathy

Scientific reports, 2020-12, Vol.10 (1), p.21908-21908, Article 21908 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-020-78945-4 ;PMID: 33318610

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10
Identification of key immune-related genes in dilated cardiomyopathy using bioinformatics analysis
Identification of key immune-related genes in dilated cardiomyopathy using bioinformatics analysis
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Identification of key immune-related genes in dilated cardiomyopathy using bioinformatics analysis

Scientific reports, 2023-02, Vol.13 (1), p.1820-1820, Article 1820 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. corrected publication 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-26277-w ;PMID: 36725968

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11
Comparison of the antiremodeling effects of losartan and mirabegron in a rat model of uremic cardiomyopathy
Comparison of the antiremodeling effects of losartan and mirabegron in a rat model of uremic cardiomyopathy
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Comparison of the antiremodeling effects of losartan and mirabegron in a rat model of uremic cardiomyopathy

Scientific reports, 2021-09, Vol.11 (1), p.17495-17495, Article 17495 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-96815-5 ;PMID: 34471171

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12
Mutation analysis of the phospholamban gene in 315 South Africans with dilated, hypertrophic, peripartum and arrhythmogenic right ventricular cardiomyopathies
Mutation analysis of the phospholamban gene in 315 South Africans with dilated, hypertrophic, peripartum and arrhythmogenic right ventricular cardiomyopathies
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Mutation analysis of the phospholamban gene in 315 South Africans with dilated, hypertrophic, peripartum and arrhythmogenic right ventricular cardiomyopathies

Scientific reports, 2016-02, Vol.6 (1), p.22235-22235, Article 22235 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2016 ;Copyright © 2016, Macmillan Publishers Limited 2016 Macmillan Publishers Limited ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep22235 ;PMID: 26917049

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13
Association of the transthyretin variant V122I with polyneuropathy among individuals of African ancestry
Association of the transthyretin variant V122I with polyneuropathy among individuals of African ancestry
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Association of the transthyretin variant V122I with polyneuropathy among individuals of African ancestry

Scientific reports, 2021-06, Vol.11 (1), p.11645-10, Article 11645 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-91113-6 ;PMID: 34079032

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14
Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy
Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy
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Mechanism based therapies enable personalised treatment of hypertrophic cardiomyopathy

Scientific reports, 2022-12, Vol.12 (1), p.22501-22501, Article 22501 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-26889-2 ;PMID: 36577774

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15
Identification of MYLK3 mutations in familial dilated cardiomyopathy
Identification of MYLK3 mutations in familial dilated cardiomyopathy
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Identification of MYLK3 mutations in familial dilated cardiomyopathy

Scientific reports, 2017-12, Vol.7 (1), p.17495-9, Article 17495 [Peer Reviewed Journal]

2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2017 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-17769-1 ;PMID: 29235529

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16
Retrospective analysis of clinical phenotype and prognosis of hypertrophic cardiomyopathy complicated with hypertension
Retrospective analysis of clinical phenotype and prognosis of hypertrophic cardiomyopathy complicated with hypertension
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Retrospective analysis of clinical phenotype and prognosis of hypertrophic cardiomyopathy complicated with hypertension

Scientific reports, 2020-01, Vol.10 (1), p.349-349, Article 349 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-57230-z ;PMID: 31941943

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17
A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy
A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy
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A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy

Scientific reports, 2018-09, Vol.8 (1), p.13804-12, Article 13804 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-31772-0 ;PMID: 30218094

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18
A deep learning framework assisted echocardiography with diagnosis, lesion localization, phenogrouping heterogeneous disease, and anomaly detection
A deep learning framework assisted echocardiography with diagnosis, lesion localization, phenogrouping heterogeneous disease, and anomaly detection
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A deep learning framework assisted echocardiography with diagnosis, lesion localization, phenogrouping heterogeneous disease, and anomaly detection

Scientific reports, 2023-01, Vol.13 (1), p.3-3, Article 3 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-022-27211-w ;PMID: 36593284

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19
Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration
Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration
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Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration

Scientific reports, 2019-02, Vol.9 (1), p.2002, Article 2002 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-37862-3 ;PMID: 30765730

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20
A missense mutation in the RSRSP stretch of Rbm20 causes dilated cardiomyopathy and atrial fibrillation in mice
A missense mutation in the RSRSP stretch of Rbm20 causes dilated cardiomyopathy and atrial fibrillation in mice
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A missense mutation in the RSRSP stretch of Rbm20 causes dilated cardiomyopathy and atrial fibrillation in mice

Scientific reports, 2020-10, Vol.10 (1), p.17894-17894, Article 17894 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-020-74800-8 ;PMID: 33110103

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