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Material Type: Article
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Causal relationships between obesity and the leading causes of death in women and menPLoS genetics, 2019-10, Vol.15 (10), p.e1008405-e1008405 [Peer Reviewed Journal]COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Censin et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Censin et al 2019 Censin et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1008405 ;PMID: 31647808Full text available |
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Multimodal CRISPR perturbations of GWAS loci associated with coronary artery disease in vascular endothelial cellsPLoS genetics, 2023-03, Vol.19 (3), p.e1010680-e1010680 [Peer Reviewed Journal]Copyright: © 2023 Wünnemann et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. ;COPYRIGHT 2023 Public Library of Science ;2023 Wünnemann et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 Wünnemann et al 2023 Wünnemann et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1010680 ;PMID: 36928188Full text available |
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Material Type: Article
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Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease riskPLoS genetics, 2018-10, Vol.14 (10), p.e1007681-e1007681 [Peer Reviewed Journal]COPYRIGHT 2018 Public Library of Science ;2018 Iyer et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Iyer et al 2018 Iyer et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1007681 ;PMID: 30307970Full text available |
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Material Type: Article
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Alu elements in ANRIL non-coding RNA at chromosome 9p21 modulate atherogenic cell functions through trans-regulation of gene networksPLoS genetics, 2013-07, Vol.9 (7), p.e1003588 [Peer Reviewed Journal]COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 Holdt et al 2013 Holdt et al ;2013 Holdt et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Holdt LM, Hoffmann S, Sass K, Langenberger D, Scholz M, et al. (2013) Alu Elements in ANRIL Non-Coding RNA at Chromosome 9p21 Modulate Atherogenic Cell Functions through Trans-Regulation of Gene Networks. PLoS Genet 9(7): e1003588. doi:10.1371/journal.pgen.1003588 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1003588 ;PMID: 23861667Full text available |
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Material Type: Article
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A novel MMP12 locus is associated with large artery atherosclerotic stroke using a genome-wide age-at-onset informed approachPLoS genetics, 2014-07, Vol.10 (7), p.e1004469-e1004469 [Peer Reviewed Journal]COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 ;2014 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Locus Is Associated with Large Artery Atherosclerotic Stroke Using a Genome-Wide Age-at-Onset Informed Approach. PLoS Genet 10(7): e1004469. doi:10.1371/journal.pgen.1004469 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1004469 ;PMID: 25078452Full text available |
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Material Type: Article
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Mapping of 79 loci for 83 plasma protein biomarkers in cardiovascular diseasePLoS genetics, 2017-04, Vol.13 (4), p.e1006706 [Peer Reviewed Journal]COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Folkersen L, Fauman E, Sabater-Lleal M, Strawbridge RJ, Frånberg M, Sennblad B, et al. (2017) Mapping of 79 loci for 83 plasma protein biomarkers in cardiovascular disease. PLoS Genet 13(4): e1006706. https://doi.org/10.1371/journal.pgen.1006706 ;2017 Folkersen et al 2017 Folkersen et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Folkersen L, Fauman E, Sabater-Lleal M, Strawbridge RJ, Frånberg M, Sennblad B, et al. (2017) Mapping of 79 loci for 83 plasma protein biomarkers in cardiovascular disease. PLoS Genet 13(4): e1006706. https://doi.org/10.1371/journal.pgen.1006706 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1006706 ;PMID: 28369058Full text available |
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Material Type: Article
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Integrative genomics reveals novel molecular pathways and gene networks for coronary artery diseasePLoS genetics, 2014-07, Vol.10 (7), p.e1004502-e1004502 [Peer Reviewed Journal]COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Mäkinen et al 2014 Mäkinen et al ;2014 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Mäkinen V-P, Civelek M, Meng Q, Zhang B, Zhu J, Levian C, et al. (2014) Integrative Genomics Reveals Novel Molecular Pathways and Gene Networks for Coronary Artery Disease. PLoS Genet 10(7): e1004502. doi:10.1371/journal.pgen.1004502 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1004502 ;PMID: 25033284Full text available |
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Material Type: Article
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Functional regulatory mechanism of smooth muscle cell-restricted LMOD1 coronary artery disease locusPLoS genetics, 2018-11, Vol.14 (11), p.e1007755-e1007755 [Peer Reviewed Journal]COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Nanda et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Nanda et al 2018 Nanda et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1007755 ;PMID: 30444878Full text available |
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Material Type: Article
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Disproportionate Contributions of Select Genomic Compartments and Cell Types to Genetic Risk for Coronary Artery DiseasePLoS genetics, 2015-10, Vol.11 (10), p.e1005622-e1005622 [Peer Reviewed Journal]COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Won et al 2015 Won et al ;2015 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Won H-H, Natarajan P, Dobbyn A, Jordan DM, Roussos P, Lage K, et al. (2015) Disproportionate Contributions of Select Genomic Compartments and Cell Types to Genetic Risk for Coronary Artery Disease. PLoS Genet 11(10): e1005622. doi:10.1371/journal.pgen.1005622 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1005622 ;PMID: 26509271Full text available |
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Material Type: Article
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Coronary Artery Disease Associated Transcription Factor TCF21 Regulates Smooth Muscle Precursor Cells That Contribute to the Fibrous CapPLoS genetics, 2015-05, Vol.11 (5), p.e1005155-e1005155 [Peer Reviewed Journal]COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Nurnberg et al 2015 Nurnberg et al ;2015 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Nurnberg ST, Cheng K, Raiesdana A, Kundu R, Miller CL, Kim JB, et al. (2015) Coronary Artery Disease Associated Transcription Factor TCF21 Regulates Smooth Muscle Precursor Cells That Contribute to the Fibrous Cap. PLoS Genet 11(5): e1005155. doi:10.1371/journal.pgen.1005155 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1005155 ;PMID: 26020946Full text available |
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Material Type: Article
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Genetic loci associated with coronary artery disease harbor evidence of selection and antagonistic pleiotropyPLoS genetics, 2017-06, Vol.13 (6), p.e1006328-e1006328 [Peer Reviewed Journal]COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Byars SG, Huang QQ, Gray L-A, Bakshi A, Ripatti S, Abraham G, et al. (2017) Genetic loci associated with coronary artery disease harbor evidence of selection and antagonistic pleiotropy. PLoS Genet 13(6): e1006328. https://doi.org/10.1371/journal.pgen.1006328 ;2017 Byars et al 2017 Byars et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Byars SG, Huang QQ, Gray L-A, Bakshi A, Ripatti S, Abraham G, et al. (2017) Genetic loci associated with coronary artery disease harbor evidence of selection and antagonistic pleiotropy. PLoS Genet 13(6): e1006328. https://doi.org/10.1371/journal.pgen.1006328 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1006328 ;PMID: 28640878Full text available |
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Material Type: Article
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The Contribution of GWAS Loci in Familial DyslipidemiasPLoS genetics, 2016-05, Vol.12 (5), p.e1006078-e1006078 [Peer Reviewed Journal]COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Ripatti P, Rämö JT, Söderlund S, Surakka I, Matikainen N, Pirinen M, et al. (2016) The Contribution of GWAS Loci in Familial Dyslipidemias. PLoS Genet 12(5): e1006078. doi:10.1371/journal.pgen.1006078 ;2016 Ripatti et al 2016 Ripatti et al ;2016 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Ripatti P, Rämö JT, Söderlund S, Surakka I, Matikainen N, Pirinen M, et al. (2016) The Contribution of GWAS Loci in Familial Dyslipidemias. PLoS Genet 12(5): e1006078. doi:10.1371/journal.pgen.1006078 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1006078 ;PMID: 27227539Full text available |
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Material Type: Article
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A missense variant in Mitochondrial Amidoxime Reducing Component 1 gene and protection against liver diseasePLoS genetics, 2020-04, Vol.16 (4), p.e1008629 [Peer Reviewed Journal]2020 Emdin et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Emdin et al 2020 Emdin et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1008629 ;PMID: 32282858Full text available |
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Material Type: Article
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RNAi-based functional profiling of loci from blood lipid genome-wide association studies identifies genes with cholesterol-regulatory functionPLoS genetics, 2013-02, Vol.9 (2), p.e1003338-e1003338 [Peer Reviewed Journal]COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 Blattmann et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Blattmann P, Schuberth C, Pepperkok R, Runz H (2013) RNAi-Based Functional Profiling of Loci from Blood Lipid Genome-Wide Association Studies Identifies Genes with Cholesterol-Regulatory Function. PLoS Genet 9(2): e1003338. doi:10.1371/journal.pgen.1003338 ;2013 Blattmann et al 2013 Blattmann et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1003338 ;PMID: 23468663Full text available |
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Material Type: Article
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Identifying systematic heterogeneity patterns in genetic association meta-analysis studiesPLoS genetics, 2017-05, Vol.13 (5), p.e1006755-e1006755 [Peer Reviewed Journal]COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Magosi LE, Goel A, Hopewell JC, Farrall M, on behalf of the CARDIoGRAMplusC4D Consortium (2017) Identifying systematic heterogeneity patterns in genetic association meta-analysis studies. PLoS Genet 13(5): e1006755. https://doi.org/10.1371/journal.pgen.1006755 ;2017 Magosi et al 2017 Magosi et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Magosi LE, Goel A, Hopewell JC, Farrall M, on behalf of the CARDIoGRAMplusC4D Consortium (2017) Identifying systematic heterogeneity patterns in genetic association meta-analysis studies. PLoS Genet 13(5): e1006755. https://doi.org/10.1371/journal.pgen.1006755 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1006755 ;PMID: 28459806Full text available |
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Material Type: Article
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De novo variants in congenital diaphragmatic hernia identify MYRF as a new syndrome and reveal genetic overlaps with other developmental disordersPLoS genetics, 2018-12, Vol.14 (12), p.e1007822-e1007822 [Peer Reviewed Journal]COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Qi et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Qi et al 2018 Qi et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1007822 ;PMID: 30532227Full text available |
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Material Type: Article
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The impact of age on genetic risk for common diseasesPLoS genetics, 2021-08, Vol.17 (8), p.e1009723 [Peer Reviewed Journal]COPYRIGHT 2021 Public Library of Science ;2021 Jiang et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 Jiang et al 2021 Jiang et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1009723 ;PMID: 34437535Full text available |
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Integrative Tissue-Specific Functional Annotations in the Human Genome Provide Novel Insights on Many Complex Traits and Improve Signal Prioritization in Genome Wide Association StudiesPLoS genetics, 2016-04, Vol.12 (4), p.e1005947-e1005947 [Peer Reviewed Journal]COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Lu Q, Powles RL, Wang Q, He BJ, Zhao H (2016) Integrative Tissue-Specific Functional Annotations in the Human Genome Provide Novel Insights on Many Complex Traits and Improve Signal Prioritization in Genome Wide Association Studies. PLoS Genet 12(4): e1005947. doi:10.1371/journal.pgen.1005947 ;2016 Lu et al 2016 Lu et al ;2016 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Lu Q, Powles RL, Wang Q, He BJ, Zhao H (2016) Integrative Tissue-Specific Functional Annotations in the Human Genome Provide Novel Insights on Many Complex Traits and Improve Signal Prioritization in Genome Wide Association Studies. PLoS Genet 12(4): e1005947. doi:10.1371/journal.pgen.1005947 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1005947 ;PMID: 27058395Full text available |
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Genetics meets metabolomics: a genome-wide association study of metabolite profiles in human serumPLoS genetics, 2008-11, Vol.4 (11), p.e1000282-e1000282 [Peer Reviewed Journal]COPYRIGHT 2008 Public Library of Science ;Gieger et al. 2008 ;2008 Gieger et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Gieger C, Geistlinger L, Altmaier E, Hrabé de Angelis M, Kronenberg F, et al. (2008) Genetics Meets Metabolomics: A Genome-Wide Association Study of Metabolite Profiles in Human Serum. PLoS Genet 4(11): e1000282. doi:10.1371/journal.pgen.1000282 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1000282 ;PMID: 19043545Full text available |
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Material Type: Article
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Multi-organ expression profiling uncovers a gene module in coronary artery disease involving transendothelial migration of leukocytes and LIM domain binding 2: the Stockholm Atherosclerosis Gene Expression (STAGE) studyPLoS genetics, 2009-12, Vol.5 (12), p.e1000754-e1000754 [Peer Reviewed Journal]Hägg et al. 2009 ;2009 Hägg et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Hägg S, Skogsberg J, Lundström J, Noori P, Nilsson R, et al. (2009) Multi-Organ Expression Profiling Uncovers a Gene Module in Coronary Artery Disease Involving Transendothelial Migration of Leukocytes and LIM Domain Binding 2: The Stockholm Atherosclerosis Gene Expression (STAGE) Study. PLoS Genet 5(12): e1000754. doi:10.1371/journal.pgen.1000754 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1000754 ;PMID: 19997623Full text available |