skip to main content
Language:
Search Limited to: Search Limited to: Resource type Show Results with: Show Results with: Search type Index

Results 21 - 40 of 346  for All Library Resources

previous page 1 Results 2 3 4 5 next page
Refined by: Journal Title: Cardiovascular Research remove subject: Humans remove
Result Number Material Type Add to My Shelf Action Record Details and Options
21
Mutations in MYH7 reduce the force generating capacity of sarcomeres in human familial hypertrophic cardiomyopathy
Material Type:
Article
Add to My Research

Mutations in MYH7 reduce the force generating capacity of sarcomeres in human familial hypertrophic cardiomyopathy

Cardiovascular research, 2013-08, Vol.99 (3), p.432-441 [Peer Reviewed Journal]

ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvt119 ;PMID: 23674513

Full text available

22
Sporadic dilated cardiomyopathy is often familial
Material Type:
Article
Add to My Research

Sporadic dilated cardiomyopathy is often familial

Cardiovascular research, 2022-07, Vol.118 (10), p.e69-e71 [Peer Reviewed Journal]

The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please email: journals.permissions@oup.com. 2022 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac075 ;PMID: 35726845

Full text available

23
Pivotal role of miR-448 in the development of ROS-induced cardiomyopathy
Material Type:
Article
Add to My Research

Pivotal role of miR-448 in the development of ROS-induced cardiomyopathy

Cardiovascular research, 2015-12, Vol.108 (3), p.324-334 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2015. For permissions please email: journals.permissions@oup.com. ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2015. For permissions please email: journals.permissions@oup.com. 2015 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvv238 ;PMID: 26503985

Full text available

24
Titin (TTN): from molecule to modifications, mechanics, and medical significance
Material Type:
Article
Add to My Research

Titin (TTN): from molecule to modifications, mechanics, and medical significance

Cardiovascular research, 2022-11, Vol.118 (14), p.2903-2918 [Peer Reviewed Journal]

The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. ;The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. 2021 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab328 ;PMID: 34662387

Full text available

25
Dyrk1a regulates the cardiomyocyte cell cycle via D-cyclin-dependent Rb/E2f-signalling
Material Type:
Article
Add to My Research

Dyrk1a regulates the cardiomyocyte cell cycle via D-cyclin-dependent Rb/E2f-signalling

Cardiovascular research, 2016-06, Vol.110 (3), p.381-394 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2016. For permissions please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvw074 ;PMID: 27056896

Full text available

26
Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice
Material Type:
Article
Add to My Research

Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice

Cardiovascular research, 2017-08, Vol.113 (10), p.1124-1136 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2017. For permissions please email: journals.permissions@oup.com. ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2017. For permissions, please email: journals.permissions@oup.com. 2017 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvx060 ;PMID: 28371863

Full text available

27
Energy metabolism in heart failure and remodelling
Material Type:
Article
Add to My Research

Energy metabolism in heart failure and remodelling

Cardiovascular research, 2009-02, Vol.81 (3), p.412-419 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org 2009 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvn301 ;PMID: 18987051

Full text available

28
Cell therapy in patients with heart failure: a comprehensive review and emerging concepts
Material Type:
Article
Add to My Research

Cell therapy in patients with heart failure: a comprehensive review and emerging concepts

Cardiovascular research, 2022-03, Vol.118 (4), p.951-976 [Peer Reviewed Journal]

The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. 2021 ;The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab135 ;PMID: 33871588

Full text available

29
Autophagy in cardiac myocyte homeostasis, aging, and pathology
Material Type:
Article
Add to My Research

Autophagy in cardiac myocyte homeostasis, aging, and pathology

Cardiovascular research, 2005-12, Vol.68 (3), p.355-365 [Peer Reviewed Journal]

2005 INIST-CNRS ;ISSN: 0008-6363 ;ISSN: 1755-3245 ;EISSN: 1755-3245 ;DOI: 10.1016/j.cardiores.2005.08.014 ;PMID: 16213475 ;CODEN: CVREAU

Full text available

30
Nuclear accumulation of androgen receptor in gender difference of dilated cardiomyopathy due to lamin A/C mutations
Material Type:
Article
Add to My Research

Nuclear accumulation of androgen receptor in gender difference of dilated cardiomyopathy due to lamin A/C mutations

Cardiovascular research, 2013-08, Vol.99 (3), p.382-394 [Peer Reviewed Journal]

ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvt106 ;PMID: 23631840

Full text available

31
Takotsubo syndrome: getting closer to its causes
Material Type:
Article
Add to My Research

Takotsubo syndrome: getting closer to its causes

Cardiovascular research, 2023-07, Vol.119 (7), p.1480-1494 [Peer Reviewed Journal]

The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2023 ;The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad053 ;PMID: 37183265

Full text available

32
Multilevel analyses of SCN5A mutations in arrhythmogenic right ventricular dysplasia/cardiomyopathy suggest non-canonical mechanisms for disease pathogenesis
Material Type:
Article
Add to My Research

Multilevel analyses of SCN5A mutations in arrhythmogenic right ventricular dysplasia/cardiomyopathy suggest non-canonical mechanisms for disease pathogenesis

Cardiovascular research, 2017-01, Vol.113 (1), p.102-111 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2016. For permissions, please email: journals.permissions@oup.com. ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2016. For permissions, please email: journals.permissions@oup.com. 2016 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvw234 ;PMID: 28069705

Full text available

33
Myofibrillar Ca(2+) sensitivity is uncoupled from troponin I phosphorylation in hypertrophic obstructive cardiomyopathy due to abnormal troponin T
Material Type:
Article
Add to My Research

Myofibrillar Ca(2+) sensitivity is uncoupled from troponin I phosphorylation in hypertrophic obstructive cardiomyopathy due to abnormal troponin T

Cardiovascular research, 2013-03, Vol.97 (3), p.500 [Peer Reviewed Journal]

EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvs322 ;PMID: 23097574

Full text available

34
Arrhythmogenic cardiomyopathy: pathology, genetics, and concepts in pathogenesis
Material Type:
Article
Add to My Research

Arrhythmogenic cardiomyopathy: pathology, genetics, and concepts in pathogenesis

Cardiovascular research, 2017-10, Vol.113 (12), p.1521-1531 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2017. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvx150 ;PMID: 28957532

Full text available

35
Pathophysiology of the heart in Chagas' disease: current status and new developments
Material Type:
Article
Add to My Research

Pathophysiology of the heart in Chagas' disease: current status and new developments

Cardiovascular research, 2003-10, Vol.60 (1), p.96-107 [Peer Reviewed Journal]

2004 INIST-CNRS ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1016/S0008-6363(03)00361-4 ;PMID: 14522411 ;CODEN: CVREAU

Full text available

36
Impaired binding of ZASP/Cypher with phosphoglucomutase 1 is associated with dilated cardiomyopathy
Material Type:
Article
Add to My Research

Impaired binding of ZASP/Cypher with phosphoglucomutase 1 is associated with dilated cardiomyopathy

Cardiovascular research, 2009-07, Vol.83 (1), p.80-88 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2009. For permissions please email: journals.permissions@oxfordjournals.org. 2009 ;2009 INIST-CNRS ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvp119 ;PMID: 19377068 ;CODEN: CVREAU

Full text available

37
Association of DJ-1/PTEN/AKT- and ASK1/p38-mediated cell signalling with ischaemic cardiomyopathy
Material Type:
Article
Add to My Research

Association of DJ-1/PTEN/AKT- and ASK1/p38-mediated cell signalling with ischaemic cardiomyopathy

Cardiovascular research, 2013-01, Vol.97 (1), p.66-76 [Peer Reviewed Journal]

ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvs302 ;PMID: 23015639

Full text available

38
Elevated p53 expression is associated with dysregulation of the ubiquitin-proteasome system in dilated cardiomyopathy
Material Type:
Article
Add to My Research

Elevated p53 expression is associated with dysregulation of the ubiquitin-proteasome system in dilated cardiomyopathy

Cardiovascular research, 2008-08, Vol.79 (3), p.472-480 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org 2008 ;2008 INIST-CNRS ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvn083 ;PMID: 18375498 ;CODEN: CVREAU

Full text available

39
The ubiquitin-proteasome system and nonsense-mediated mRNA decay in hypertrophic cardiomyopathy
Material Type:
Article
Add to My Research

The ubiquitin-proteasome system and nonsense-mediated mRNA decay in hypertrophic cardiomyopathy

Cardiovascular research, 2010-01, Vol.85 (2), p.330-338 [Peer Reviewed Journal]

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2009. For permissions please email: journals.permissions@oxfordjournals.org. 2010 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvp247 ;PMID: 19617224

Full text available

40
Scientists on the spot: Relaxing the heart in hypertrophic cardiomyopathy
Material Type:
Article
Add to My Research

Scientists on the spot: Relaxing the heart in hypertrophic cardiomyopathy

Cardiovascular research, 2023-05, Vol.119 (5), p.e131-e132 [Peer Reviewed Journal]

The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. 2023 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad049 ;PMID: 37102235

Full text available

Results 21 - 40 of 346  for All Library Resources

previous page 1 Results 2 3 4 5 next page

Searching Remote Databases, Please Wait