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1
Thyroid and Glucocorticoid Hormones Promote Functional T-Tubule Development in Human-Induced Pluripotent Stem Cell–Derived Cardiomyocytes
Thyroid and Glucocorticoid Hormones Promote Functional T-Tubule Development in Human-Induced Pluripotent Stem Cell–Derived Cardiomyocytes
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Thyroid and Glucocorticoid Hormones Promote Functional T-Tubule Development in Human-Induced Pluripotent Stem Cell–Derived Cardiomyocytes

Circulation research, 2017-12, Vol.121 (12), p.1323-1330 [Peer Reviewed Journal]

2017 American Heart Association, Inc. ;Copyright Lippincott Williams & Wilkins Ovid Technologies Dec 8, 2017 ;ISSN: 0009-7330 ;EISSN: 1524-4571 ;DOI: 10.1161/CIRCRESAHA.117.311920 ;PMID: 28974554

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2
Glucocorticoids Acutely Increase Brown Adipose Tissue Activity in Humans, Revealing Species-Specific Differences in UCP-1 Regulation
Glucocorticoids Acutely Increase Brown Adipose Tissue Activity in Humans, Revealing Species-Specific Differences in UCP-1 Regulation
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Glucocorticoids Acutely Increase Brown Adipose Tissue Activity in Humans, Revealing Species-Specific Differences in UCP-1 Regulation

Cell metabolism, 2016-07, Vol.24 (1), p.130-141 [Peer Reviewed Journal]

2016 The Author(s) ;Copyright © 2016 The Author(s). Published by Elsevier Inc. All rights reserved. ;2016 The Author(s) 2016 ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2016.06.011 ;PMID: 27411014

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3
Glucocorticoids Drive Diurnal Oscillations in T Cell Distribution and Responses by Inducing Interleukin-7 Receptor and CXCR4
Glucocorticoids Drive Diurnal Oscillations in T Cell Distribution and Responses by Inducing Interleukin-7 Receptor and CXCR4
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Glucocorticoids Drive Diurnal Oscillations in T Cell Distribution and Responses by Inducing Interleukin-7 Receptor and CXCR4

Immunity (Cambridge, Mass.), 2018-02, Vol.48 (2), p.286-298.e6 [Peer Reviewed Journal]

2018 Elsevier Inc. ;Copyright © 2018 Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited Feb 20, 2018 ;ISSN: 1074-7613 ;EISSN: 1097-4180 ;DOI: 10.1016/j.immuni.2018.01.004 ;PMID: 29396162

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4
An epithelial circadian clock controls pulmonary inflammation and glucocorticoid action
An epithelial circadian clock controls pulmonary inflammation and glucocorticoid action
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An epithelial circadian clock controls pulmonary inflammation and glucocorticoid action

Nature medicine, 2014-08, Vol.20 (8), p.919-926 [Peer Reviewed Journal]

COPYRIGHT 2014 Nature Publishing Group ;COPYRIGHT 2014 Nature Publishing Group ;Copyright Nature Publishing Group Aug 2014 ;ISSN: 1078-8956 ;EISSN: 1546-170X ;DOI: 10.1038/nm.3599 ;PMID: 25064128

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5
Adenosine A 2A receptor regulation of microglia morphological remodeling-gender bias in physiology and in a model of chronic anxiety
Adenosine A 2A receptor regulation of microglia morphological remodeling-gender bias in physiology and in a model of chronic anxiety
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Adenosine A 2A receptor regulation of microglia morphological remodeling-gender bias in physiology and in a model of chronic anxiety

Molecular psychiatry, 2017-07, Vol.22 (7), p.1035 [Peer Reviewed Journal]

EISSN: 1476-5578 ;DOI: 10.1038/mp.2016.173 ;PMID: 27725661

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6
Genomic redistribution of GR monomers and dimers mediates transcriptional response to exogenous glucocorticoid in vivo
Genomic redistribution of GR monomers and dimers mediates transcriptional response to exogenous glucocorticoid in vivo
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Genomic redistribution of GR monomers and dimers mediates transcriptional response to exogenous glucocorticoid in vivo

Genome research, 2015-06, Vol.25 (6), p.836-844 [Peer Reviewed Journal]

2015 Lim et al.; Published by Cold Spring Harbor Laboratory Press. ;2015 ;ISSN: 1088-9051 ;EISSN: 1549-5469 ;DOI: 10.1101/gr.188581.114 ;PMID: 25957148

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7
Glucocorticoids induce differentiation of monocytes towards macrophages that share functional and phenotypical aspects with erythroblastic island macrophages
Glucocorticoids induce differentiation of monocytes towards macrophages that share functional and phenotypical aspects with erythroblastic island macrophages
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Glucocorticoids induce differentiation of monocytes towards macrophages that share functional and phenotypical aspects with erythroblastic island macrophages

Haematologica (Roma), 2018-03, Vol.103 (3), p.395-405 [Peer Reviewed Journal]

Copyright© 2018 Ferrata Storti Foundation. ;Copyright© 2018 Ferrata Storti Foundation 2018 ;ISSN: 0390-6078 ;EISSN: 1592-8721 ;DOI: 10.3324/haematol.2017.179341 ;PMID: 29284682

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8
Dexamethasone Stiffens Trabecular Meshwork, Trabecular Meshwork Cells, and Matrix
Dexamethasone Stiffens Trabecular Meshwork, Trabecular Meshwork Cells, and Matrix
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Dexamethasone Stiffens Trabecular Meshwork, Trabecular Meshwork Cells, and Matrix

Investigative ophthalmology & visual science, 2015-07, Vol.56 (8), p.4447-4459 [Peer Reviewed Journal]

Copyright 2015 The Association for Research in Vision and Ophthalmology, Inc. 2015 ;ISSN: 1552-5783 ;ISSN: 0146-0404 ;EISSN: 1552-5783 ;DOI: 10.1167/iovs.15-16739 ;PMID: 26193921

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9
Anti-Inflammatory Chromatinscape Suggests Alternative Mechanisms of Glucocorticoid Receptor Action
Anti-Inflammatory Chromatinscape Suggests Alternative Mechanisms of Glucocorticoid Receptor Action
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Anti-Inflammatory Chromatinscape Suggests Alternative Mechanisms of Glucocorticoid Receptor Action

Immunity (Cambridge, Mass.), 2017-08, Vol.47 (2), p.298-309.e5 [Peer Reviewed Journal]

2017 ;Published by Elsevier Inc. ;Copyright Elsevier Limited Aug 15, 2017 ;ISSN: 1074-7613 ;EISSN: 1097-4180 ;DOI: 10.1016/j.immuni.2017.07.012 ;PMID: 28801231

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10
Excess glucocorticoid exposure contributes to adipose tissue fibrosis which involves macrophage interaction with adipose precursor cells
Excess glucocorticoid exposure contributes to adipose tissue fibrosis which involves macrophage interaction with adipose precursor cells
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Excess glucocorticoid exposure contributes to adipose tissue fibrosis which involves macrophage interaction with adipose precursor cells

Biochemical pharmacology, 2022-04, Vol.198 [Peer Reviewed Journal]

ISSN: 0006-2952 ;ISSN: 1356-1839 ;EISSN: 1873-2968 ;DOI: 10.1016/j.bcp.2022.114976

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11
Resolvin-D2 targets myogenic cells and improves muscle regeneration in Duchenne muscular dystrophy
Resolvin-D2 targets myogenic cells and improves muscle regeneration in Duchenne muscular dystrophy
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Resolvin-D2 targets myogenic cells and improves muscle regeneration in Duchenne muscular dystrophy

Nature communications, 2021-10, Vol.12 (1), p.6264-6264, Article 6264 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-26516-0 ;PMID: 34716330

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12
Patients with hypercortisolemic Cushing disease possess a distinct class of hematopoietic progenitor cells leading to erythrocytosis
Patients with hypercortisolemic Cushing disease possess a distinct class of hematopoietic progenitor cells leading to erythrocytosis
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Patients with hypercortisolemic Cushing disease possess a distinct class of hematopoietic progenitor cells leading to erythrocytosis

Haematologica (Roma), 2023-04, Vol.108 (4), p.1053-1067 [Peer Reviewed Journal]

Copyright© 2023 Ferrata Storti Foundation ;ISSN: 0390-6078 ;EISSN: 1592-8721 ;DOI: 10.3324/haematol.2021.280542 ;PMID: 35861015

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13
Insights into Negative Regulation by the Glucocorticoid Receptor from Genome-wide Profiling of Inflammatory Cistromes
Insights into Negative Regulation by the Glucocorticoid Receptor from Genome-wide Profiling of Inflammatory Cistromes
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Insights into Negative Regulation by the Glucocorticoid Receptor from Genome-wide Profiling of Inflammatory Cistromes

Molecular cell, 2013-01, Vol.49 (1), p.158-171 [Peer Reviewed Journal]

2013 Elsevier Inc. ;Copyright © 2013 Elsevier Inc. All rights reserved. ;2013 Elsevier Inc. All rights reserved. 2013 ;ISSN: 1097-2765 ;EISSN: 1097-4164 ;DOI: 10.1016/j.molcel.2012.10.013 ;PMID: 23159735

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14
Cortical bone loss caused by glucocorticoid excess requires RANKL production by osteocytes and is associated with reduced OPG expression in mice
Cortical bone loss caused by glucocorticoid excess requires RANKL production by osteocytes and is associated with reduced OPG expression in mice
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Cortical bone loss caused by glucocorticoid excess requires RANKL production by osteocytes and is associated with reduced OPG expression in mice

American journal of physiology: endocrinology and metabolism, 2016-09, Vol.311 (3), p.E587-E593 [Peer Reviewed Journal]

Copyright American Physiological Society Sep 1, 2016 ;ISSN: 0193-1849 ;EISSN: 1522-1555 ;DOI: 10.1152/ajpendo.00219.2016 ;PMID: 27460899 ;CODEN: AJPMD9

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15
Downregulation of MicroRNA-30 Facilitates Podocyte Injury and Is Prevented by Glucocorticoids
Downregulation of MicroRNA-30 Facilitates Podocyte Injury and Is Prevented by Glucocorticoids
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Downregulation of MicroRNA-30 Facilitates Podocyte Injury and Is Prevented by Glucocorticoids

Journal of the American Society of Nephrology, 2014-01, Vol.25 (1), p.92-104 [Peer Reviewed Journal]

2015 INIST-CNRS ;Copyright © 2014 by the American Society of Nephrology 2014 ;ISSN: 1046-6673 ;EISSN: 1533-3450 ;DOI: 10.1681/asn.2012111101 ;PMID: 24029422 ;CODEN: JASNEU

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16
Dexamethasone and Glucocorticoid-Induced Matrix Temporally Modulate Key Integrins, Caveolins, Contractility, and Stiffness in Human Trabecular Meshwork Cells
Dexamethasone and Glucocorticoid-Induced Matrix Temporally Modulate Key Integrins, Caveolins, Contractility, and Stiffness in Human Trabecular Meshwork Cells
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Dexamethasone and Glucocorticoid-Induced Matrix Temporally Modulate Key Integrins, Caveolins, Contractility, and Stiffness in Human Trabecular Meshwork Cells

Investigative ophthalmology & visual science, 2020-11, Vol.61 (13), p.16-16 [Peer Reviewed Journal]

Copyright 2020 The Authors 2020 ;ISSN: 1552-5783 ;ISSN: 0146-0404 ;EISSN: 1552-5783 ;DOI: 10.1167/iovs.61.13.16 ;PMID: 33170205

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17
PPAR-α and glucocorticoid receptor synergize to promote erythroid progenitor self-renewal
PPAR-α and glucocorticoid receptor synergize to promote erythroid progenitor self-renewal
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PPAR-α and glucocorticoid receptor synergize to promote erythroid progenitor self-renewal

Nature (London), 2015-06, Vol.522 (7557), p.474-477 [Peer Reviewed Journal]

ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature14326 ;PMID: 25970251

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18
IL-17 stimulates differentiation of human anti-inflammatory macrophages and phagocytosis of apoptotic neutrophils in response to IL-10 and glucocorticoids
IL-17 stimulates differentiation of human anti-inflammatory macrophages and phagocytosis of apoptotic neutrophils in response to IL-10 and glucocorticoids
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IL-17 stimulates differentiation of human anti-inflammatory macrophages and phagocytosis of apoptotic neutrophils in response to IL-10 and glucocorticoids

The Journal of immunology (1950), 2013-05, Vol.190 (10), p.5237-5246 [Peer Reviewed Journal]

ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.1203017 ;PMID: 23596310

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19
Distinct expression of SARS‐CoV‐2 receptor ACE2 correlates with endotypes of chronic rhinosinusitis with nasal polyps
Distinct expression of SARS‐CoV‐2 receptor ACE2 correlates with endotypes of chronic rhinosinusitis with nasal polyps
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Distinct expression of SARS‐CoV‐2 receptor ACE2 correlates with endotypes of chronic rhinosinusitis with nasal polyps

Allergy, 2021-03 [Peer Reviewed Journal]

2020. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://novel-coronavirus.onlinelibrary.wiley.com ;DOI: 10.1111/all.14665

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20
Glucocorticoid regulation of ATP release from spinal astrocytes underlies diurnal exacerbation of neuropathic mechanical allodynia
Glucocorticoid regulation of ATP release from spinal astrocytes underlies diurnal exacerbation of neuropathic mechanical allodynia
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Glucocorticoid regulation of ATP release from spinal astrocytes underlies diurnal exacerbation of neuropathic mechanical allodynia

Nature communications, 2016-10, Vol.7 (1), p.13102-13102, Article 13102 [Peer Reviewed Journal]

Copyright Nature Publishing Group Oct 2016 ;Copyright © 2016, The Author(s) 2016 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms13102 ;PMID: 27739425

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