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1
Emerging Microbiome Genetic Engineering Technology for Stable Levodopa Delivery in Parkinson’s Disease
Emerging Microbiome Genetic Engineering Technology for Stable Levodopa Delivery in Parkinson’s Disease
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Emerging Microbiome Genetic Engineering Technology for Stable Levodopa Delivery in Parkinson’s Disease

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a [Peer Reviewed Journal]

FASEB ;FASEB. ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2022.36.S1.R6272 ;PMID: 35553060

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2
Novel NOX2 inhibitor Mito‐Apocynin Protects Against LPS‐induced Endotoxemia Pre‐Clinical Animal Model
Novel NOX2 inhibitor Mito‐Apocynin Protects Against LPS‐induced Endotoxemia Pre‐Clinical Animal Model
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Novel NOX2 inhibitor Mito‐Apocynin Protects Against LPS‐induced Endotoxemia Pre‐Clinical Animal Model

The FASEB journal, 2019-04, Vol.33 (S1), p.lb39-lb39 [Peer Reviewed Journal]

2019 FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.lb39

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3
Mitochondrial Impairment Upregulates MICOS Expression in a Human Microglial Cell Model
Mitochondrial Impairment Upregulates MICOS Expression in a Human Microglial Cell Model
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Mitochondrial Impairment Upregulates MICOS Expression in a Human Microglial Cell Model

The FASEB journal, 2019-04, Vol.33 (S1), p.lb19-lb19 [Peer Reviewed Journal]

2019 FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.lb19

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4
The Role of ZBP1 in the Neuroinflammatory Response in Glia Cell Models of Parkinson's Disease
The Role of ZBP1 in the Neuroinflammatory Response in Glia Cell Models of Parkinson's Disease
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The Role of ZBP1 in the Neuroinflammatory Response in Glia Cell Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1), p.lb16-lb16 [Peer Reviewed Journal]

2019 FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.lb16

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5
Ultra‐sensitive detection of pathological α‐synuclein in human tissues and biofluids using the RT‐QuIC Assay: Relevance to development of circulating and peripheral biomarkers for diagnosing α‐synucleinopathies and other protein misfolding diseases
Ultra‐sensitive detection of pathological α‐synuclein in human tissues and biofluids using the RT‐QuIC Assay: Relevance to development of circulating and peripheral biomarkers for diagnosing α‐synucleinopathies and other protein misfolding diseases
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Ultra‐sensitive detection of pathological α‐synuclein in human tissues and biofluids using the RT‐QuIC Assay: Relevance to development of circulating and peripheral biomarkers for diagnosing α‐synucleinopathies and other protein misfolding diseases

The FASEB journal, 2019-04, Vol.33 (S1), p.lb88-lb88 [Peer Reviewed Journal]

2019 FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.lb88

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6
Calcium‐activated K+ channel KCa3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease
Calcium‐activated K+ channel KCa3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease
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Calcium‐activated K+ channel KCa3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1), p.500.20-500.20 [Peer Reviewed Journal]

2019 FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.500.20

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7
Mitochondrial Stress Modulates Elongator Protein 3 (ELP3) Function to Enhance Dopaminergic Neurotoxicity in Cell Culture and Animal Models of Parkinson's Disease
Mitochondrial Stress Modulates Elongator Protein 3 (ELP3) Function to Enhance Dopaminergic Neurotoxicity in Cell Culture and Animal Models of Parkinson's Disease
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Mitochondrial Stress Modulates Elongator Protein 3 (ELP3) Function to Enhance Dopaminergic Neurotoxicity in Cell Culture and Animal Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1), p.461.1-461.1 [Peer Reviewed Journal]

2019 FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.461.1

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8
Calcium‐activated K + channel K Ca 3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease
Calcium‐activated K + channel K Ca 3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease
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Calcium‐activated K + channel K Ca 3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1) [Peer Reviewed Journal]

ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.500.20

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9
Manganese exposure augments misfolded α‐synuclein‐induced proinflammatory M1 microglial phenotype and inflammasome activation
Manganese exposure augments misfolded α‐synuclein‐induced proinflammatory M1 microglial phenotype and inflammasome activation
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Manganese exposure augments misfolded α‐synuclein‐induced proinflammatory M1 microglial phenotype and inflammasome activation

The FASEB journal, 2019-04, Vol.33 (S1), p.505.7-505.7 [Peer Reviewed Journal]

2019 FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2019.33.1_supplement.505.7

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10
Hepcidin plays a key role in 6‐OHDA induced iron overload and apoptotic cell death in a cell culture model of Parkinson’s disease (1038.2)
Hepcidin plays a key role in 6‐OHDA induced iron overload and apoptotic cell death in a cell culture model of Parkinson’s disease (1038.2)
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Hepcidin plays a key role in 6‐OHDA induced iron overload and apoptotic cell death in a cell culture model of Parkinson’s disease (1038.2)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a [Peer Reviewed Journal]

FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.28.1_supplement.1038.2

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11
Manganese Exposure Induces Neuroinflammation by Impairing Mitochondrial Dynamics in Astrocytes
Manganese Exposure Induces Neuroinflammation by Impairing Mitochondrial Dynamics in Astrocytes
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Manganese Exposure Induces Neuroinflammation by Impairing Mitochondrial Dynamics in Astrocytes

The FASEB journal, 2017-04, Vol.31 (S1) [Peer Reviewed Journal]

ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.31.1_supplement.lb626

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12
The Fyn‐dependent voltage‐gated potassium channel Kv1.3 modulates neuroinflammation and neurodegeneration in Parkinson's disease models
The Fyn‐dependent voltage‐gated potassium channel Kv1.3 modulates neuroinflammation and neurodegeneration in Parkinson's disease models
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The Fyn‐dependent voltage‐gated potassium channel Kv1.3 modulates neuroinflammation and neurodegeneration in Parkinson's disease models

The FASEB journal, 2018-04, Vol.32 (S1), p.553.1-553.1 [Peer Reviewed Journal]

FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2018.32.1_supplement.553.1

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13
Manganese Exposure Activates NLRP3 Inflammasome Signaling and Propagates Exosomal Release of ASC in Microglial Cells
Manganese Exposure Activates NLRP3 Inflammasome Signaling and Propagates Exosomal Release of ASC in Microglial Cells
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Manganese Exposure Activates NLRP3 Inflammasome Signaling and Propagates Exosomal Release of ASC in Microglial Cells

The FASEB journal, 2018-04, Vol.32 (S1), p.823.8-823.8 [Peer Reviewed Journal]

FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.2018.32.1_supplement.823.8

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14
Pesticide‐induced Mitochondrial Dysfunction Augments NLRP3 Inflammasome Signaling Pathway in Primary Microglia
Pesticide‐induced Mitochondrial Dysfunction Augments NLRP3 Inflammasome Signaling Pathway in Primary Microglia
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Pesticide‐induced Mitochondrial Dysfunction Augments NLRP3 Inflammasome Signaling Pathway in Primary Microglia

The FASEB journal, 2015-04, Vol.29 (S1) [Peer Reviewed Journal]

ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.29.1_supplement.777.5

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15
MITO‐APOCYNIN, A NOVEL MITOCHONDRIA‐TARGETED DERIVATIVE OF APOCYNIN, INHIBITS NLRP3 INFLAMMASOME ACTIVATION IN PRIMARY MURINE MICROGLIA IN DOPAMINERGIC NEUROTOXIC MODELS
MITO‐APOCYNIN, A NOVEL MITOCHONDRIA‐TARGETED DERIVATIVE OF APOCYNIN, INHIBITS NLRP3 INFLAMMASOME ACTIVATION IN PRIMARY MURINE MICROGLIA IN DOPAMINERGIC NEUROTOXIC MODELS
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MITO‐APOCYNIN, A NOVEL MITOCHONDRIA‐TARGETED DERIVATIVE OF APOCYNIN, INHIBITS NLRP3 INFLAMMASOME ACTIVATION IN PRIMARY MURINE MICROGLIA IN DOPAMINERGIC NEUROTOXIC MODELS

The FASEB journal, 2016-04, Vol.30 (S1) [Peer Reviewed Journal]

ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.30.1_supplement.1195.2

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16
Microglial Kv1.3, a voltage‐gated potassium channel, mediates neuroinflammation and neurodegeneration in cell culture and animal models of Parkinson's disease
Microglial Kv1.3, a voltage‐gated potassium channel, mediates neuroinflammation and neurodegeneration in cell culture and animal models of Parkinson's disease
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Article 		Add to My Research

Microglial Kv1.3, a voltage‐gated potassium channel, mediates neuroinflammation and neurodegeneration in cell culture and animal models of Parkinson's disease

The FASEB journal, 2017-04, Vol.31 (S1) [Peer Reviewed Journal]

ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.31.1_supplement.814.12

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17
Fyn kinase regulates microglial neuroinflammatory responses in cell culture and animal models of Parkinson’s disease (1055.7)
Fyn kinase regulates microglial neuroinflammatory responses in cell culture and animal models of Parkinson’s disease (1055.7)
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Fyn kinase regulates microglial neuroinflammatory responses in cell culture and animal models of Parkinson’s disease (1055.7)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a [Peer Reviewed Journal]

FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fasebj.28.1_supplement.1055.7

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