Result Number | Material Type | Add to My Shelf Action | Record Details and Options |
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1 |
Material Type: Article
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Specific deletion of NaV1.1 sodium channels in inhibitory interneurons causes seizures and premature death in a mouse model of Dravet syndromeProceedings of the National Academy of Sciences - PNAS, 2012-09, Vol.109 (36), p.14646-14651 [Peer Reviewed Journal]copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Sep 4, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1211591109 ;PMID: 22908258Full text available |
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2 |
Material Type: Article
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Temperature- and Age-Dependent Seizures in a Mouse Model of Severe Myoclonic Epilepsy in InfancyProceedings of the National Academy of Sciences - PNAS, 2009-03, Vol.106 (10), p.3994-3999 [Peer Reviewed Journal]Copyright 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Mar 10, 2009 ;2009 by The National Academy of Sciences of the USA ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.0813330106 ;PMID: 19234123Full text available |
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3 |
Material Type: Article
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Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancyNature neuroscience, 2006-09, Vol.9 (9), p.1142-1149 [Peer Reviewed Journal]COPYRIGHT 2006 Nature Publishing Group ;Copyright Nature Publishing Group Sep 2006 ;ISSN: 1097-6256 ;EISSN: 1546-1726 ;DOI: 10.1038/nn1754 ;PMID: 16921370 ;CODEN: NANEFNFull text available |
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4 |
Material Type: Article
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Reduced Sodium Current in Purkinje Neurons from NaV1.1 Mutant Mice: Implications for Ataxia in Severe Myoclonic Epilepsy in InfancyThe Journal of neuroscience, 2007-10, Vol.27 (41), p.11065-11074 [Peer Reviewed Journal]Copyright © 2007 Society for Neuroscience 0270-6474/07/2711065-10$15.00/0 2007 ;ISSN: 0270-6474 ;EISSN: 1529-2401 ;DOI: 10.1523/JNEUROSCI.2162-07.2007 ;PMID: 17928448Full text available |
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5 |
Material Type: Article
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Genetic background modulates impaired excitability of inhibitory neurons in a mouse model of Dravet syndromeNeurobiology of disease, 2015-01, Vol.73, p.106-117 [Peer Reviewed Journal]EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2014.09.017Digital Resources/Online E-Resources |
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6 |
Material Type: Article
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NaV1.6 and NaV1.7 channels are major endogenous voltage-gated sodium channels in ND7/23 cellsPloS one, 2019-08, Vol.14 (8), p.e0221156-e0221156 [Peer Reviewed Journal]2019 Lee et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Lee et al 2019 Lee et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0221156 ;PMID: 31419255Full text available |
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7 |
Material Type: Article
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Overview of molecular relationships in the voltage-gated ion channel superfamilyPharmacological reviews, 2005-12, Vol.57 (4), p.387-395 [Peer Reviewed Journal]ISSN: 0031-6997 ;EISSN: 1521-0081 ;DOI: 10.1124/pr.57.4.13 ;PMID: 16382097Full text available |
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8 |
Material Type: Article
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A BAC transgenic mouse model reveals neuron subtype-specific effects of a Generalized Epilepsy with Febrile Seizures Plus (GEFS+) mutationNeurobiology of disease, 2009-07, Vol.35 (1), p.91-102 [Peer Reviewed Journal]EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2009.04.007Digital Resources/Online E-Resources |
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9 |
Material Type: Article
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Functional Roles of a C-terminal Signaling Complex of CaV1 Channels and A-kinase Anchoring Protein 15 in Brain NeuronsThe Journal of biological chemistry, 2011-04, Vol.286 (14), p.12627-12639 [Peer Reviewed Journal]2011 © 2011 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2011 by The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;ISSN: 1083-351X ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M110.175257 ;PMID: 21224388Full text available |
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10 |
Material Type: Article
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Cytotoxic effects of one‐step self‐etching adhesives on an odontoblast cell lineScanning, 2016-01, Vol.38 (1), p.36-42 [Peer Reviewed Journal]Wiley Periodicals, Inc. ;ISSN: 0161-0457 ;EISSN: 1932-8745 ;DOI: 10.1002/sca.21236 ;PMID: 26186405Full text available |
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11 |
Material Type: Article
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Overview of the voltage-gated sodium channel familyGenome biology, 2003, Vol.4 (3), p.207-207 [Peer Reviewed Journal]Copyright © 2003 BioMed Central Ltd ;ISSN: 1465-6906 ;EISSN: 1474-760X ;EISSN: 1465-6914 ;DOI: 10.1186/gb-2003-4-3-207 ;PMID: 12620097Full text available |
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12 |
Material Type: Article
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Autistic-like behaviour in Scn1a+/- mice and rescue by enhanced GABA-mediated neurotransmissionNature (London), 2012-09, Vol.489 (7416), p.385-390 [Peer Reviewed Journal]ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature11356 ;PMID: 22914087Full text available |
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13 |
Material Type: Article
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Sudden unexpected death in a mouse model of Dravet syndromeThe Journal of clinical investigation, 2013-04, Vol.123 (4), p.1798-1808 [Peer Reviewed Journal]COPYRIGHT 2013 American Society for Clinical Investigation ;COPYRIGHT 2013 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Apr 2013 ;Copyright © 2013, American Society for Clinical Investigation 2013 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI66220 ;PMID: 23524966Full text available |
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14 |
Material Type: Article
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Too many roads not takenNature (London), 2011-02, Vol.470 (7333), p.163-165 [Peer Reviewed Journal]Copyright Nature Publishing Group Feb 10, 2011 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/470163a ;PMID: 21307913 ;CODEN: NATUASFull text available |
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15 |
Material Type: Article
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Di-(2 ethylhexyl) phthalate and flutamide alter gene expression in the testis of immature male ratsReproductive biology and endocrinology, 2009-09, Vol.7 (1), p.104-104, Article 104 [Peer Reviewed Journal]Copyright © 2009 Vo et al; licensee BioMed Central Ltd. 2009 Vo et al; licensee BioMed Central Ltd. ;ISSN: 1477-7827 ;EISSN: 1477-7827 ;DOI: 10.1186/1477-7827-7-104 ;PMID: 19781091Full text available |
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16 |
Material Type: Article
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Role of the C-terminal Domain in Inactivation of Brain and Cardiac Sodium ChannelsProceedings of the National Academy of Sciences - PNAS, 2001-12, Vol.98 (26), p.15348-15353 [Peer Reviewed Journal]Copyright 1993-2001 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Dec 18, 2001 ;Copyright © 2001, The National Academy of Sciences 2001 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.211563298 ;PMID: 11742069Full text available |
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17 |
Material Type: Article
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NaV1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythmsProceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (6), p.E368-E377 [Peer Reviewed Journal]ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1115729109 ;PMID: 22223655Full text available |
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18 |
Material Type: Article
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Deletion of the Distal C Terminus of CaV1.2 Channels Leads to Loss of β-Adrenergic Regulation and Heart Failure in VivoThe Journal of biological chemistry, 2011-04, Vol.286 (14), p.12617-12626 [Peer Reviewed Journal]2011 © 2011 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2011 by The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;ISSN: 1083-351X ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M110.175307 ;PMID: 21216955Full text available |
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19 |
Material Type: Article
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Na(V)1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythmsProceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (6), p.E368-E377 [Peer Reviewed Journal]EISSN: 1091-6490 ;DOI: 10.1073/pnas.1115729109 ;PMID: 22223655Full text available |
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20 |
Material Type: Article
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The voltage-gated sodium channel Scn8a is a genetic modifier of severe myoclonic epilepsy of infancyHuman molecular genetics, 2007-12, Vol.16 (23), p.2892-2899 [Peer Reviewed Journal]The Author 2007. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org 2007 ;2008 INIST-CNRS ;ISSN: 0964-6906 ;EISSN: 1460-2083 ;DOI: 10.1093/hmg/ddm248 ;PMID: 17881658 ;CODEN: HNGEE5Full text available |