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1
Specific deletion of NaV1.1 sodium channels in inhibitory interneurons causes seizures and premature death in a mouse model of Dravet syndrome
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Specific deletion of NaV1.1 sodium channels in inhibitory interneurons causes seizures and premature death in a mouse model of Dravet syndrome

Proceedings of the National Academy of Sciences - PNAS, 2012-09, Vol.109 (36), p.14646-14651 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Sep 4, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1211591109 ;PMID: 22908258

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2
Temperature- and Age-Dependent Seizures in a Mouse Model of Severe Myoclonic Epilepsy in Infancy
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Temperature- and Age-Dependent Seizures in a Mouse Model of Severe Myoclonic Epilepsy in Infancy

Proceedings of the National Academy of Sciences - PNAS, 2009-03, Vol.106 (10), p.3994-3999 [Peer Reviewed Journal]

Copyright 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Mar 10, 2009 ;2009 by The National Academy of Sciences of the USA ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.0813330106 ;PMID: 19234123

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3
Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancy
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Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancy

Nature neuroscience, 2006-09, Vol.9 (9), p.1142-1149 [Peer Reviewed Journal]

COPYRIGHT 2006 Nature Publishing Group ;Copyright Nature Publishing Group Sep 2006 ;ISSN: 1097-6256 ;EISSN: 1546-1726 ;DOI: 10.1038/nn1754 ;PMID: 16921370 ;CODEN: NANEFN

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4
Reduced Sodium Current in Purkinje Neurons from NaV1.1 Mutant Mice: Implications for Ataxia in Severe Myoclonic Epilepsy in Infancy
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Reduced Sodium Current in Purkinje Neurons from NaV1.1 Mutant Mice: Implications for Ataxia in Severe Myoclonic Epilepsy in Infancy

The Journal of neuroscience, 2007-10, Vol.27 (41), p.11065-11074 [Peer Reviewed Journal]

Copyright © 2007 Society for Neuroscience 0270-6474/07/2711065-10$15.00/0 2007 ;ISSN: 0270-6474 ;EISSN: 1529-2401 ;DOI: 10.1523/JNEUROSCI.2162-07.2007 ;PMID: 17928448

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5
Genetic background modulates impaired excitability of inhibitory neurons in a mouse model of Dravet syndrome
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Genetic background modulates impaired excitability of inhibitory neurons in a mouse model of Dravet syndrome

Neurobiology of disease, 2015-01, Vol.73, p.106-117 [Peer Reviewed Journal]

EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2014.09.017

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6
NaV1.6 and NaV1.7 channels are major endogenous voltage-gated sodium channels in ND7/23 cells
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NaV1.6 and NaV1.7 channels are major endogenous voltage-gated sodium channels in ND7/23 cells

PloS one, 2019-08, Vol.14 (8), p.e0221156-e0221156 [Peer Reviewed Journal]

2019 Lee et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Lee et al 2019 Lee et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0221156 ;PMID: 31419255

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7
Overview of molecular relationships in the voltage-gated ion channel superfamily
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Overview of molecular relationships in the voltage-gated ion channel superfamily

Pharmacological reviews, 2005-12, Vol.57 (4), p.387-395 [Peer Reviewed Journal]

ISSN: 0031-6997 ;EISSN: 1521-0081 ;DOI: 10.1124/pr.57.4.13 ;PMID: 16382097

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8
A BAC transgenic mouse model reveals neuron subtype-specific effects of a Generalized Epilepsy with Febrile Seizures Plus (GEFS+) mutation
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A BAC transgenic mouse model reveals neuron subtype-specific effects of a Generalized Epilepsy with Febrile Seizures Plus (GEFS+) mutation

Neurobiology of disease, 2009-07, Vol.35 (1), p.91-102 [Peer Reviewed Journal]

EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2009.04.007

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9
Functional Roles of a C-terminal Signaling Complex of CaV1 Channels and A-kinase Anchoring Protein 15 in Brain Neurons
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Functional Roles of a C-terminal Signaling Complex of CaV1 Channels and A-kinase Anchoring Protein 15 in Brain Neurons

The Journal of biological chemistry, 2011-04, Vol.286 (14), p.12627-12639 [Peer Reviewed Journal]

2011 © 2011 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2011 by The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;ISSN: 1083-351X ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M110.175257 ;PMID: 21224388

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10
Cytotoxic effects of one‐step self‐etching adhesives on an odontoblast cell line
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Cytotoxic effects of one‐step self‐etching adhesives on an odontoblast cell line

Scanning, 2016-01, Vol.38 (1), p.36-42 [Peer Reviewed Journal]

Wiley Periodicals, Inc. ;ISSN: 0161-0457 ;EISSN: 1932-8745 ;DOI: 10.1002/sca.21236 ;PMID: 26186405

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11
Overview of the voltage-gated sodium channel family
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Article
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Overview of the voltage-gated sodium channel family

Genome biology, 2003, Vol.4 (3), p.207-207 [Peer Reviewed Journal]

Copyright © 2003 BioMed Central Ltd ;ISSN: 1465-6906 ;EISSN: 1474-760X ;EISSN: 1465-6914 ;DOI: 10.1186/gb-2003-4-3-207 ;PMID: 12620097

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12
Autistic-like behaviour in Scn1a+/- mice and rescue by enhanced GABA-mediated neurotransmission
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Autistic-like behaviour in Scn1a+/- mice and rescue by enhanced GABA-mediated neurotransmission

Nature (London), 2012-09, Vol.489 (7416), p.385-390 [Peer Reviewed Journal]

ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature11356 ;PMID: 22914087

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13
Sudden unexpected death in a mouse model of Dravet syndrome
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Sudden unexpected death in a mouse model of Dravet syndrome

The Journal of clinical investigation, 2013-04, Vol.123 (4), p.1798-1808 [Peer Reviewed Journal]

COPYRIGHT 2013 American Society for Clinical Investigation ;COPYRIGHT 2013 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Apr 2013 ;Copyright © 2013, American Society for Clinical Investigation 2013 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI66220 ;PMID: 23524966

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14
Too many roads not taken
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Too many roads not taken

Nature (London), 2011-02, Vol.470 (7333), p.163-165 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 10, 2011 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/470163a ;PMID: 21307913 ;CODEN: NATUAS

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15
Di-(2 ethylhexyl) phthalate and flutamide alter gene expression in the testis of immature male rats
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Di-(2 ethylhexyl) phthalate and flutamide alter gene expression in the testis of immature male rats

Reproductive biology and endocrinology, 2009-09, Vol.7 (1), p.104-104, Article 104 [Peer Reviewed Journal]

Copyright © 2009 Vo et al; licensee BioMed Central Ltd. 2009 Vo et al; licensee BioMed Central Ltd. ;ISSN: 1477-7827 ;EISSN: 1477-7827 ;DOI: 10.1186/1477-7827-7-104 ;PMID: 19781091

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16
Role of the C-terminal Domain in Inactivation of Brain and Cardiac Sodium Channels
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Role of the C-terminal Domain in Inactivation of Brain and Cardiac Sodium Channels

Proceedings of the National Academy of Sciences - PNAS, 2001-12, Vol.98 (26), p.15348-15353 [Peer Reviewed Journal]

Copyright 1993-2001 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Dec 18, 2001 ;Copyright © 2001, The National Academy of Sciences 2001 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.211563298 ;PMID: 11742069

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17
NaV1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythms
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NaV1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythms

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (6), p.E368-E377 [Peer Reviewed Journal]

ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1115729109 ;PMID: 22223655

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18
Deletion of the Distal C Terminus of CaV1.2 Channels Leads to Loss of β-Adrenergic Regulation and Heart Failure in Vivo
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Deletion of the Distal C Terminus of CaV1.2 Channels Leads to Loss of β-Adrenergic Regulation and Heart Failure in Vivo

The Journal of biological chemistry, 2011-04, Vol.286 (14), p.12617-12626 [Peer Reviewed Journal]

2011 © 2011 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2011 by The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;ISSN: 1083-351X ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M110.175307 ;PMID: 21216955

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19
Na(V)1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythms
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Na(V)1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythms

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (6), p.E368-E377 [Peer Reviewed Journal]

EISSN: 1091-6490 ;DOI: 10.1073/pnas.1115729109 ;PMID: 22223655

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20
The voltage-gated sodium channel Scn8a is a genetic modifier of severe myoclonic epilepsy of infancy
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The voltage-gated sodium channel Scn8a is a genetic modifier of severe myoclonic epilepsy of infancy

Human molecular genetics, 2007-12, Vol.16 (23), p.2892-2899 [Peer Reviewed Journal]

The Author 2007. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org 2007 ;2008 INIST-CNRS ;ISSN: 0964-6906 ;EISSN: 1460-2083 ;DOI: 10.1093/hmg/ddm248 ;PMID: 17881658 ;CODEN: HNGEE5

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