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1
Chemosensitivity of human melanoma xenografts in immunocompetent mice and its histological evaluation
Chemosensitivity of human melanoma xenografts in immunocompetent mice and its histological evaluation
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Chemosensitivity of human melanoma xenografts in immunocompetent mice and its histological evaluation

International journal of cancer, 1984-04, Vol.33 (4), p.447-451 [Peer Reviewed Journal]

Copyright © 1984 Wiley‐Liss, Inc., A Wiley Company ;1985 INIST-CNRS ;ISSN: 0020-7136 ;EISSN: 1097-0215 ;DOI: 10.1002/ijc.2910330405 ;PMID: 6706432 ;CODEN: IJCNAW

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2
Stearoyl-CoA Desaturase 1 Protects Ovarian Cancer Cells from Ferroptotic Cell Death
Stearoyl-CoA Desaturase 1 Protects Ovarian Cancer Cells from Ferroptotic Cell Death
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Stearoyl-CoA Desaturase 1 Protects Ovarian Cancer Cells from Ferroptotic Cell Death

Cancer research (Chicago, Ill.), 2019-10, Vol.79 (20), p.5355-5366 [Peer Reviewed Journal]

2019 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.can-19-0369 ;PMID: 31270077

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3
CD4 T-Cell Exhaustion: Does It Exist and What Are Its Roles in Cancer?
CD4 T-Cell Exhaustion: Does It Exist and What Are Its Roles in Cancer?
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CD4 T-Cell Exhaustion: Does It Exist and What Are Its Roles in Cancer?

Clinical cancer research, 2021-11, Vol.27 (21), p.5742-5752 [Peer Reviewed Journal]

2021 The Authors; Published by the American Association for Cancer Research. ;ISSN: 1078-0432 ;EISSN: 1557-3265 ;DOI: 10.1158/1078-0432.CCR-21-0206 ;PMID: 34127507

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4
The Deubiquitylase OTUB1 Mediates Ferroptosis via Stabilization of SLC7A11
The Deubiquitylase OTUB1 Mediates Ferroptosis via Stabilization of SLC7A11
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The Deubiquitylase OTUB1 Mediates Ferroptosis via Stabilization of SLC7A11

Cancer research (Chicago, Ill.), 2019-04, Vol.79 (8), p.1913-1924 [Peer Reviewed Journal]

2019 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.can-18-3037 ;PMID: 30709928

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5
Recurrent Loss of STING Signaling in Melanoma Correlates with Susceptibility to Viral Oncolysis
Recurrent Loss of STING Signaling in Melanoma Correlates with Susceptibility to Viral Oncolysis
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Recurrent Loss of STING Signaling in Melanoma Correlates with Susceptibility to Viral Oncolysis

Cancer research (Chicago, Ill.), 2016-11, Vol.76 (22), p.6747-6759 [Peer Reviewed Journal]

2016 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.CAN-16-1404 ;PMID: 27680683

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6
NOX4 Inhibition Potentiates Immunotherapy by Overcoming Cancer-Associated Fibroblast-Mediated CD8 T-cell Exclusion from Tumors
NOX4 Inhibition Potentiates Immunotherapy by Overcoming Cancer-Associated Fibroblast-Mediated CD8 T-cell Exclusion from Tumors
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NOX4 Inhibition Potentiates Immunotherapy by Overcoming Cancer-Associated Fibroblast-Mediated CD8 T-cell Exclusion from Tumors

Cancer research (Chicago, Ill.), 2020-05, Vol.80 (9), p.1846-1860 [Peer Reviewed Journal]

2020 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.CAN-19-3158 ;PMID: 32122909

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7
A Three-Dimensional Organoid Culture System Derived from Human Glioblastomas Recapitulates the Hypoxic Gradients and Cancer Stem Cell Heterogeneity of Tumors Found In Vivo
A Three-Dimensional Organoid Culture System Derived from Human Glioblastomas Recapitulates the Hypoxic Gradients and Cancer Stem Cell Heterogeneity of Tumors Found In Vivo
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A Three-Dimensional Organoid Culture System Derived from Human Glioblastomas Recapitulates the Hypoxic Gradients and Cancer Stem Cell Heterogeneity of Tumors Found In Vivo

Cancer research (Chicago, Ill.), 2016-04, Vol.76 (8), p.2465-2477 [Peer Reviewed Journal]

2016 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.can-15-2402 ;PMID: 26896279

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8
Neutrophils in cancer: neutral no more
Neutrophils in cancer: neutral no more
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Neutrophils in cancer: neutral no more

Nature reviews. Cancer, 2016-07, Vol.16 (7), p.431-446 [Peer Reviewed Journal]

COPYRIGHT 2016 Nature Publishing Group ;COPYRIGHT 2016 Nature Publishing Group ;Copyright Nature Publishing Group Jul 2016 ;ISSN: 1474-175X ;EISSN: 1474-1768 ;DOI: 10.1038/nrc.2016.52 ;PMID: 27282249

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9
Inhibition of ATM Increases Interferon Signaling and Sensitizes Pancreatic Cancer to Immune Checkpoint Blockade Therapy
Inhibition of ATM Increases Interferon Signaling and Sensitizes Pancreatic Cancer to Immune Checkpoint Blockade Therapy
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Inhibition of ATM Increases Interferon Signaling and Sensitizes Pancreatic Cancer to Immune Checkpoint Blockade Therapy

Cancer research (Chicago, Ill.), 2019-08, Vol.79 (15), p.3940-3951 [Peer Reviewed Journal]

2019 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.can-19-0761 ;PMID: 31101760

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10
The Pancreatic Cancer Microbiome Promotes Oncogenesis by Induction of Innate and Adaptive Immune Suppression
The Pancreatic Cancer Microbiome Promotes Oncogenesis by Induction of Innate and Adaptive Immune Suppression
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The Pancreatic Cancer Microbiome Promotes Oncogenesis by Induction of Innate and Adaptive Immune Suppression

Cancer discovery, 2018-04, Vol.8 (4), p.403-416

2018 American Association for Cancer Research. ;ISSN: 2159-8274 ;EISSN: 2159-8290 ;DOI: 10.1158/2159-8290.cd-17-1134 ;PMID: 29567829

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11
Mouse Models for Cancer Immunotherapy Research
Mouse Models for Cancer Immunotherapy Research
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Mouse Models for Cancer Immunotherapy Research

Cancer discovery, 2018-11, Vol.8 (11), p.1358-1365

2018 American Association for Cancer Research. ;ISSN: 2159-8274 ;EISSN: 2159-8290 ;DOI: 10.1158/2159-8290.cd-18-0044 ;PMID: 30309862

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12
Patterns of transcription factor programs and immune pathway activation define four major subtypes of SCLC with distinct therapeutic vulnerabilities
Patterns of transcription factor programs and immune pathway activation define four major subtypes of SCLC with distinct therapeutic vulnerabilities
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Patterns of transcription factor programs and immune pathway activation define four major subtypes of SCLC with distinct therapeutic vulnerabilities

Cancer cell, 2021-03, Vol.39 (3), p.346-360.e7 [Peer Reviewed Journal]

2020 Elsevier Inc. ;Copyright © 2020 Elsevier Inc. All rights reserved. ;ISSN: 1535-6108 ;EISSN: 1878-3686 ;DOI: 10.1016/j.ccell.2020.12.014 ;PMID: 33482121

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13
Diverse Functions of Macrophages in Different Tumor Microenvironments
Diverse Functions of Macrophages in Different Tumor Microenvironments
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Diverse Functions of Macrophages in Different Tumor Microenvironments

Cancer research (Chicago, Ill.), 2018-10, Vol.78 (19), p.5492-5503 [Peer Reviewed Journal]

2018 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.can-18-1367 ;PMID: 30206177

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14
Molecular and Genetic Characterization of MHC Deficiency Identifies EZH2 as Therapeutic Target for Enhancing Immune Recognition
Molecular and Genetic Characterization of MHC Deficiency Identifies EZH2 as Therapeutic Target for Enhancing Immune Recognition
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Molecular and Genetic Characterization of MHC Deficiency Identifies EZH2 as Therapeutic Target for Enhancing Immune Recognition

Cancer discovery, 2019-04, Vol.9 (4), p.546-563

2019 American Association for Cancer Research. ;ISSN: 2159-8274 ;EISSN: 2159-8290 ;DOI: 10.1158/2159-8290.cd-18-1090 ;PMID: 30705065

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15
Personalized In Vitro and In Vivo Cancer Models to Guide Precision Medicine
Personalized In Vitro and In Vivo Cancer Models to Guide Precision Medicine
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Personalized In Vitro and In Vivo Cancer Models to Guide Precision Medicine

Cancer discovery, 2017-05, Vol.7 (5), p.462-477

2017 American Association for Cancer Research. ;ISSN: 2159-8274 ;EISSN: 2159-8290 ;DOI: 10.1158/2159-8290.cd-16-1154 ;PMID: 28331002

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16
The Novel ATR Inhibitor BAY 1895344 Is Efficacious as Monotherapy and Combined with DNA Damage-Inducing or Repair-Compromising Therapies in Preclinical Cancer Models
The Novel ATR Inhibitor BAY 1895344 Is Efficacious as Monotherapy and Combined with DNA Damage-Inducing or Repair-Compromising Therapies in Preclinical Cancer Models
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The Novel ATR Inhibitor BAY 1895344 Is Efficacious as Monotherapy and Combined with DNA Damage-Inducing or Repair-Compromising Therapies in Preclinical Cancer Models

Molecular cancer therapeutics, 2020-01, Vol.19 (1), p.26-38 [Peer Reviewed Journal]

2019 American Association for Cancer Research. ;ISSN: 1535-7163 ;EISSN: 1538-8514 ;DOI: 10.1158/1535-7163.MCT-19-0019 ;PMID: 31582533

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17
Phagocytosis checkpoints as new targets for cancer immunotherapy
Phagocytosis checkpoints as new targets for cancer immunotherapy
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Phagocytosis checkpoints as new targets for cancer immunotherapy

Nature reviews. Cancer, 2019-10, Vol.19 (10), p.568-586 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;COPYRIGHT 2019 Nature Publishing Group ;Copyright Nature Publishing Group Oct 2019 ;ISSN: 1474-175X ;EISSN: 1474-1768 ;DOI: 10.1038/s41568-019-0183-z ;PMID: 31462760

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18
Acquired resistance to fractionated radiotherapy can be overcome by concurrent PD-L1 blockade
Acquired resistance to fractionated radiotherapy can be overcome by concurrent PD-L1 blockade
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Acquired resistance to fractionated radiotherapy can be overcome by concurrent PD-L1 blockade

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19), p.5458-5468 [Peer Reviewed Journal]

2014 American Association for Cancer Research. ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.CAN-14-1258 ;PMID: 25274032

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19
Genome-wide CRISPR screens of T cell exhaustion identify chromatin remodeling factors that limit T cell persistence
Genome-wide CRISPR screens of T cell exhaustion identify chromatin remodeling factors that limit T cell persistence
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Genome-wide CRISPR screens of T cell exhaustion identify chromatin remodeling factors that limit T cell persistence

Cancer cell, 2022-07, Vol.40 (7), p.768-786.e7 [Peer Reviewed Journal]

2022 Elsevier Inc. ;Copyright © 2022 Elsevier Inc. All rights reserved. ;ISSN: 1535-6108 ;EISSN: 1878-3686 ;DOI: 10.1016/j.ccell.2022.06.001 ;PMID: 35750052

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20
Alternative tumour-specific antigens
Alternative tumour-specific antigens
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Alternative tumour-specific antigens

Nature reviews. Cancer, 2019-08, Vol.19 (8), p.465-478 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;Copyright Nature Publishing Group Aug 2019 ;ISSN: 1474-175X ;EISSN: 1474-1768 ;DOI: 10.1038/s41568-019-0162-4 ;PMID: 31278396

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