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Epigallocatechin Gallate Has a Neurorescue Effect in a Mouse Model of Parkinson Disease
Epigallocatechin Gallate Has a Neurorescue Effect in a Mouse Model of Parkinson Disease
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Epigallocatechin Gallate Has a Neurorescue Effect in a Mouse Model of Parkinson Disease

The Journal of nutrition, 2017-10, Vol.147 (10), p.1926-1931 [Peer Reviewed Journal]

2017 American Society for Nutrition. ;Copyright American Institute of Nutrition Oct 1, 2017 ;2017 American Society for Nutrition 2017 ;ISSN: 0022-3166 ;EISSN: 1541-6100 ;DOI: 10.3945/jn.117.255034 ;PMID: 28835392

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Molecular mechanisms underlying protective effects of quercetin against mitochondrial dysfunction and progressive dopaminergic neurodegeneration in cell culture and MitoPark transgenic mouse models of Parkinson's Disease
Molecular mechanisms underlying protective effects of quercetin against mitochondrial dysfunction and progressive dopaminergic neurodegeneration in cell culture and MitoPark transgenic mouse models of Parkinson's Disease
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Molecular mechanisms underlying protective effects of quercetin against mitochondrial dysfunction and progressive dopaminergic neurodegeneration in cell culture and MitoPark transgenic mouse models of Parkinson's Disease

Journal of neurochemistry, 2017-06, Vol.141 (5), p.766-782 [Peer Reviewed Journal]

2017 International Society for Neurochemistry ;2017 International Society for Neurochemistry. ;Copyright © 2017 International Society for Neurochemistry ;ISSN: 0022-3042 ;EISSN: 1471-4159 ;DOI: 10.1111/jnc.14033 ;PMID: 28376279

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Manganese-Induced Neurotoxicity: New Insights Into the Triad of Protein Misfolding, Mitochondrial Impairment, and Neuroinflammation
Manganese-Induced Neurotoxicity: New Insights Into the Triad of Protein Misfolding, Mitochondrial Impairment, and Neuroinflammation
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Manganese-Induced Neurotoxicity: New Insights Into the Triad of Protein Misfolding, Mitochondrial Impairment, and Neuroinflammation

Frontiers in neuroscience, 2019-06, Vol.13, p.654-654 [Peer Reviewed Journal]

2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2019 Harischandra, Ghaisas, Zenitsky, Jin, Kanthasamy, Anantharam and Kanthasamy. 2019 Harischandra, Ghaisas, Zenitsky, Jin, Kanthasamy, Anantharam and Kanthasamy ;ISSN: 1662-4548 ;ISSN: 1662-453X ;EISSN: 1662-453X ;DOI: 10.3389/fnins.2019.00654 ;PMID: 31293375

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4
Organophosphate pesticide chlorpyrifos impairs STAT1 signaling to induce dopaminergic neurotoxicity: Implications for mitochondria mediated oxidative stress signaling events
Organophosphate pesticide chlorpyrifos impairs STAT1 signaling to induce dopaminergic neurotoxicity: Implications for mitochondria mediated oxidative stress signaling events
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Organophosphate pesticide chlorpyrifos impairs STAT1 signaling to induce dopaminergic neurotoxicity: Implications for mitochondria mediated oxidative stress signaling events

Neurobiology of disease, 2018-09, Vol.117, p.82-113 [Peer Reviewed Journal]

EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2018.05.019

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5
Mechanistic Interplay Between Autophagy and Apoptotic Signaling in Endosulfan-Induced Dopaminergic Neurotoxicity: Relevance to the Adverse Outcome Pathway in Pesticide Neurotoxicity
Mechanistic Interplay Between Autophagy and Apoptotic Signaling in Endosulfan-Induced Dopaminergic Neurotoxicity: Relevance to the Adverse Outcome Pathway in Pesticide Neurotoxicity
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Mechanistic Interplay Between Autophagy and Apoptotic Signaling in Endosulfan-Induced Dopaminergic Neurotoxicity: Relevance to the Adverse Outcome Pathway in Pesticide Neurotoxicity

Toxicological sciences, 2019-06, Vol.169 (2), p.333-352 [Peer Reviewed Journal]

The Author(s) 2019. Published by Oxford University Press on behalf of the Society of Toxicology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2019 ;The Author(s) 2019. Published by Oxford University Press on behalf of the Society of Toxicology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com. ;ISSN: 1096-6080 ;EISSN: 1096-0929 ;DOI: 10.1093/toxsci/kfz049 ;PMID: 30796443

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6
Mito-Apocynin Prevents Mitochondrial Dysfunction, Microglial Activation, Oxidative Damage, and Progressive Neurodegeneration in MitoPark Transgenic Mice
Mito-Apocynin Prevents Mitochondrial Dysfunction, Microglial Activation, Oxidative Damage, and Progressive Neurodegeneration in MitoPark Transgenic Mice
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Mito-Apocynin Prevents Mitochondrial Dysfunction, Microglial Activation, Oxidative Damage, and Progressive Neurodegeneration in MitoPark Transgenic Mice

Antioxidants & redox signaling, 2017-11, Vol.27 (14), p.1048-1066 [Peer Reviewed Journal]

(©) Copyright 2017, Mary Ann Liebert, Inc. ;Copyright 2017, Mary Ann Liebert, Inc. 2017 ;ISSN: 1523-0864 ;EISSN: 1557-7716 ;DOI: 10.1089/ars.2016.6905 ;PMID: 28375739

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7
Role of the Fyn-PKCδ signaling in SE-induced neuroinflammation and epileptogenesis in experimental models of temporal lobe epilepsy
Role of the Fyn-PKCδ signaling in SE-induced neuroinflammation and epileptogenesis in experimental models of temporal lobe epilepsy
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Role of the Fyn-PKCδ signaling in SE-induced neuroinflammation and epileptogenesis in experimental models of temporal lobe epilepsy

Neurobiology of disease, 2018-02, Vol.110, p.102-121 [Peer Reviewed Journal]

EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2017.11.008

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8
Status Epilepticus: Behavioral and Electroencephalography Seizure Correlates in Kainate Experimental Models
Status Epilepticus: Behavioral and Electroencephalography Seizure Correlates in Kainate Experimental Models
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Status Epilepticus: Behavioral and Electroencephalography Seizure Correlates in Kainate Experimental Models

Frontiers in neurology, 2018-01, Vol.9, p.7-7 [Peer Reviewed Journal]

COPYRIGHT 2018 Frontiers Research Foundation ;Copyright © 2018 Sharma, Puttachary, Thippeswamy, Kanthasamy and Thippeswamy. 2018 Sharma, Puttachary, Thippeswamy, Kanthasamy and Thippeswamy ;ISSN: 1664-2295 ;EISSN: 1664-2295 ;DOI: 10.3389/fneur.2018.00007 ;PMID: 29410648

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9
Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration
Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration
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Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration

Nature communications, 2016-10, Vol.7 (1), p.12932-18, Article 12932 [Peer Reviewed Journal]

Copyright Nature Publishing Group Oct 2016 ;Copyright © 2016, The Author(s) 2016 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms12932 ;PMID: 27703142

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10
Impact of Environmental Risk Factors on Mitochondrial Dysfunction, Neuroinflammation, Protein Misfolding, and Oxidative Stress in the Etiopathogenesis of Parkinson’s Disease
Impact of Environmental Risk Factors on Mitochondrial Dysfunction, Neuroinflammation, Protein Misfolding, and Oxidative Stress in the Etiopathogenesis of Parkinson’s Disease
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Impact of Environmental Risk Factors on Mitochondrial Dysfunction, Neuroinflammation, Protein Misfolding, and Oxidative Stress in the Etiopathogenesis of Parkinson’s Disease

International journal of molecular sciences, 2022-09, Vol.23 (18), p.10808 [Peer Reviewed Journal]

2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 by the authors. 2022 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms231810808 ;PMID: 36142718

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11
Mechanistic Insights Into Gut Microbiome Dysbiosis-Mediated Neuroimmune Dysregulation and Protein Misfolding and Clearance in the Pathogenesis of Chronic Neurodegenerative Disorders
Mechanistic Insights Into Gut Microbiome Dysbiosis-Mediated Neuroimmune Dysregulation and Protein Misfolding and Clearance in the Pathogenesis of Chronic Neurodegenerative Disorders
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Mechanistic Insights Into Gut Microbiome Dysbiosis-Mediated Neuroimmune Dysregulation and Protein Misfolding and Clearance in the Pathogenesis of Chronic Neurodegenerative Disorders

Frontiers in neuroscience, 2022-02, Vol.16, p.836605-836605 [Peer Reviewed Journal]

Copyright © 2022 Padhi, Worth, Zenitsky, Jin, Sambamurti, Anantharam, Kanthasamy and Kanthasamy. ;Copyright © 2022 Padhi, Worth, Zenitsky, Jin, Sambamurti, Anantharam, Kanthasamy and Kanthasamy. 2022 Padhi, Worth, Zenitsky, Jin, Sambamurti, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1662-4548 ;ISSN: 1662-453X ;EISSN: 1662-453X ;DOI: 10.3389/fnins.2022.836605 ;PMID: 35281490

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12
Accelerated accumulation of retinal α-synuclein (pSer129) and tau, neuroinflammation, and autophagic dysregulation in a seeded mouse model of Parkinson's disease
Accelerated accumulation of retinal α-synuclein (pSer129) and tau, neuroinflammation, and autophagic dysregulation in a seeded mouse model of Parkinson's disease
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Accelerated accumulation of retinal α-synuclein (pSer129) and tau, neuroinflammation, and autophagic dysregulation in a seeded mouse model of Parkinson's disease

Neurobiology of disease, 2019-01, Vol.121, p.1-16 [Peer Reviewed Journal]

Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0969-9961 ;EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2018.09.013

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13
Microglial immune regulation by epigenetic reprogramming through histone H3K27 acetylation in neuroinflammation
Microglial immune regulation by epigenetic reprogramming through histone H3K27 acetylation in neuroinflammation
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Microglial immune regulation by epigenetic reprogramming through histone H3K27 acetylation in neuroinflammation

Frontiers in immunology, 2023-03, Vol.14, p.1052925-1052925 [Peer Reviewed Journal]

Copyright © 2023 Huang, Malovic, Ealy, Jin, Anantharam, Kanthasamy and Kanthasamy. ;Copyright © 2023 Huang, Malovic, Ealy, Jin, Anantharam, Kanthasamy and Kanthasamy 2023 Huang, Malovic, Ealy, Jin, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1664-3224 ;EISSN: 1664-3224 ;DOI: 10.3389/fimmu.2023.1052925 ;PMID: 37033967

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14
Molecular Signatures of Neuroinflammation Induced by αSynuclein Aggregates in Microglial Cells
Molecular Signatures of Neuroinflammation Induced by αSynuclein Aggregates in Microglial Cells
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Molecular Signatures of Neuroinflammation Induced by αSynuclein Aggregates in Microglial Cells

Frontiers in immunology, 2020-01, Vol.11, p.33 [Peer Reviewed Journal]

Copyright © 2020 Sarkar, Dammer, Malovic, Olsen, Raza, Gao, Xiao, Oliver, Duong, Joers, Seyfried, Huang, Kukar, Tansey, Kanthasamy and Rangaraju. ;Copyright © 2020 Sarkar, Dammer, Malovic, Olsen, Raza, Gao, Xiao, Oliver, Duong, Joers, Seyfried, Huang, Kukar, Tansey, Kanthasamy and Rangaraju. 2020 Sarkar, Dammer, Malovic, Olsen, Raza, Gao, Xiao, Oliver, Duong, Joers, Seyfried, Huang, Kukar, Tansey, Kanthasamy and Rangaraju ;ISSN: 1664-3224 ;EISSN: 1664-3224 ;DOI: 10.3389/fimmu.2020.00033 ;PMID: 32082315

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15
Protein kinase D1 (PKD1) phosphorylation promotes dopaminergic neuronal survival during 6-OHDA-induced oxidative stress
Protein kinase D1 (PKD1) phosphorylation promotes dopaminergic neuronal survival during 6-OHDA-induced oxidative stress
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Protein kinase D1 (PKD1) phosphorylation promotes dopaminergic neuronal survival during 6-OHDA-induced oxidative stress

PloS one, 2014-05, Vol.9 (5), p.e96947 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Asaithambi et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Asaithambi et al 2014 Asaithambi et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0096947 ;PMID: 24806360

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16
Proteolytic activation of proapoptotic kinase protein kinase Cδ by tumor necrosis factor α death receptor signaling in dopaminergic neurons during neuroinflammation
Proteolytic activation of proapoptotic kinase protein kinase Cδ by tumor necrosis factor α death receptor signaling in dopaminergic neurons during neuroinflammation
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Proteolytic activation of proapoptotic kinase protein kinase Cδ by tumor necrosis factor α death receptor signaling in dopaminergic neurons during neuroinflammation

Journal of neuroinflammation, 2012-04, Vol.9 (1), p.82-82, Article 82 [Peer Reviewed Journal]

Copyright ©2012 Gordon et al.; licensee BioMed Central Ltd. 2012 Gordon et al.; licensee BioMed Central Ltd. ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/1742-2094-9-82 ;PMID: 22540228

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17
Mito-metformin protects against mitochondrial dysfunction and dopaminergic neuronal degeneration by activating upstream PKD1 signaling in cell culture and MitoPark animal models of Parkinson's disease
Mito-metformin protects against mitochondrial dysfunction and dopaminergic neuronal degeneration by activating upstream PKD1 signaling in cell culture and MitoPark animal models of Parkinson's disease
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Mito-metformin protects against mitochondrial dysfunction and dopaminergic neuronal degeneration by activating upstream PKD1 signaling in cell culture and MitoPark animal models of Parkinson's disease

Frontiers in neuroscience, 2024, Vol.18, p.1356703-1356703 [Peer Reviewed Journal]

Copyright © 2024 Ay, Charli, Langley, Jang, Padhi, Jin, Anantharam, Kalyanaraman, Kanthasamy and Kanthasamy. ;Copyright © 2024 Ay, Charli, Langley, Jang, Padhi, Jin, Anantharam, Kalyanaraman, Kanthasamy and Kanthasamy. 2024 Ay, Charli, Langley, Jang, Padhi, Jin, Anantharam, Kalyanaraman, Kanthasamy and Kanthasamy ;ISSN: 1662-4548 ;EISSN: 1662-453X ;DOI: 10.3389/fnins.2024.1356703 ;PMID: 38449738

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18
Anti-inflammatory and neuroprotective effects of an orally active apocynin derivative in pre-clinical models of Parkinson's disease
Anti-inflammatory and neuroprotective effects of an orally active apocynin derivative in pre-clinical models of Parkinson's disease
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Anti-inflammatory and neuroprotective effects of an orally active apocynin derivative in pre-clinical models of Parkinson's disease

Journal of neuroinflammation, 2012-10, Vol.9 (1), p.241-241, Article 241 [Peer Reviewed Journal]

COPYRIGHT 2012 BioMed Central Ltd. ;2012 Ghosh et al.; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Copyright ©2012 Ghosh et al.; licensee BioMed Central Ltd. 2012 Ghosh et al.; licensee BioMed Central Ltd. ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/1742-2094-9-241 ;PMID: 23092448

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19
PKC Delta Activation Promotes Endoplasmic Reticulum Stress (ERS) and NLR Family Pyrin Domain-Containing 3 (NLRP3) Inflammasome Activation Subsequent to Asynuclein-Induced Microglial Activation: Involvement of Thioredoxin-Interacting Protein (TXNIP)/Thioredoxin (Trx) Redoxisome Pathway
PKC Delta Activation Promotes Endoplasmic Reticulum Stress (ERS) and NLR Family Pyrin Domain-Containing 3 (NLRP3) Inflammasome Activation Subsequent to Asynuclein-Induced Microglial Activation: Involvement of Thioredoxin-Interacting Protein (TXNIP)/Thioredoxin (Trx) Redoxisome Pathway
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PKC Delta Activation Promotes Endoplasmic Reticulum Stress (ERS) and NLR Family Pyrin Domain-Containing 3 (NLRP3) Inflammasome Activation Subsequent to Asynuclein-Induced Microglial Activation: Involvement of Thioredoxin-Interacting Protein (TXNIP)/Thioredoxin (Trx) Redoxisome Pathway

Frontiers in aging neuroscience, 2021-07, Vol.13, p.661505-661505 [Peer Reviewed Journal]

Copyright © 2021 Samidurai, Palanisamy, Bargues-Carot, Hepker, Kondru, Manne, Zenitsky, Jin, Anantharam, Kanthasamy and Kanthasamy. ;2021. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2021 Samidurai, Palanisamy, Bargues-Carot, Hepker, Kondru, Manne, Zenitsky, Jin, Anantharam, Kanthasamy and Kanthasamy. 2021 Samidurai, Palanisamy, Bargues-Carot, Hepker, Kondru, Manne, Zenitsky, Jin, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1663-4365 ;EISSN: 1663-4365 ;DOI: 10.3389/fnagi.2021.661505 ;PMID: 34276337

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20
Fyn Kinase-Mediated PKCδ Y311 Phosphorylation Induces Dopaminergic Degeneration in Cell Culture and Animal Models: Implications for the Identification of a New Pharmacological Target for Parkinson's Disease
Fyn Kinase-Mediated PKCδ Y311 Phosphorylation Induces Dopaminergic Degeneration in Cell Culture and Animal Models: Implications for the Identification of a New Pharmacological Target for Parkinson's Disease
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Fyn Kinase-Mediated PKCδ Y311 Phosphorylation Induces Dopaminergic Degeneration in Cell Culture and Animal Models: Implications for the Identification of a New Pharmacological Target for Parkinson's Disease

Frontiers in pharmacology, 2021-04, Vol.12, p.631375 [Peer Reviewed Journal]

Copyright © 2021 Saminathan, Ghosh, Zhang, Song, Jin, Anantharam, Kanthasamy and Kanthasamy. ;Copyright © 2021 Saminathan, Ghosh, Zhang, Song, Jin, Anantharam, Kanthasamy and Kanthasamy. 2021 Saminathan, Ghosh, Zhang, Song, Jin, Anantharam, Kanthasamy and Kanthasamy ;ISSN: 1663-9812 ;EISSN: 1663-9812 ;DOI: 10.3389/fphar.2021.631375 ;PMID: 33995031

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