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| Result Number | Material Type | Add to My Shelf Action | Record Details and Options |
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Human-induced pluripotent stem cells for modelling metabolic perturbations and impaired bioenergetics underlying cardiomyopathiesCardiovascular research, 2021-02, Vol.117 (3), p.694-711 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2020. For permissions, please email: journals.permissions@oup.com. 2020 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2020. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvaa125 ;PMID: 32365198Full text available |
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Research priorities in sarcomeric cardiomyopathiesCardiovascular research, 2015-04, Vol.105 (4), p.449-456 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2015. For permissions please email: journals.permissions@oup.com. ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2015. For permissions please email: journals.permissions@oup.com. 2015 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvv019 ;PMID: 25631582Full text available |
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Metabolic changes in hypertrophic cardiomyopathies: scientific update from the Working Group of Myocardial Function of the European Society of CardiologyCardiovascular research, 2018-08, Vol.114 (10), p.1273-1280 [Peer Reviewed Journal]The Author(s) 2018. Published by Oxford University Press on behalf of the European Society of Cardiology 2018 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvy147 ;PMID: 29912308Full text available |
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Cardiovascular consequences of viral infections: from COVID to other viral diseasesCardiovascular research, 2021-11, Vol.117 (13), p.2610-2623 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. 2021 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab315 ;PMID: 34609508Full text available |
| 5 |
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A mutation in the glutamate-rich region of RNA-binding motif protein 20 causes dilated cardiomyopathy through missplicing of titin and impaired Frank-Starling mechanismCardiovascular research, 2016-10, Vol.112 (1), p.452-463 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2016. For permissions please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvw192 ;PMID: 27496873Full text available |
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Inhibiting cardiac myeloperoxidase alleviates the relaxation defect in hypertrophic cardiomyocytesCardiovascular research, 2022-01, Vol.118 (2), p.517-530 [Peer Reviewed Journal]The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. 2021 ;The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. ;The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. 2021 ;ISSN: 0008-6363 ;ISSN: 1755-3245 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab077 ;PMID: 33705529Full text available |
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Animal and in silico models for the study of sarcomeric cardiomyopathiesCardiovascular research, 2015-04, Vol.105 (4), p.439-448 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2015. For permissions please email: journals.permissions@oup.com. ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2015. For permissions please email: journals.permissions@oup.com. 2015 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvv006 ;PMID: 25600962Full text available |
| 8 |
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Titin-truncating mutations associated with dilated cardiomyopathy alter length-dependent activation and its modulation via phosphorylationCardiovascular research, 2022-01, Vol.118 (1), p.241-253 [Peer Reviewed Journal]The Author(s) 2020. Published by Oxford University Press on behalf of the European Society of Cardiology. 2020 ;The Author(s) 2020. Published by Oxford University Press on behalf of the European Society of Cardiology. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvaa316 ;PMID: 33135063Full text available |
| 9 |
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Complex roads from genotype to phenotype in dilated cardiomyopathy: scientific update from the Working Group of Myocardial Function of the European Society of CardiologyCardiovascular research, 2018-08, Vol.114 (10), p.1287-1303 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2018. For permissions, please email: journals.permissions@oup.com. 2018 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvy122 ;PMID: 29800419Full text available |
| 10 |
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Adiponectin protects against doxorubicin-induced cardiomyopathy by anti-apoptotic effects through AMPK up-regulationCardiovascular research, 2011-02, Vol.89 (2), p.309-319 [Peer Reviewed Journal]2015 INIST-CNRS ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvq335 ;PMID: 20978005 ;CODEN: CVREAUFull text available |
| 11 |
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A novel patient–patient network medicine approach to refine hypertrophic cardiomyopathy subgrouping: implications for risk stratificationCardiovascular research, 2023-05, Vol.119 (3), p.e125-e127 [Peer Reviewed Journal]Published by Oxford University Press on behalf of the European Society of Cardiology 2023. 2023 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad036 ;PMID: 37129987Full text available |
| 12 |
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Therapeutic strategies utilizing SDF-1α in ischaemic cardiomyopathyCardiovascular research, 2018-03, Vol.114 (3), p.358-367 [Peer Reviewed Journal]The Author 2017. Published by Oxford University Press on behalf of the European Society of Cardiology 2017 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvx203 ;PMID: 29040423Full text available |
| 13 |
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Deficit in PINK1/PARKIN-mediated mitochondrial autophagy at late stages of dystrophic cardiomyopathyCardiovascular research, 2018-01, Vol.114 (1), p.90-102 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2017. For permissions, please email: journals.permissions@oup.com. 2017 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2017. For permissions please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvx201 ;PMID: 29036556Full text available |
| 14 |
Material Type: Article
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Hypertrophic cardiomyopathy dysfunction mimicked in human engineered heart tissue and improved by sodium–glucose cotransporter 2 inhibitorsCardiovascular research, 2024-03, Vol.120 (3), p.301-317 [Peer Reviewed Journal]The Author(s) 2024. Published by Oxford University Press on behalf of the European Society of Cardiology. 2024 ;The Author(s) 2024. Published by Oxford University Press on behalf of the European Society of Cardiology. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvae004 ;PMID: 38240646Full text available |
| 15 |
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Management of cardiac fibrosis is the largest unmet medical need in heart failureCardiovascular research, 2022-01, Vol.118 (2), p.e20-e22 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. 2021 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab228 ;PMID: 34244741Full text available |
| 16 |
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Dilated cardiomyopathy-linked heat shock protein family D member 1 mutations cause up-regulation of reactive oxygen species and autophagy through mitochondrial dysfunctionCardiovascular research, 2021-03, Vol.117 (4), p.1118-1131 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2020. For permissions, please email: journals.permissions@oup.com. 2020 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2020. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvaa158 ;PMID: 32520982Full text available |
| 17 |
Material Type: Article
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The impact of patient sex on the response to intramyocardial mesenchymal stem cell administration in patients with non-ischaemic dilated cardiomyopathyCardiovascular research, 2020-11, Vol.116 (13), p.2131-2141 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2020. For permissions, please email: journals.permissions@oup.com. 2020 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2020. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvaa004 ;PMID: 32053144Full text available |
| 18 |
Material Type: Article
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Sense and stretchability: The role of titin and titin-associated proteins in myocardial stress-sensing and mechanical dysfunctionCardiovascular research, 2008-03, Vol.77 (4), p.637-648 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2007. For permissions please email: journals.permissions@oxfordjournals.org 2008 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1016/j.cardiores.2007.03.029 ;PMID: 17475230Full text available |
| 19 |
Material Type: Article
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Cardioprotective effects of cerium oxide nanoparticles in a transgenic murine model of cardiomyopathyCardiovascular research, 2007-02, Vol.73 (3), p.549-559 [Peer Reviewed Journal]2007 INIST-CNRS ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1016/j.cardiores.2006.11.031 ;PMID: 17207782 ;CODEN: CVREAUFull text available |
| 20 |
Material Type: Article
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Fropofol prevents disease progression in mice with hypertrophic cardiomyopathyCardiovascular research, 2020-05, Vol.116 (6), p.1175-1185 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2019. For permissions, please email: journals.permissions@oup.com. 2019 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2019. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvz218 ;PMID: 31424496Full text available |
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