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1
Mitochondrial ubiquinol oxidation is necessary for tumour growth
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Mitochondrial ubiquinol oxidation is necessary for tumour growth

Nature (London), 2020-09, Vol.585 (7824), p.288-292 [Peer Reviewed Journal]

COPYRIGHT 2020 Nature Publishing Group ;Copyright Nature Publishing Group Sep 10, 2020 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-020-2475-6 ;PMID: 32641834

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2
Cryo-EM structures of complex I from mouse heart mitochondria in two biochemically defined states
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Cryo-EM structures of complex I from mouse heart mitochondria in two biochemically defined states

Nature structural & molecular biology, 2018-07, Vol.25 (7), p.548-556 [Peer Reviewed Journal]

COPYRIGHT 2018 Nature Publishing Group ;COPYRIGHT 2018 Nature Publishing Group ;Copyright Nature Publishing Group Jul 2018 ;ISSN: 1545-9993 ;EISSN: 1545-9985 ;DOI: 10.1038/s41594-018-0073-1 ;PMID: 29915388

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3
Correlating kinetic and structural data on ubiquinone binding and reduction by respiratory complex I
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Correlating kinetic and structural data on ubiquinone binding and reduction by respiratory complex I

Proceedings of the National Academy of Sciences - PNAS, 2017-11, Vol.114 (48), p.12737-12742 [Peer Reviewed Journal]

Volumes 1–89 and 106–114, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Nov 28, 2017 ;2017 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1714074114 ;PMID: 29133414

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4
Bioavailability of coenzyme Q10 supplements depends on carrier lipids and solubilization
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Bioavailability of coenzyme Q10 supplements depends on carrier lipids and solubilization

Nutrition (Burbank, Los Angeles County, Calif.), 2019-01, Vol.57, p.133-140 [Peer Reviewed Journal]

2019 Elsevier Ltd ;Copyright © 2019 Elsevier Ltd. All rights reserved. ;2019. Elsevier Ltd ;ISSN: 0899-9007 ;EISSN: 1873-1244 ;DOI: 10.1016/j.nut.2018.05.020 ;PMID: 30153575

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5
ADCK4 mutations promote steroid-resistant nephrotic syndrome through CoQ10 biosynthesis disruption
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ADCK4 mutations promote steroid-resistant nephrotic syndrome through CoQ10 biosynthesis disruption

The Journal of clinical investigation, 2013-12, Vol.123 (12), p.5179-5189 [Peer Reviewed Journal]

COPYRIGHT 2013 American Society for Clinical Investigation ;COPYRIGHT 2013 American Society for Clinical Investigation ;Copyright © 2013, American Society for Clinical Investigation 2013 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/jci69000 ;PMID: 24270420

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6
COQ11 deletion mitigates respiratory deficiency caused by mutations in the gene encoding the coenzyme Q chaperone protein Coq10
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COQ11 deletion mitigates respiratory deficiency caused by mutations in the gene encoding the coenzyme Q chaperone protein Coq10

The Journal of biological chemistry, 2020-05, Vol.295 (18), p.6023-6042 [Peer Reviewed Journal]

2020 © 2020 Bradley et al. ;2020 Bradley et al. ;2020 Bradley et al. 2020 Bradley et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA119.012420 ;PMID: 32205446

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7
Locking loop movement in the ubiquinone pocket of complex I disengages the proton pumps
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Locking loop movement in the ubiquinone pocket of complex I disengages the proton pumps

Nature communications, 2018-10, Vol.9 (1), p.4500-10, Article 4500 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-06955-y ;PMID: 30374105

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8
FSP1 is a glutathione-independent ferroptosis suppressor
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FSP1 is a glutathione-independent ferroptosis suppressor

Nature (London), 2019-11, Vol.575 (7784), p.693-698 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;COPYRIGHT 2019 Nature Publishing Group ;Copyright Nature Publishing Group Nov 28, 2019 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-019-1707-0 ;PMID: 31634899

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9
The CoQ oxidoreductase FSP1 acts parallel to GPX4 to inhibit ferroptosis
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The CoQ oxidoreductase FSP1 acts parallel to GPX4 to inhibit ferroptosis

Nature (London), 2019-11, Vol.575 (7784), p.688-692 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;COPYRIGHT 2019 Nature Publishing Group ;Copyright Nature Publishing Group Nov 28, 2019 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-019-1705-2 ;PMID: 31634900

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10
Human COQ10A and COQ10B are distinct lipid-binding START domain proteins required for coenzyme Q function[S]
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Human COQ10A and COQ10B are distinct lipid-binding START domain proteins required for coenzyme Q function[S]

Journal of lipid research, 2019-07, Vol.60 (7), p.1293-1310 [Peer Reviewed Journal]

2019 ;ISSN: 0022-2275 ;EISSN: 1539-7262 ;DOI: 10.1194/jlr.M093534 ;PMID: 31048406

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11
The coupling mechanism of mammalian mitochondrial complex I
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The coupling mechanism of mammalian mitochondrial complex I

Nature structural & molecular biology, 2022-02, Vol.29 (2), p.172-182 [Peer Reviewed Journal]

2022. The Author(s), under exclusive licence to Springer Nature America, Inc. ;The Author(s), under exclusive licence to Springer Nature America, Inc. 2022. ;ISSN: 1545-9993 ;EISSN: 1545-9985 ;DOI: 10.1038/s41594-022-00722-w ;PMID: 35145322

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12
Inhibitors of a Na+-pumping NADH-ubiquinone oxidoreductase play multiple roles to block enzyme function
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Inhibitors of a Na+-pumping NADH-ubiquinone oxidoreductase play multiple roles to block enzyme function

The Journal of biological chemistry, 2020-09, Vol.295 (36), p.12739-12754 [Peer Reviewed Journal]

2020 © 2020 Masuya et al. ;2020 Masuya et al. ;2020 Masuya et al. 2020 Masuya et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA120.014229 ;PMID: 32690607

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13
A targetable CoQ-FSP1 axis drives ferroptosis- and radiation-resistance in KEAP1 inactive lung cancers
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A targetable CoQ-FSP1 axis drives ferroptosis- and radiation-resistance in KEAP1 inactive lung cancers

Nature communications, 2022-04, Vol.13 (1), p.2206-2206, Article 2206 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-29905-1 ;PMID: 35459868

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14
Prenyldiphosphate synthase, subunit 1 (PDSS1) and OH-benzoate polyprenyltransferase (COQ2) mutations in ubiquinone deficiency and oxidative phosphorylation disorders
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Prenyldiphosphate synthase, subunit 1 (PDSS1) and OH-benzoate polyprenyltransferase (COQ2) mutations in ubiquinone deficiency and oxidative phosphorylation disorders

The Journal of clinical investigation, 2007-03, Vol.117 (3), p.765-772 [Peer Reviewed Journal]

COPYRIGHT 2007 American Society for Clinical Investigation ;COPYRIGHT 2007 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Mar 2007 ;Distributed under a Creative Commons Attribution 4.0 International License ;Copyright © 2007, American Society for Clinical Investigation 2007 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/jci29089 ;PMID: 17332895

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15
Coenzyme Q10 modulates sulfide metabolism and links the mitochondrial respiratory chain to pathways associated to one carbon metabolism
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Coenzyme Q10 modulates sulfide metabolism and links the mitochondrial respiratory chain to pathways associated to one carbon metabolism

Human molecular genetics, 2020-11, Vol.29 (19), p.3296-3311 [Peer Reviewed Journal]

The Author(s) 2020. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oup.com. ;The Author(s) 2020. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oup.com 2020 ;ISSN: 0964-6906 ;EISSN: 1460-2083 ;DOI: 10.1093/hmg/ddaa214 ;PMID: 32975579

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16
Genetic bases and clinical manifestations of coenzyme Q10 (CoQ10) deficiency
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Genetic bases and clinical manifestations of coenzyme Q10 (CoQ10) deficiency

Journal of inherited metabolic disease, 2015-01, Vol.38 (1), p.145-156 [Peer Reviewed Journal]

SSIEM 2014 ;2015 SSIEM ;ISSN: 0141-8955 ;EISSN: 1573-2665 ;DOI: 10.1007/s10545-014-9749-9 ;PMID: 25091424

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17
Mitochondrial complex I structure reveals ordered water molecules for catalysis and proton translocation
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Mitochondrial complex I structure reveals ordered water molecules for catalysis and proton translocation

Nature structural & molecular biology, 2020-10, Vol.27 (10), p.892-900 [Peer Reviewed Journal]

ISSN: 1545-9993 ;EISSN: 1545-9985 ;DOI: 10.1038/s41594-020-0473-x ;PMID: 32747785

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18
The kynurenine pathway is essential for rhodoquinone biosynthesis in Caenorhabditis elegans
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The kynurenine pathway is essential for rhodoquinone biosynthesis in Caenorhabditis elegans

The Journal of biological chemistry, 2019-07, Vol.294 (28), p.11047-11053 [Peer Reviewed Journal]

2019 Roberts Buceta et al. ;2019 Roberts Buceta et al. 2019 Roberts Buceta et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.AC119.009475 ;PMID: 31177094

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19
Mitochondrial dysfunction in inherited renal disease and acute kidney injury
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Mitochondrial dysfunction in inherited renal disease and acute kidney injury

Nature reviews. Nephrology, 2016-05, Vol.12 (5), p.267-280 [Peer Reviewed Journal]

COPYRIGHT 2016 Nature Publishing Group ;COPYRIGHT 2016 Nature Publishing Group ;Copyright Nature Publishing Group May 2016 ;ISSN: 1759-5061 ;EISSN: 1759-507X ;DOI: 10.1038/nrneph.2015.214 ;PMID: 26804019

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20
New Amino Acid Schiff Bases as Anticancer Agents via Potential Mitochondrial Complex I-Associated Hexokinase Inhibition and Targeting AMP-Protein Kinases/mTOR Signaling Pathway
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New Amino Acid Schiff Bases as Anticancer Agents via Potential Mitochondrial Complex I-Associated Hexokinase Inhibition and Targeting AMP-Protein Kinases/mTOR Signaling Pathway

Molecules (Basel, Switzerland), 2021-09, Vol.26 (17), p.5332 [Peer Reviewed Journal]

2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 1420-3049 ;EISSN: 1420-3049 ;DOI: 10.3390/molecules26175332 ;PMID: 34500765

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