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Exercise is Associated With Impaired Left Ventricular Systolic Function in Patients With Lamin A/C Genotype
Exercise is Associated With Impaired Left Ventricular Systolic Function in Patients With Lamin A/C Genotype
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Exercise is Associated With Impaired Left Ventricular Systolic Function in Patients With Lamin A/C Genotype

Journal of the American Heart Association, 2020-01, Vol.9 (2), p.e012937-e012937 [Peer Reviewed Journal]

info:eu-repo/semantics/openAccess ;2020 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley. ;ISSN: 2047-9980 ;EISSN: 2047-9980 ;DOI: 10.1161/JAHA.119.012937 ;PMID: 31957533

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2
Left Ventricular Dysfunction in Arrhythmogenic Cardiomyopathy: Association With Exercise Exposure, Genetic Basis, and Prognosis
Left Ventricular Dysfunction in Arrhythmogenic Cardiomyopathy: Association With Exercise Exposure, Genetic Basis, and Prognosis
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Left Ventricular Dysfunction in Arrhythmogenic Cardiomyopathy: Association With Exercise Exposure, Genetic Basis, and Prognosis

Journal of the American Heart Association, 2021-04, Vol.10 (8), p.e018680-e018680 [Peer Reviewed Journal]

info:eu-repo/semantics/openAccess ;2021 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley. ;ISSN: 2047-9980 ;EISSN: 2047-9980 ;DOI: 10.1161/JAHA.120.018680 ;PMID: 33821670

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3
Plasma catecholamine levels in the acute and subacute stages of takotsubo syndrome: Results from the Stockholm myocardial infarction with normal coronaries 2 study
Plasma catecholamine levels in the acute and subacute stages of takotsubo syndrome: Results from the Stockholm myocardial infarction with normal coronaries 2 study
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Plasma catecholamine levels in the acute and subacute stages of takotsubo syndrome: Results from the Stockholm myocardial infarction with normal coronaries 2 study

Clinical cardiology (Mahwah, N.J.), 2021-11, Vol.44 (11), p.1567-1574 [Peer Reviewed Journal]

2021 The Authors. published by Wiley Periodicals LLC. ;2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;ISSN: 0160-9289 ;ISSN: 1932-8737 ;EISSN: 1932-8737 ;DOI: 10.1002/clc.23723 ;PMID: 34490898

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4
Respiratory chain signalling is essential for adaptive remodelling following cardiac ischaemia
Respiratory chain signalling is essential for adaptive remodelling following cardiac ischaemia
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Respiratory chain signalling is essential for adaptive remodelling following cardiac ischaemia

Journal of cellular and molecular medicine, 2020-03, Vol.24 (6), p.3534-3548 [Peer Reviewed Journal]

2020 The Authors. published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd ;2020 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.15043 ;PMID: 32040259

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5
QRS fragmentation is associated with increased risk of ventricular arrhythmias in high‐risk patients; Data from the SMASH 1 Study
QRS fragmentation is associated with increased risk of ventricular arrhythmias in high‐risk patients; Data from the SMASH 1 Study
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QRS fragmentation is associated with increased risk of ventricular arrhythmias in high‐risk patients; Data from the SMASH 1 Study

Annals of noninvasive electrocardiology, 2022-09, Vol.27 (5), p.e12985-n/a [Peer Reviewed Journal]

2022 The Authors. published by Wiley Periodicals LLC. ;COPYRIGHT 2022 John Wiley & Sons, Inc. ;2022. This article is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;ISSN: 1082-720X ;EISSN: 1542-474X ;DOI: 10.1111/anec.12985 ;PMID: 35839068

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6
Invasive haemodynamics at rest and exercise in cardiac amyloidosis
Invasive haemodynamics at rest and exercise in cardiac amyloidosis
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Invasive haemodynamics at rest and exercise in cardiac amyloidosis

ESC Heart Failure, 2024-04, Vol.11 (2), p.1263-1268 [Peer Reviewed Journal]

2023 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of European Society of Cardiology. ;2024. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;ISSN: 2055-5822 ;EISSN: 2055-5822 ;DOI: 10.1002/ehf2.14621 ;PMID: 38158705

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7
Syndecan-4 Protects the Heart From the Profibrotic Effects of Thrombin-Cleaved Osteopontin
Syndecan-4 Protects the Heart From the Profibrotic Effects of Thrombin-Cleaved Osteopontin
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Syndecan-4 Protects the Heart From the Profibrotic Effects of Thrombin-Cleaved Osteopontin

Journal of the American Heart Association, 2020-02, Vol.9 (3), p.e013518-e013518 [Peer Reviewed Journal]

info:eu-repo/semantics/openAccess ;2020 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley. ;ISSN: 2047-9980 ;EISSN: 2047-9980 ;DOI: 10.1161/JAHA.119.013518 ;PMID: 32000579

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