Result Number | Material Type | Add to My Shelf Action | Record Details and Options |
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1 |
Material Type: Article
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Decoding the vasoregulatory activities of bile acid-activated receptors in systemic and portal circulation: role of gaseous mediatorsAmerican journal of physiology. Heart and circulatory physiology, 2017-01, Vol.312 (1), p.H21-H32 [Peer Reviewed Journal]Copyright © 2017 the American Physiological Society. ;Copyright American Physiological Society Jan 1, 2017 ;ISSN: 0363-6135 ;EISSN: 1522-1539 ;DOI: 10.1152/ajpheart.00577.2016 ;PMID: 27765751 ;CODEN: AJPPDIFull text available |
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2 |
Material Type: Article
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Cystathionine γ-lyase, a H2S-generating enzyme, is a GPBAR1-regulated gene and contributes to vasodilation caused by secondary bile acidsAmerican journal of physiology. Heart and circulatory physiology, 2015-07, Vol.309 (1), p.H114-H126 [Peer Reviewed Journal]Copyright © 2015 the American Physiological Society. ;EISSN: 1522-1539 ;DOI: 10.1152/ajpheart.00087.2015 ;PMID: 25934094Full text available |
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3 |
Material Type: Article
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Cystathionine γ-lyase, a H 2 S-generating enzyme, is a GPBAR1-regulated gene and contributes to vasodilation caused by secondary bile acidsAmerican journal of physiology. Heart and circulatory physiology, 2015-07, Vol.309 (1), p.H114-H126 [Peer Reviewed Journal]ISSN: 0363-6135 ;EISSN: 1522-1539 ;DOI: 10.1152/ajpheart.00087.2015Full text available |
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4 |
Material Type: Article
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Cystathionine [gamma]-lyase, a H^sub 2^S-generating enzyme, is a GPBAR1-regulated gene and contributes to vasodilation caused by secondary bile acidsAmerican journal of physiology. Heart and circulatory physiology, 2015-07, Vol.309 (1), p.H114 [Peer Reviewed Journal]ISSN: 0363-6135 ;EISSN: 1522-1539 ;CODEN: AJPPDIFull text available |
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5 |
Material Type: Article
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Cystathionine gamma -lyase, a H sub( 2)S-generating enzyme, is a GPBAR1-regulated gene and contributes to vasodilation caused by secondary bile acidsAmerican journal of physiology. Heart and circulatory physiology, 2015-07, Vol.309 (1), p.H114-H114 [Peer Reviewed Journal]ISSN: 0363-6135 ;EISSN: 1522-1539Full text available |