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1
Dynamic Edematous Response of the Human Heart to Myocardial Infarction: Implications for Assessing Myocardial Area at Risk and Salvage
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Dynamic Edematous Response of the Human Heart to Myocardial Infarction: Implications for Assessing Myocardial Area at Risk and Salvage

Circulation (New York, N.Y.), 2017-10, Vol.136 (14), p.1288-1300 [Peer Reviewed Journal]

2017 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2017 The Authors. ;2017 The Authors. 2017 ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.116.025582 ;PMID: 28687712

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2
Selective Stimulation of Cardiac Lymphangiogenesis Reduces Myocardial Edema and Fibrosis Leading to Improved Cardiac Function Following Myocardial Infarction
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Selective Stimulation of Cardiac Lymphangiogenesis Reduces Myocardial Edema and Fibrosis Leading to Improved Cardiac Function Following Myocardial Infarction

Circulation (New York, N.Y.), 2016-04, Vol.133 (15), p.1484-1497 [Peer Reviewed Journal]

2016 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2016 American Heart Association, Inc. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.115.020143 ;PMID: 26933083

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3
Effective Treatment of Edema and Endothelial Barrier Dysfunction With Imatinib
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Effective Treatment of Edema and Endothelial Barrier Dysfunction With Imatinib

Circulation (New York, N.Y.), 2012-12, Vol.126 (23), p.2728-2738 [Peer Reviewed Journal]

2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.112.134304 ;PMID: 23099479 ;CODEN: CIRCAZ

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4
Vascular Endothelial Adrenomedullin-RAMP2 System Is Essential for Vascular Integrity and Organ Homeostasis
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Vascular Endothelial Adrenomedullin-RAMP2 System Is Essential for Vascular Integrity and Organ Homeostasis

Circulation (New York, N.Y.), 2013-02, Vol.127 (7), p.842-853 [Peer Reviewed Journal]

2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.112.000756 ;PMID: 23355623 ;CODEN: CIRCAZ

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5
Targeting Mannose-Binding Lectin Confers Long-Lasting Protection With a Surprisingly Wide Therapeutic Window in Cerebral Ischemia
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Targeting Mannose-Binding Lectin Confers Long-Lasting Protection With a Surprisingly Wide Therapeutic Window in Cerebral Ischemia

Circulation (New York, N.Y.), 2012-09, Vol.126 (12), p.1484-1494 [Peer Reviewed Journal]

2015 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.112.103051 ;PMID: 22879370 ;CODEN: CIRCAZ

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6
Deletion of Cardiomyocyte Mineralocorticoid Receptor Ameliorates Adverse Remodeling After Myocardial Infarction
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Deletion of Cardiomyocyte Mineralocorticoid Receptor Ameliorates Adverse Remodeling After Myocardial Infarction

Circulation (New York, N.Y.), 2011-02, Vol.123 (4), p.400-408 [Peer Reviewed Journal]

2015 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.110.983023 ;PMID: 21242479 ;CODEN: CIRCAZ

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7
Retrospective determination of the area at risk for reperfused acute myocardial infarction with T2-weighted cardiac magnetic resonance imaging : Histopathological and displacement encoding with stimulated echoes (DENSE) functional validations
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Retrospective determination of the area at risk for reperfused acute myocardial infarction with T2-weighted cardiac magnetic resonance imaging : Histopathological and displacement encoding with stimulated echoes (DENSE) functional validations

Circulation (New York, N.Y.), 2006-04, Vol.113 (15), p.1865-1870 [Peer Reviewed Journal]

2006 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.105.576025 ;PMID: 16606793 ;CODEN: CIRCAZ

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8
Endothelial p110γPI3K Mediates Endothelial Regeneration and Vascular Repair After Inflammatory Vascular Injury
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Endothelial p110γPI3K Mediates Endothelial Regeneration and Vascular Repair After Inflammatory Vascular Injury

Circulation (New York, N.Y.), 2016-03, Vol.133 (11), p.1093-1103 [Peer Reviewed Journal]

2016 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2016 American Heart Association, Inc. ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.115.020918 ;PMID: 26839042

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9
CD4+ T Cells Promote the Transition From Hypertrophy to Heart Failure During Chronic Pressure Overload
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CD4+ T Cells Promote the Transition From Hypertrophy to Heart Failure During Chronic Pressure Overload

Circulation (New York, N.Y.), 2014-05, Vol.129 (21), p.2111-2124 [Peer Reviewed Journal]

2014 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2015 INIST-CNRS ;2014 American Heart Association, Inc. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.113.007101 ;PMID: 24657994 ;CODEN: CIRCAZ

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10
Protective role of angiopoietin-1 in endotoxic shock
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Protective role of angiopoietin-1 in endotoxic shock

Circulation (New York, N.Y.), 2005-01, Vol.111 (1), p.97-105 [Peer Reviewed Journal]

2005 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/01.cir.0000151287.08202.8e ;PMID: 15611372 ;CODEN: CIRCAZ

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11
Protective Effects of Ticagrelor on Myocardial Injury After Infarction
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Protective Effects of Ticagrelor on Myocardial Injury After Infarction

Circulation (New York, N.Y.), 2016-11, Vol.134 (22), p.1708-1719 [Peer Reviewed Journal]

2016 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2016 American Heart Association, Inc. ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.116.024014 ;PMID: 27789556

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12
Therapeutic Inhibition of miR-208a Improves Cardiac Function and Survival During Heart Failure
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Therapeutic Inhibition of miR-208a Improves Cardiac Function and Survival During Heart Failure

Circulation (New York, N.Y.), 2011-10, Vol.124 (14), p.1537-1547 [Peer Reviewed Journal]

2015 INIST-CNRS ;2011 American Heart Association, Inc. 2011 ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.111.030932 ;PMID: 21900086 ;CODEN: CIRCAZ

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13
Macrophage MicroRNA-155 Promotes Cardiac Hypertrophy and Failure
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Macrophage MicroRNA-155 Promotes Cardiac Hypertrophy and Failure

Circulation (New York, N.Y.), 2013-09, Vol.128 (13), p.1420-1432 [Peer Reviewed Journal]

2013 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.112.001357 ;PMID: 23956210 ;CODEN: CIRCAZ

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14
H2S Protects Against Pressure Overload―Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase
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H2S Protects Against Pressure Overload―Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase

Circulation (New York, N.Y.), 2013-03, Vol.127 (10), p.1116-1127 [Peer Reviewed Journal]

2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.112.000855 ;PMID: 23393010 ;CODEN: CIRCAZ

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15
Intracoronary Delivery of Autologous Cardiac Stem Cells Improves Cardiac Function in a Porcine Model of Chronic Ischemic Cardiomyopathy
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Intracoronary Delivery of Autologous Cardiac Stem Cells Improves Cardiac Function in a Porcine Model of Chronic Ischemic Cardiomyopathy

Circulation (New York, N.Y.), 2013-07, Vol.128 (2), p.122-131 [Peer Reviewed Journal]

2013 by the American College of Cardiology Foundation and the American Heart Association, Inc. ;2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.112.001075 ;PMID: 23757309 ;CODEN: CIRCAZ

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16
Cardiomyocyte-Specific Deletion of the Vitamin D Receptor Gene Results in Cardiac Hypertrophy
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Cardiomyocyte-Specific Deletion of the Vitamin D Receptor Gene Results in Cardiac Hypertrophy

Circulation (New York, N.Y.), 2011-10, Vol.124 (17), p.1838-1847 [Peer Reviewed Journal]

2015 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.111.032680 ;PMID: 21947295 ;CODEN: CIRCAZ

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17
Monocyte-Directed RNAi Targeting CCR2 Improves Infarct Healing in Atherosclerosis-Prone Mice
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Monocyte-Directed RNAi Targeting CCR2 Improves Infarct Healing in Atherosclerosis-Prone Mice

Circulation (New York, N.Y.), 2013-05, Vol.127 (20), p.2038-2046 [Peer Reviewed Journal]

2013 American Heart Association, Inc. ;2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.112.000116 ;PMID: 23616627 ;CODEN: CIRCAZ

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18
Novel mechanisms for maintaining endothelial barrier function in sepsis
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Novel mechanisms for maintaining endothelial barrier function in sepsis

Circulation (New York, N.Y.), 2012-12, Vol.126 (23), p.2677-2679 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.112.146100 ;PMID: 23212995

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19
Elevated Cytosolic Na+ Increases Mitochondrial Formation of Reactive Oxygen Species in Failing Cardiac Myocytes
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Elevated Cytosolic Na+ Increases Mitochondrial Formation of Reactive Oxygen Species in Failing Cardiac Myocytes

Circulation (New York, N.Y.), 2010-04, Vol.121 (14), p.1606-1613 [Peer Reviewed Journal]

2015 INIST-CNRS ;2010 American Heart Association, Inc. 2010 ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.109.914911 ;PMID: 20351235 ;CODEN: CIRCAZ

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20
Epac2 Mediates Cardiac β1-Adrenergic―Dependent Sarcoplasmic Reticulum Ca2+ Leak and Arrhythmia
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Epac2 Mediates Cardiac β1-Adrenergic―Dependent Sarcoplasmic Reticulum Ca2+ Leak and Arrhythmia

Circulation (New York, N.Y.), 2013-02, Vol.127 (8), p.913-922 [Peer Reviewed Journal]

2014 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.12.148619 ;PMID: 23363625 ;CODEN: CIRCAZ

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Results 1 - 20 of 397  for All Library Resources

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