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1
Anatomic Versus Physiologic Assessment of Coronary Artery Disease: Role of Coronary Flow Reserve, Fractional Flow Reserve, and Positron Emission Tomography Imaging in Revascularization Decision-Making
Anatomic Versus Physiologic Assessment of Coronary Artery Disease: Role of Coronary Flow Reserve, Fractional Flow Reserve, and Positron Emission Tomography Imaging in Revascularization Decision-Making
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Anatomic Versus Physiologic Assessment of Coronary Artery Disease: Role of Coronary Flow Reserve, Fractional Flow Reserve, and Positron Emission Tomography Imaging in Revascularization Decision-Making

Journal of the American College of Cardiology, 2013-10, Vol.62 (18), p.1639-1653 [Tạp chí có phản biện]

2014 INIST-CNRS ;Copyright © 2013 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2013.07.076 ;PMID: 23954338 ;CODEN: JACCDI

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2
Targeted Disruption of JCAD (Junctional Protein Associated With Coronary Artery Disease)/KIAA1462, a Coronary Artery Disease–Associated Gene Product, Inhibits Angiogenic Processes In Vitro and In Vivo
Targeted Disruption of JCAD (Junctional Protein Associated With Coronary Artery Disease)/KIAA1462, a Coronary Artery Disease–Associated Gene Product, Inhibits Angiogenic Processes In Vitro and In Vivo
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Targeted Disruption of JCAD (Junctional Protein Associated With Coronary Artery Disease)/KIAA1462, a Coronary Artery Disease–Associated Gene Product, Inhibits Angiogenic Processes In Vitro and In Vivo

Arteriosclerosis, thrombosis, and vascular biology, 2017-09, Vol.37 (9), p.1667-1673 [Tạp chí có phản biện]

2017 American Heart Association, Inc. ;ISSN: 1079-5642 ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.117.309721 ;PMID: 28705794

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3
Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease
Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease
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Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease

The Journal of clinical investigation, 2011-07, Vol.121 (7), p.2693-2708 [Tạp chí có phản biện]

COPYRIGHT 2011 American Society for Clinical Investigation ;COPYRIGHT 2011 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Jul 2011 ;Copyright © 2011, American Society for Clinical Investigation 2011 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI42946 ;PMID: 21701070

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4
Central Obesity and Survival in Subjects With Coronary Artery Disease: A Systematic Review of the Literature and Collaborative Analysis With Individual Subject Data
Central Obesity and Survival in Subjects With Coronary Artery Disease: A Systematic Review of the Literature and Collaborative Analysis With Individual Subject Data
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Central Obesity and Survival in Subjects With Coronary Artery Disease: A Systematic Review of the Literature and Collaborative Analysis With Individual Subject Data

Journal of the American College of Cardiology, 2011-05, Vol.57 (19), p.1877-1886 [Tạp chí có phản biện]

2015 INIST-CNRS ;Copyright © 2011 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited May 10, 2011 ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2010.11.058 ;PMID: 21545944 ;CODEN: JACCDI

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5
Severe Aortic Stenosis and Coronary Artery Disease-Implications for Management in the Transcatheter Aortic Valve Replacement Era: A Comprehensive Review
Severe Aortic Stenosis and Coronary Artery Disease-Implications for Management in the Transcatheter Aortic Valve Replacement Era: A Comprehensive Review
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Severe Aortic Stenosis and Coronary Artery Disease-Implications for Management in the Transcatheter Aortic Valve Replacement Era: A Comprehensive Review

Journal of the American College of Cardiology, 2013-07, Vol.62 (1), p.1-10 [Tạp chí có phản biện]

2014 INIST-CNRS ;Copyright © 2013 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2013.01.096 ;PMID: 23644089 ;CODEN: JACCDI

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6
Defects of High-Density Lipoproteins in Coronary Artery Disease Caused by Low Sphingosine-1-Phosphate Content: Correction by Sphingosine-1-Phosphate-Loading
Defects of High-Density Lipoproteins in Coronary Artery Disease Caused by Low Sphingosine-1-Phosphate Content: Correction by Sphingosine-1-Phosphate-Loading
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Defects of High-Density Lipoproteins in Coronary Artery Disease Caused by Low Sphingosine-1-Phosphate Content: Correction by Sphingosine-1-Phosphate-Loading

Journal of the American College of Cardiology, 2015-09, Vol.66 (13), p.1470-1485 [Tạp chí có phản biện]

Copyright © 2015 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2015.07.057 ;PMID: 26403344

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7
Triglyceride-Rich Lipoproteins and Coronary Artery Disease Risk: New Insights From Human Genetics
Triglyceride-Rich Lipoproteins and Coronary Artery Disease Risk: New Insights From Human Genetics
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Triglyceride-Rich Lipoproteins and Coronary Artery Disease Risk: New Insights From Human Genetics

Arteriosclerosis, thrombosis, and vascular biology, 2015-02, Vol.35 (2), p.e3-e9 [Tạp chí có phản biện]

2015 American Heart Association, Inc. ;ISSN: 1079-5642 ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.114.305172 ;PMID: 25573854

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8
Deficiency of MMP1a (Matrix Metalloprotease 1a) Collagenase Suppresses Development of Atherosclerosis in Mice: Translational Implications for Human Coronary Artery Disease
Deficiency of MMP1a (Matrix Metalloprotease 1a) Collagenase Suppresses Development of Atherosclerosis in Mice: Translational Implications for Human Coronary Artery Disease
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Deficiency of MMP1a (Matrix Metalloprotease 1a) Collagenase Suppresses Development of Atherosclerosis in Mice: Translational Implications for Human Coronary Artery Disease

Arteriosclerosis, thrombosis, and vascular biology, 2021-05, Vol.41 (5), p.e265-e279 [Tạp chí có phản biện]

ISSN: 1079-5642 ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.120.315837 ;PMID: 33761760

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9
Overexpression of tissue-nonspecific alkaline phosphatase (TNAP) in endothelial cells accelerates coronary artery disease in a mouse model of familial hypercholesterolemia
Overexpression of tissue-nonspecific alkaline phosphatase (TNAP) in endothelial cells accelerates coronary artery disease in a mouse model of familial hypercholesterolemia
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Overexpression of tissue-nonspecific alkaline phosphatase (TNAP) in endothelial cells accelerates coronary artery disease in a mouse model of familial hypercholesterolemia

PloS one, 2017-10, Vol.12 (10), p.e0186426-e0186426 [Tạp chí có phản biện]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Romanelli et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Romanelli et al 2017 Romanelli et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0186426 ;PMID: 29023576

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10
Lipocalin (LCN) 2 Mediates Pro-Atherosclerotic Processes and Is Elevated in Patients with Coronary Artery Disease
Lipocalin (LCN) 2 Mediates Pro-Atherosclerotic Processes and Is Elevated in Patients with Coronary Artery Disease
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Lipocalin (LCN) 2 Mediates Pro-Atherosclerotic Processes and Is Elevated in Patients with Coronary Artery Disease

PloS one, 2015-09, Vol.10 (9), p.e0137924-e0137924 [Tạp chí có phản biện]

COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Oberoi et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Oberoi et al 2015 Oberoi et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0137924 ;PMID: 26367277

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