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Manganese-Induced Neurotoxicity: New Insights Into the Triad of Protein Misfolding, Mitochondrial Impairment, and Neuroinflammation
Manganese-Induced Neurotoxicity: New Insights Into the Triad of Protein Misfolding, Mitochondrial Impairment, and Neuroinflammation
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Manganese-Induced Neurotoxicity: New Insights Into the Triad of Protein Misfolding, Mitochondrial Impairment, and Neuroinflammation

Frontiers in neuroscience, 2019-06, Vol.13, p.654-654 [Peer Reviewed Journal]

2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2019 Harischandra, Ghaisas, Zenitsky, Jin, Kanthasamy, Anantharam and Kanthasamy. 2019 Harischandra, Ghaisas, Zenitsky, Jin, Kanthasamy, Anantharam and Kanthasamy ;ISSN: 1662-4548 ;ISSN: 1662-453X ;EISSN: 1662-453X ;DOI: 10.3389/fnins.2019.00654 ;PMID: 31293375

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Modified CAR T cells targeting membrane-proximal epitope of mesothelin enhances the antitumor function against large solid tumor
Modified CAR T cells targeting membrane-proximal epitope of mesothelin enhances the antitumor function against large solid tumor
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Modified CAR T cells targeting membrane-proximal epitope of mesothelin enhances the antitumor function against large solid tumor

Cell death & disease, 2019-06, Vol.10 (7), p.476-476, Article 476 [Peer Reviewed Journal]

The Author(s) 2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-019-1711-1 ;PMID: 31209210

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3
Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration
Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration
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Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration

Nature communications, 2016-10, Vol.7 (1), p.12932-18, Article 12932 [Peer Reviewed Journal]

Copyright Nature Publishing Group Oct 2016 ;Copyright © 2016, The Author(s) 2016 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms12932 ;PMID: 27703142

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4
Impact of Environmental Risk Factors on Mitochondrial Dysfunction, Neuroinflammation, Protein Misfolding, and Oxidative Stress in the Etiopathogenesis of Parkinson’s Disease
Impact of Environmental Risk Factors on Mitochondrial Dysfunction, Neuroinflammation, Protein Misfolding, and Oxidative Stress in the Etiopathogenesis of Parkinson’s Disease
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Impact of Environmental Risk Factors on Mitochondrial Dysfunction, Neuroinflammation, Protein Misfolding, and Oxidative Stress in the Etiopathogenesis of Parkinson’s Disease

International journal of molecular sciences, 2022-09, Vol.23 (18), p.10808 [Peer Reviewed Journal]

2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 by the authors. 2022 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms231810808 ;PMID: 36142718

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5
Mechanistic Insights Into Gut Microbiome Dysbiosis-Mediated Neuroimmune Dysregulation and Protein Misfolding and Clearance in the Pathogenesis of Chronic Neurodegenerative Disorders
Mechanistic Insights Into Gut Microbiome Dysbiosis-Mediated Neuroimmune Dysregulation and Protein Misfolding and Clearance in the Pathogenesis of Chronic Neurodegenerative Disorders
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Mechanistic Insights Into Gut Microbiome Dysbiosis-Mediated Neuroimmune Dysregulation and Protein Misfolding and Clearance in the Pathogenesis of Chronic Neurodegenerative Disorders

Frontiers in neuroscience, 2022-02, Vol.16, p.836605-836605 [Peer Reviewed Journal]

Copyright © 2022 Padhi, Worth, Zenitsky, Jin, Sambamurti, Anantharam, Kanthasamy and Kanthasamy. ;Copyright © 2022 Padhi, Worth, Zenitsky, Jin, Sambamurti, Anantharam, Kanthasamy and Kanthasamy. 2022 Padhi, Worth, Zenitsky, Jin, Sambamurti, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1662-4548 ;ISSN: 1662-453X ;EISSN: 1662-453X ;DOI: 10.3389/fnins.2022.836605 ;PMID: 35281490

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6
Microglial immune regulation by epigenetic reprogramming through histone H3K27 acetylation in neuroinflammation
Microglial immune regulation by epigenetic reprogramming through histone H3K27 acetylation in neuroinflammation
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Microglial immune regulation by epigenetic reprogramming through histone H3K27 acetylation in neuroinflammation

Frontiers in immunology, 2023-03, Vol.14, p.1052925-1052925 [Peer Reviewed Journal]

Copyright © 2023 Huang, Malovic, Ealy, Jin, Anantharam, Kanthasamy and Kanthasamy. ;Copyright © 2023 Huang, Malovic, Ealy, Jin, Anantharam, Kanthasamy and Kanthasamy 2023 Huang, Malovic, Ealy, Jin, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1664-3224 ;EISSN: 1664-3224 ;DOI: 10.3389/fimmu.2023.1052925 ;PMID: 37033967

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7
Mito-metformin protects against mitochondrial dysfunction and dopaminergic neuronal degeneration by activating upstream PKD1 signaling in cell culture and MitoPark animal models of Parkinson's disease
Mito-metformin protects against mitochondrial dysfunction and dopaminergic neuronal degeneration by activating upstream PKD1 signaling in cell culture and MitoPark animal models of Parkinson's disease
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Mito-metformin protects against mitochondrial dysfunction and dopaminergic neuronal degeneration by activating upstream PKD1 signaling in cell culture and MitoPark animal models of Parkinson's disease

Frontiers in neuroscience, 2024, Vol.18, p.1356703-1356703 [Peer Reviewed Journal]

Copyright © 2024 Ay, Charli, Langley, Jang, Padhi, Jin, Anantharam, Kalyanaraman, Kanthasamy and Kanthasamy. ;Copyright © 2024 Ay, Charli, Langley, Jang, Padhi, Jin, Anantharam, Kalyanaraman, Kanthasamy and Kanthasamy. 2024 Ay, Charli, Langley, Jang, Padhi, Jin, Anantharam, Kalyanaraman, Kanthasamy and Kanthasamy ;ISSN: 1662-4548 ;EISSN: 1662-453X ;DOI: 10.3389/fnins.2024.1356703 ;PMID: 38449738

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8
PKC Delta Activation Promotes Endoplasmic Reticulum Stress (ERS) and NLR Family Pyrin Domain-Containing 3 (NLRP3) Inflammasome Activation Subsequent to Asynuclein-Induced Microglial Activation: Involvement of Thioredoxin-Interacting Protein (TXNIP)/Thioredoxin (Trx) Redoxisome Pathway
PKC Delta Activation Promotes Endoplasmic Reticulum Stress (ERS) and NLR Family Pyrin Domain-Containing 3 (NLRP3) Inflammasome Activation Subsequent to Asynuclein-Induced Microglial Activation: Involvement of Thioredoxin-Interacting Protein (TXNIP)/Thioredoxin (Trx) Redoxisome Pathway
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PKC Delta Activation Promotes Endoplasmic Reticulum Stress (ERS) and NLR Family Pyrin Domain-Containing 3 (NLRP3) Inflammasome Activation Subsequent to Asynuclein-Induced Microglial Activation: Involvement of Thioredoxin-Interacting Protein (TXNIP)/Thioredoxin (Trx) Redoxisome Pathway

Frontiers in aging neuroscience, 2021-07, Vol.13, p.661505-661505 [Peer Reviewed Journal]

Copyright © 2021 Samidurai, Palanisamy, Bargues-Carot, Hepker, Kondru, Manne, Zenitsky, Jin, Anantharam, Kanthasamy and Kanthasamy. ;2021. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2021 Samidurai, Palanisamy, Bargues-Carot, Hepker, Kondru, Manne, Zenitsky, Jin, Anantharam, Kanthasamy and Kanthasamy. 2021 Samidurai, Palanisamy, Bargues-Carot, Hepker, Kondru, Manne, Zenitsky, Jin, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1663-4365 ;EISSN: 1663-4365 ;DOI: 10.3389/fnagi.2021.661505 ;PMID: 34276337

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9
Fyn Kinase-Mediated PKCδ Y311 Phosphorylation Induces Dopaminergic Degeneration in Cell Culture and Animal Models: Implications for the Identification of a New Pharmacological Target for Parkinson's Disease
Fyn Kinase-Mediated PKCδ Y311 Phosphorylation Induces Dopaminergic Degeneration in Cell Culture and Animal Models: Implications for the Identification of a New Pharmacological Target for Parkinson's Disease
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Fyn Kinase-Mediated PKCδ Y311 Phosphorylation Induces Dopaminergic Degeneration in Cell Culture and Animal Models: Implications for the Identification of a New Pharmacological Target for Parkinson's Disease

Frontiers in pharmacology, 2021-04, Vol.12, p.631375 [Peer Reviewed Journal]

Copyright © 2021 Saminathan, Ghosh, Zhang, Song, Jin, Anantharam, Kanthasamy and Kanthasamy. ;Copyright © 2021 Saminathan, Ghosh, Zhang, Song, Jin, Anantharam, Kanthasamy and Kanthasamy. 2021 Saminathan, Ghosh, Zhang, Song, Jin, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1663-9812 ;EISSN: 1663-9812 ;DOI: 10.3389/fphar.2021.631375 ;PMID: 33995031

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10
New protocol for kinetic assay seeding ability recovery "KASAR" from formalin-fixed paraffin-embedded tissues
New protocol for kinetic assay seeding ability recovery "KASAR" from formalin-fixed paraffin-embedded tissues
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New protocol for kinetic assay seeding ability recovery "KASAR" from formalin-fixed paraffin-embedded tissues

Frontiers in molecular biosciences, 2023-01, Vol.10, p.1087982-1087982 [Peer Reviewed Journal]

Copyright © 2023 Hepker, Clabaugh, Jin and Kanthasamy. ;Copyright © 2023 Hepker, Clabaugh, Jin and Kanthasamy. 2023 Hepker, Clabaugh, Jin and Kanthasamy ;ISSN: 2296-889X ;EISSN: 2296-889X ;DOI: 10.3389/fmolb.2023.1087982 ;PMID: 36793788

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11
Protein kinase D1 (PKD1) phosphorylation promotes dopaminergic neuronal survival during 6-OHDA-induced oxidative stress
Protein kinase D1 (PKD1) phosphorylation promotes dopaminergic neuronal survival during 6-OHDA-induced oxidative stress
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Protein kinase D1 (PKD1) phosphorylation promotes dopaminergic neuronal survival during 6-OHDA-induced oxidative stress

PloS one, 2014-05, Vol.9 (5), p.e96947 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Asaithambi et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Asaithambi et al 2014 Asaithambi et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0096947 ;PMID: 24806360

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12
Use of microRNA Let-7 to control the replication specificity of oncolytic adenovirus in hepatocellular carcinoma cells
Use of microRNA Let-7 to control the replication specificity of oncolytic adenovirus in hepatocellular carcinoma cells
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Use of microRNA Let-7 to control the replication specificity of oncolytic adenovirus in hepatocellular carcinoma cells

PloS one, 2011-07, Vol.6 (7), p.e21307-e21307 [Peer Reviewed Journal]

COPYRIGHT 2011 Public Library of Science ;COPYRIGHT 2011 Public Library of Science ;2011 Jin et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Jin et al. 2011 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0021307 ;PMID: 21814544

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13
Mitochondrial dysfunction-induced H3K27 hyperacetylation perturbs enhancers in Parkinson's disease
Mitochondrial dysfunction-induced H3K27 hyperacetylation perturbs enhancers in Parkinson's disease
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Mitochondrial dysfunction-induced H3K27 hyperacetylation perturbs enhancers in Parkinson's disease

JCI insight, 2021-09, Vol.6 (17) [Peer Reviewed Journal]

2021 Huang et al. 2021 Huang et al. ;ISSN: 2379-3708 ;EISSN: 2379-3708 ;DOI: 10.1172/jci.insight.138088 ;PMID: 34494552

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14
Correction: Modified CAR T cells targeting membrane-proximal epitope of mesothelin enhances the antitumor function against large solid tumor
Correction: Modified CAR T cells targeting membrane-proximal epitope of mesothelin enhances the antitumor function against large solid tumor
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Correction: Modified CAR T cells targeting membrane-proximal epitope of mesothelin enhances the antitumor function against large solid tumor

Cell death & disease, 2020-04, Vol.11 (4), p.235-235, Article 235 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-020-2450-z ;PMID: 32300103

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15
Eradicating tumor in a recurrent cervical cancer patient with autologous tumor-infiltrating lymphocytes and a modified lymphodepleting regimen
Eradicating tumor in a recurrent cervical cancer patient with autologous tumor-infiltrating lymphocytes and a modified lymphodepleting regimen
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Eradicating tumor in a recurrent cervical cancer patient with autologous tumor-infiltrating lymphocytes and a modified lymphodepleting regimen

Journal for immunotherapy of cancer, 2022-02, Vol.10 (2), p.e003887 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2022. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. ;2022 Author(s) (or their employer(s)) 2022. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. http://creativecommons.org/licenses/by-nc/4.0/ This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See http://creativecommons.org/licenses/by-nc/4.0/ . Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Author(s) (or their employer(s)) 2022. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. 2022 ;ISSN: 2051-1426 ;EISSN: 2051-1426 ;DOI: 10.1136/jitc-2021-003887 ;PMID: 35177415

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16
N-Acetyl Cysteine Protects against Methamphetamine-Induced Dopaminergic Neurodegeneration via Modulation of Redox Status and Autophagy in Dopaminergic Cells
N-Acetyl Cysteine Protects against Methamphetamine-Induced Dopaminergic Neurodegeneration via Modulation of Redox Status and Autophagy in Dopaminergic Cells
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N-Acetyl Cysteine Protects against Methamphetamine-Induced Dopaminergic Neurodegeneration via Modulation of Redox Status and Autophagy in Dopaminergic Cells

Parkinson's disease, 2012-01, Vol.2012, p.424285-11 [Peer Reviewed Journal]

Copyright © 2012 Prashanth Chandramani Shivalingappa et al. ;COPYRIGHT 2012 COPYRIGHT 2010 SAGE-Hindawi Access to Research ;COPYRIGHT 2012 Hindawi Limited ;Copyright © 2012 Prashanth Chandramani Shivalingappa et al. 2012 ;ISSN: 2042-0080 ;ISSN: 2090-8083 ;EISSN: 2042-0080 ;DOI: 10.1155/2012/424285 ;PMID: 23056996

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17
Tumor Necrosis Factor-Like Weak Inducer of Apoptosis (TWEAK) Enhances Activation of STAT3/NLRC4 Inflammasome Signaling Axis through PKCδ in Astrocytes: Implications for Parkinson's Disease
Tumor Necrosis Factor-Like Weak Inducer of Apoptosis (TWEAK) Enhances Activation of STAT3/NLRC4 Inflammasome Signaling Axis through PKCδ in Astrocytes: Implications for Parkinson's Disease
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Tumor Necrosis Factor-Like Weak Inducer of Apoptosis (TWEAK) Enhances Activation of STAT3/NLRC4 Inflammasome Signaling Axis through PKCδ in Astrocytes: Implications for Parkinson's Disease

Cells (Basel, Switzerland), 2020-08, Vol.9 (8), p.1831 [Peer Reviewed Journal]

2020. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells9081831 ;PMID: 32759670

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18
Hepcidin Plays a Key Role in 6-OHDA Induced Iron Overload and Apoptotic Cell Death in a Cell Culture Model of Parkinson's Disease
Hepcidin Plays a Key Role in 6-OHDA Induced Iron Overload and Apoptotic Cell Death in a Cell Culture Model of Parkinson's Disease
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Hepcidin Plays a Key Role in 6-OHDA Induced Iron Overload and Apoptotic Cell Death in a Cell Culture Model of Parkinson's Disease

Parkinson's disease, 2016-01, Vol.2016, p.8684130-7 [Peer Reviewed Journal]

Copyright © 2016 Qi Xu et al. ;COPYRIGHT 2016 COPYRIGHT 2010 SAGE-Hindawi Access to Research ;COPYRIGHT 2016 Hindawi Limited ;Copyright © 2016 Qi Xu et al. 2016 ;ISSN: 2042-0080 ;ISSN: 2090-8083 ;EISSN: 2042-0080 ;DOI: 10.1155/2016/8684130 ;PMID: 27298749

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19
Establishment of mouse teratocarcinomas stem cells line and screening genes responsible for malignancy
Establishment of mouse teratocarcinomas stem cells line and screening genes responsible for malignancy
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Establishment of mouse teratocarcinomas stem cells line and screening genes responsible for malignancy

PloS one, 2012-08, Vol.7 (8), p.e43955 [Peer Reviewed Journal]

COPYRIGHT 2012 Public Library of Science ;COPYRIGHT 2012 Public Library of Science ;Liu et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2012 Liu et al 2012 Liu et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0043955 ;PMID: 22952821

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20
Wnt/beta-catenin signaling in embryonic stem cell converted tumor cells
Wnt/beta-catenin signaling in embryonic stem cell converted tumor cells
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Wnt/beta-catenin signaling in embryonic stem cell converted tumor cells

Journal of translational medicine, 2012-09, Vol.10 (1), p.196-196, Article 196 [Peer Reviewed Journal]

COPYRIGHT 2012 BioMed Central Ltd. ;2012 Peng et al.; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Copyright ©2012 Peng et al.; licensee BioMed Central Ltd. 2012 Peng et al.; licensee BioMed Central Ltd. ;ISSN: 1479-5876 ;EISSN: 1479-5876 ;DOI: 10.1186/1479-5876-10-196 ;PMID: 22995718

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