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1
Prevention of exercised induced cardiomyopathy following Pip-PMO treatment in dystrophic mdx mice
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Prevention of exercised induced cardiomyopathy following Pip-PMO treatment in dystrophic mdx mice

Scientific reports, 2015-03, Vol.5 (1), p.8986-8986, Article 8986 [Peer Reviewed Journal]

Copyright Nature Publishing Group Mar 2015 ;Copyright © 2015, Macmillan Publishers Limited. All rights reserved 2015 Macmillan Publishers Limited. All rights reserved ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep08986 ;PMID: 25758104

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2
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling
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Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling

Scientific reports, 2018-01, Vol.8 (1), p.1998-11, Article 1998 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-018-20114-9 ;PMID: 29386531

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3
Metabolomic insights in advanced cardiomyopathy of chronic chagasic and idiopathic patients that underwent heart transplant
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Metabolomic insights in advanced cardiomyopathy of chronic chagasic and idiopathic patients that underwent heart transplant

Scientific reports, 2024-04, Vol.14 (1), p.9810-9810 [Peer Reviewed Journal]

2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2024 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-024-53875-7 ;PMID: 38684702

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4
Silencing long non-coding RNA Kcnq1ot1 alleviates pyroptosis and fibrosis in diabetic cardiomyopathy
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Silencing long non-coding RNA Kcnq1ot1 alleviates pyroptosis and fibrosis in diabetic cardiomyopathy

Cell death & disease, 2018-09, Vol.9 (10), p.1000-13, Article 1000 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-018-1029-4 ;PMID: 30250027

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5
Analysis of cardiac magnetic resonance imaging in 36,000 individuals yields genetic insights into dilated cardiomyopathy
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Analysis of cardiac magnetic resonance imaging in 36,000 individuals yields genetic insights into dilated cardiomyopathy

Nature communications, 2020-05, Vol.11 (1), p.2254-2254, Article 2254 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-15823-7 ;PMID: 32382064

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6
WWP2 regulates pathological cardiac fibrosis by modulating SMAD2 signaling
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WWP2 regulates pathological cardiac fibrosis by modulating SMAD2 signaling

Nature communications, 2019-08, Vol.10 (1), p.3616-19, Article 3616 [Peer Reviewed Journal]

2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Attribution ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-019-11551-9 ;PMID: 31399586

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7
Striated muscle-specific base editing enables correction of mutations causing dilated cardiomyopathy
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Striated muscle-specific base editing enables correction of mutations causing dilated cardiomyopathy

Nature communications, 2023-06, Vol.14 (1), p.3714-3714, Article 3714 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-023-39352-1 ;PMID: 37349314

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8
Homogeneous 2D and 3D alignment of cardiomyocyte in dilated cardiomyopathy revealed by intravital heart imaging
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Homogeneous 2D and 3D alignment of cardiomyocyte in dilated cardiomyopathy revealed by intravital heart imaging

Scientific reports, 2021-07, Vol.11 (1), p.14698-14698, Article 14698 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-94100-z ;PMID: 34282197

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9
Investigation of diets associated with dilated cardiomyopathy in dogs using foodomics analysis
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Investigation of diets associated with dilated cardiomyopathy in dogs using foodomics analysis

Scientific reports, 2021-08, Vol.11 (1), p.15881-12, Article 15881 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-94464-2 ;PMID: 34354102

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10
Intergenerational inheritance of high fat diet-induced cardiac lipotoxicity in Drosophila
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Article
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Intergenerational inheritance of high fat diet-induced cardiac lipotoxicity in Drosophila

Nature communications, 2019-01, Vol.10 (1), p.193-14, Article 193 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-08128-3 ;PMID: 30643137

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11
High glucose induces Drp1-mediated mitochondrial fission via the Orai1 calcium channel to participate in diabetic cardiomyocyte hypertrophy
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High glucose induces Drp1-mediated mitochondrial fission via the Orai1 calcium channel to participate in diabetic cardiomyocyte hypertrophy

Cell death & disease, 2021-02, Vol.12 (2), p.216-216, Article 216 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-021-03502-4 ;PMID: 33637715

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12
Diagnostic and prognostic relevance of using large gene panels in the genetic testing of patients with dilated cardiomyopathy
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Diagnostic and prognostic relevance of using large gene panels in the genetic testing of patients with dilated cardiomyopathy

European journal of human genetics : EJHG, 2023-07, Vol.31 (7), p.776-783 [Peer Reviewed Journal]

2023. The Author(s), under exclusive licence to European Society of Human Genetics. ;The Author(s), under exclusive licence to European Society of Human Genetics 2023. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;ISSN: 1018-4813 ;EISSN: 1476-5438 ;DOI: 10.1038/s41431-023-01384-y ;PMID: 37198425

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13
Biallelic loss of LDB3 leads to a lethal pediatric dilated cardiomyopathy
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Article
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Biallelic loss of LDB3 leads to a lethal pediatric dilated cardiomyopathy

European journal of human genetics : EJHG, 2023-01, Vol.31 (1), p.97-104 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 1018-4813 ;EISSN: 1476-5438 ;DOI: 10.1038/s41431-022-01204-9 ;PMID: 36253531

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14
Phospholamban antisense oligonucleotides improve cardiac function in murine cardiomyopathy
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Article
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Phospholamban antisense oligonucleotides improve cardiac function in murine cardiomyopathy

Nature communications, 2021-08, Vol.12 (1), p.5180-5180, Article 5180 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-25439-0 ;PMID: 34462437

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15
Propionic acidemia as a cause of adult-onset dilated cardiomyopathy
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Article
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Propionic acidemia as a cause of adult-onset dilated cardiomyopathy

European journal of human genetics : EJHG, 2017-11, Vol.25 (11), p.1195-1201 [Peer Reviewed Journal]

Copyright Nature Publishing Group Nov 2017 ;Copyright © 2017 Macmillan Publishers Limited, part of Springer Nature. 2017 Macmillan Publishers Limited, part of Springer Nature. ;ISSN: 1018-4813 ;EISSN: 1476-5438 ;DOI: 10.1038/ejhg.2017.127 ;PMID: 28853722

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16
PRMT4 promotes ferroptosis to aggravate doxorubicin-induced cardiomyopathy via inhibition of the Nrf2/GPX4 pathway
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Article
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PRMT4 promotes ferroptosis to aggravate doxorubicin-induced cardiomyopathy via inhibition of the Nrf2/GPX4 pathway

Cell death and differentiation, 2022-10, Vol.29 (10), p.1982-1995 [Peer Reviewed Journal]

2022. The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare. ;The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare 2022. ;The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare 2022 ;ISSN: 1350-9047 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-022-00990-5 ;PMID: 35383293

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17
Gain-of-function cardiomyopathic mutations in RBM20 rewire splicing regulation and re-distribute ribonucleoprotein granules within processing bodies
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Article
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Gain-of-function cardiomyopathic mutations in RBM20 rewire splicing regulation and re-distribute ribonucleoprotein granules within processing bodies

Nature communications, 2021-11, Vol.12 (1), p.6324-6324, Article 6324 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-26623-y ;PMID: 34732726

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18
Correction of human phospholamban R14del mutation associated with cardiomyopathy using targeted nucleases and combination therapy
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Correction of human phospholamban R14del mutation associated with cardiomyopathy using targeted nucleases and combination therapy

Nature communications, 2015-04, Vol.6 (1), p.6955, Article 6955 [Peer Reviewed Journal]

Copyright Nature Publishing Group Apr 2015 ;Attribution ;Copyright © 2015, Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. 2015 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms7955 ;PMID: 25923014

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19
The K219T-Lamin mutation induces conduction defects through epigenetic inhibition of SCN5A in human cardiac laminopathy
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The K219T-Lamin mutation induces conduction defects through epigenetic inhibition of SCN5A in human cardiac laminopathy

Nature communications, 2019-05, Vol.10 (1), p.2267-2267, Article 2267 [Peer Reviewed Journal]

The Author(s) 2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-019-09929-w ;PMID: 31118417

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20
Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy
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Article
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Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy

Scientific reports, 2018-01, Vol.8 (1), p.724-9, Article 724 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-19138-4 ;PMID: 29335596

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