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1
Maternal Exposure to PM 2.5 and the Risk of Congenital Heart Defects in 1.4 Million Births: A Nationwide Surveillance-Based Study
Maternal Exposure to PM 2.5 and the Risk of Congenital Heart Defects in 1.4 Million Births: A Nationwide Surveillance-Based Study
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Maternal Exposure to PM 2.5 and the Risk of Congenital Heart Defects in 1.4 Million Births: A Nationwide Surveillance-Based Study

Circulation (New York, N.Y.), 2023-02, Vol.147 (7), p.565-574 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.122.061245 ;PMID: 36780386

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2
Air pollution and cardiovascular disease : A statement for healthcare professionals from the expert panel on population and prevention science of the American heart association
Air pollution and cardiovascular disease : A statement for healthcare professionals from the expert panel on population and prevention science of the American heart association
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Air pollution and cardiovascular disease : A statement for healthcare professionals from the expert panel on population and prevention science of the American heart association

Circulation (New York, N.Y.), 2004-06, Vol.109 (21), p.2655-2671 [Peer Reviewed Journal]

2005 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.0000128587.30041.C8 ;PMID: 15173049 ;CODEN: CIRCAZ

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3
Clinical and pathophysiological implications of a bicuspid aortic valve
Clinical and pathophysiological implications of a bicuspid aortic valve
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Clinical and pathophysiological implications of a bicuspid aortic valve

Circulation (New York, N.Y.), 2002-08, Vol.106 (8), p.900-904 [Peer Reviewed Journal]

2002 INIST-CNRS ;Copyright American Heart Association, Inc. Aug 20, 2002 ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.cir.0000027905.26586.e8 ;PMID: 12186790 ;CODEN: CIRCAZ

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4
Type A Aortic Dissection After Nonaortic Cardiac Surgery
Type A Aortic Dissection After Nonaortic Cardiac Surgery
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Type A Aortic Dissection After Nonaortic Cardiac Surgery

Circulation (New York, N.Y.), 2013-10, Vol.128 (15), p.1602-1611 [Peer Reviewed Journal]

2013 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.113.002603 ;PMID: 24025592 ;CODEN: CIRCAZ

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5
Shortened outflow tract leads to altered cardiac looping after neural crest ablation
Shortened outflow tract leads to altered cardiac looping after neural crest ablation
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Shortened outflow tract leads to altered cardiac looping after neural crest ablation

Circulation (New York, N.Y.), 2002-07, Vol.106 (4), p.504-510 [Peer Reviewed Journal]

2002 INIST-CNRS ;Copyright American Heart Association, Inc. Jul 23, 2002 ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.cir.0000023044.44974.8a ;PMID: 12135953 ;CODEN: CIRCAZ

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6
Noninherited risk factors and congenital cardiovascular defects: current knowledge: a scientific statement from the American Heart Association Council on Cardiovascular Disease in the Young: endorsed by the American Academy of Pediatrics
Noninherited risk factors and congenital cardiovascular defects: current knowledge: a scientific statement from the American Heart Association Council on Cardiovascular Disease in the Young: endorsed by the American Academy of Pediatrics
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Noninherited risk factors and congenital cardiovascular defects: current knowledge: a scientific statement from the American Heart Association Council on Cardiovascular Disease in the Young: endorsed by the American Academy of Pediatrics

Circulation (New York, N.Y.), 2007-06, Vol.115 (23), p.2995-3014 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.106.183216 ;PMID: 17519397

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7
Effects of the size of lesions of the cardiac neural crest at various embryonic ages on incidence and type of cardiac defects
Effects of the size of lesions of the cardiac neural crest at various embryonic ages on incidence and type of cardiac defects
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Effects of the size of lesions of the cardiac neural crest at various embryonic ages on incidence and type of cardiac defects

Circulation (New York, N.Y.), 1986-02, Vol.73 (2), p.360-364 [Peer Reviewed Journal]

1987 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.73.2.360 ;PMID: 3943168 ;CODEN: CIRCAZ

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8
Pulmonary vascular disease with congenital heart lesions: pathologic features and causes
Pulmonary vascular disease with congenital heart lesions: pathologic features and causes
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Pulmonary vascular disease with congenital heart lesions: pathologic features and causes

Circulation (New York, N.Y.), 1981-11, Vol.64 (5), p.873-877 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.64.5.873 ;PMID: 7026082

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9
Hemodynamic, pulmonary vascular, and myocardial abnormalities secondary to pharmacologic constriction of the fetal ductus arteriosus. A possible mechanism for persistent pulmonary hypertension and transient tricuspid insufficiency in the newborn infant
Hemodynamic, pulmonary vascular, and myocardial abnormalities secondary to pharmacologic constriction of the fetal ductus arteriosus. A possible mechanism for persistent pulmonary hypertension and transient tricuspid insufficiency in the newborn infant
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Hemodynamic, pulmonary vascular, and myocardial abnormalities secondary to pharmacologic constriction of the fetal ductus arteriosus. A possible mechanism for persistent pulmonary hypertension and transient tricuspid insufficiency in the newborn infant

Circulation (New York, N.Y.), 1979-08, Vol.60 (2), p.360-364 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.60.2.360 ;PMID: 445754

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10
The evolution of specific genetic and environmental counseling in congenital heart diseases
The evolution of specific genetic and environmental counseling in congenital heart diseases
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The evolution of specific genetic and environmental counseling in congenital heart diseases

Circulation (New York, N.Y.), 1978-02, Vol.57 (2), p.205-213 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.57.2.205 ;PMID: 338196

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11
Multifactorial inheritance hypothesis for the etiology of congenital heart diseases. The genetic-environmental interaction
Multifactorial inheritance hypothesis for the etiology of congenital heart diseases. The genetic-environmental interaction
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Multifactorial inheritance hypothesis for the etiology of congenital heart diseases. The genetic-environmental interaction

Circulation (New York, N.Y.), 1968-09, Vol.38 (3), p.604-617 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.38.3.604 ;PMID: 4876982

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12
Letter: Left circumflex coronary artery in SCS
Letter: Left circumflex coronary artery in SCS
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Letter: Left circumflex coronary artery in SCS

Circulation (New York, N.Y.), 1976-06, Vol.53 (6), p.1042-1043 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.53.6.1042 ;PMID: 1269119

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13
Hormones and the pathogenesis of congestive heart failure: vasopressin, aldosterone, and angiotensin II. Further evidence for renal-adrenal interaction from studies in hypertension and in cirrhosis
Hormones and the pathogenesis of congestive heart failure: vasopressin, aldosterone, and angiotensin II. Further evidence for renal-adrenal interaction from studies in hypertension and in cirrhosis
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Hormones and the pathogenesis of congestive heart failure: vasopressin, aldosterone, and angiotensin II. Further evidence for renal-adrenal interaction from studies in hypertension and in cirrhosis

Circulation (New York, N.Y.), 1962-06, Vol.25 (6), p.1015-1023 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.25.6.1015 ;PMID: 14462379

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14
Chromosomal abnormalities and congenital heart disease
Chromosomal abnormalities and congenital heart disease
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Chromosomal abnormalities and congenital heart disease

Circulation (New York, N.Y.), 1967-12, Vol.36 (6), p.886-905 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.CIR.36.6.886 ;PMID: 4228819

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