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1
Speckle tracking echocardiographic deformation indices in Chagas and idiopathic dilated cardiomyopathy: Incremental prognostic value of longitudinal strain
Speckle tracking echocardiographic deformation indices in Chagas and idiopathic dilated cardiomyopathy: Incremental prognostic value of longitudinal strain
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Speckle tracking echocardiographic deformation indices in Chagas and idiopathic dilated cardiomyopathy: Incremental prognostic value of longitudinal strain

PloS one, 2019-08, Vol.14 (8), p.e0221028-e0221028 [Peer Reviewed Journal]

COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Santos Junior et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Santos Junior et al 2019 Santos Junior et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0221028 ;PMID: 31437176

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2
The administration of high-mobility group box 1 fragment prevents deterioration of cardiac performance by enhancement of bone marrow mesenchymal stem cell homing in the delta-sarcoglycan-deficient hamster
The administration of high-mobility group box 1 fragment prevents deterioration of cardiac performance by enhancement of bone marrow mesenchymal stem cell homing in the delta-sarcoglycan-deficient hamster
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The administration of high-mobility group box 1 fragment prevents deterioration of cardiac performance by enhancement of bone marrow mesenchymal stem cell homing in the delta-sarcoglycan-deficient hamster

PloS one, 2018-12, Vol.13 (12), p.e0202838-e0202838 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Kido et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Kido et al 2018 Kido et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0202838 ;PMID: 30517097

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3
Implications of Underlying Mechanisms for the Recognition and Management of Diabetic Cardiomyopathy
Implications of Underlying Mechanisms for the Recognition and Management of Diabetic Cardiomyopathy
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Implications of Underlying Mechanisms for the Recognition and Management of Diabetic Cardiomyopathy

Journal of the American College of Cardiology, 2018-01, Vol.71 (3), p.339-351 [Peer Reviewed Journal]

2018 American College of Cardiology Foundation ;Copyright © 2018 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited Jan 23, 2018 ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2017.11.019 ;PMID: 29348027

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4
Inherited cardiomyopathies
Inherited cardiomyopathies
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Inherited cardiomyopathies

BMJ, 2011-11, Vol.343 (7833), p.1106-1110 [Peer Reviewed Journal]

BMJ Publishing Group Ltd 2011 ;Copyright: 2011 © BMJ Publishing Group Ltd 2011 ;ISSN: 0959-8138 ;ISSN: 0959-8146 ;EISSN: 1468-5833 ;EISSN: 1756-1833 ;DOI: 10.1136/bmj.d6966 ;PMID: 22106372 ;CODEN: BMJOAE

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5
Diagnosis and management of the cardiac amyloidoses
Diagnosis and management of the cardiac amyloidoses
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Diagnosis and management of the cardiac amyloidoses

Circulation (New York, N.Y.), 2005-09, Vol.112 (13), p.2047-2060 [Peer Reviewed Journal]

2005 INIST-CNRS ;ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/CIRCULATIONAHA.104.489187 ;PMID: 16186440 ;CODEN: CIRCAZ

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6
Application of left ventricular endomyocardial biopsy in the diagnosis of mitochondrial cardiomyopathy: a case report
Application of left ventricular endomyocardial biopsy in the diagnosis of mitochondrial cardiomyopathy: a case report
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Application of left ventricular endomyocardial biopsy in the diagnosis of mitochondrial cardiomyopathy: a case report

BMC cardiovascular disorders, 2023-07, Vol.23 (1), p.338-338, Article 338 [Peer Reviewed Journal]

2023. The Author(s). ;COPYRIGHT 2023 BioMed Central Ltd. ;2023. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 1471-2261 ;EISSN: 1471-2261 ;DOI: 10.1186/s12872-023-03373-x ;PMID: 37403033

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7
Left ventricular non-compaction cardiomyopathy
Left ventricular non-compaction cardiomyopathy
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Left ventricular non-compaction cardiomyopathy

The Lancet (British edition), 2015-08, Vol.386 (9995), p.813-825 [Peer Reviewed Journal]

Elsevier Ltd ;2015 Elsevier Ltd ;Copyright © 2015 Elsevier Ltd. All rights reserved. ;Copyright Elsevier Limited Aug 22, 2015 ;ISSN: 0140-6736 ;EISSN: 1474-547X ;DOI: 10.1016/S0140-6736(14)61282-4 ;PMID: 25865865

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8
Ivabradine in Children With Dilated Cardiomyopathy and Symptomatic Chronic Heart Failure
Ivabradine in Children With Dilated Cardiomyopathy and Symptomatic Chronic Heart Failure
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Ivabradine in Children With Dilated Cardiomyopathy and Symptomatic Chronic Heart Failure

Journal of the American College of Cardiology, 2017-09, Vol.70 (10), p.1262-1272 [Peer Reviewed Journal]

2017 American College of Cardiology Foundation ;Copyright © 2017 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited Sep 5, 2017 ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2017.07.725 ;PMID: 28859790

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9
Cardiomyocyte-specific miRNA-30c over-expression causes dilated cardiomyopathy
Cardiomyocyte-specific miRNA-30c over-expression causes dilated cardiomyopathy
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Cardiomyocyte-specific miRNA-30c over-expression causes dilated cardiomyopathy

PloS one, 2014-05, Vol.9 (5), p.e96290-e96290 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Wijnen et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Wijnen et al 2014 Wijnen et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0096290 ;PMID: 24789369

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10
MiR-21 protected against diabetic cardiomyopathy induced diastolic dysfunction by targeting gelsolin
MiR-21 protected against diabetic cardiomyopathy induced diastolic dysfunction by targeting gelsolin
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MiR-21 protected against diabetic cardiomyopathy induced diastolic dysfunction by targeting gelsolin

Cardiovascular diabetology, 2018-09, Vol.17 (1), p.123-123, Article 123 [Peer Reviewed Journal]

Copyright © 2018. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 1475-2840 ;EISSN: 1475-2840 ;DOI: 10.1186/s12933-018-0767-z ;PMID: 30180843

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11
Left Atrial Mechanical Function and Global Strain in Hypertrophic Cardiomyopathy
Left Atrial Mechanical Function and Global Strain in Hypertrophic Cardiomyopathy
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Left Atrial Mechanical Function and Global Strain in Hypertrophic Cardiomyopathy

PloS one, 2016-06, Vol.11 (6), p.e0157433-e0157433 [Peer Reviewed Journal]

COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Kim et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2016 Kim et al 2016 Kim et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0157433 ;PMID: 27336444

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12
Loss-of-function mutations in co-chaperone BAG3 destabilize small HSPs and cause cardiomyopathy
Loss-of-function mutations in co-chaperone BAG3 destabilize small HSPs and cause cardiomyopathy
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Loss-of-function mutations in co-chaperone BAG3 destabilize small HSPs and cause cardiomyopathy

The Journal of clinical investigation, 2017-08, Vol.127 (8), p.3189-3200 [Peer Reviewed Journal]

COPYRIGHT 2017 American Society for Clinical Investigation ;COPYRIGHT 2017 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Aug 2017 ;Copyright © 2017, American Society for Clinical Investigation 2017 American Society for Clinical Investigation ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/jci94310 ;PMID: 28737513

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13
Familial Dilated Cardiomyopathy Caused by a Novel Frameshift in the BAG3 Gene
Familial Dilated Cardiomyopathy Caused by a Novel Frameshift in the BAG3 Gene
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Familial Dilated Cardiomyopathy Caused by a Novel Frameshift in the BAG3 Gene

PloS one, 2016-07, Vol.11 (7), p.e0158730-e0158730 [Peer Reviewed Journal]

COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Toro et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Attribution 4.0 Spain info:eu-repo/semantics/openAccess http://creativecommons.org/licenses/by/4.0/es/ ;2016 Toro et al 2016 Toro et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0158730 ;PMID: 27391596

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14
Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy
Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy
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Circulating microRNA signature for the diagnosis of childhood dilated cardiomyopathy

Scientific reports, 2018-01, Vol.8 (1), p.724-9, Article 724 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-017-19138-4 ;PMID: 29335596

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15
The clinical features, outcomes and genetic characteristics of hypertrophic cardiomyopathy patients with severe right ventricular hypertrophy
The clinical features, outcomes and genetic characteristics of hypertrophic cardiomyopathy patients with severe right ventricular hypertrophy
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The clinical features, outcomes and genetic characteristics of hypertrophic cardiomyopathy patients with severe right ventricular hypertrophy

PloS one, 2017-03, Vol.12 (3), p.e0174118-e0174118 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Guo et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Guo et al 2017 Guo et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0174118 ;PMID: 28323875

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16
Cardiac amyloidosis: An update on pathophysiology, diagnosis, and treatment
Cardiac amyloidosis: An update on pathophysiology, diagnosis, and treatment
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Cardiac amyloidosis: An update on pathophysiology, diagnosis, and treatment

Trends in cardiovascular medicine, 2018-01, Vol.28 (1), p.10 [Peer Reviewed Journal]

Copyright © 2018 Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited 2018 ;ISSN: 1050-1738 ;EISSN: 1873-2615 ;DOI: 10.1016/j.tcm.2017.07.004 ;PMID: 28739313

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17
Prognostic implications of late gadolinium enhancement at the right ventricular insertion point in patients with non-ischemic dilated cardiomyopathy: A multicenter retrospective cohort study
Prognostic implications of late gadolinium enhancement at the right ventricular insertion point in patients with non-ischemic dilated cardiomyopathy: A multicenter retrospective cohort study
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Prognostic implications of late gadolinium enhancement at the right ventricular insertion point in patients with non-ischemic dilated cardiomyopathy: A multicenter retrospective cohort study

PloS one, 2018-11, Vol.13 (11), p.e0208100-e0208100 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Yi et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Yi et al 2018 Yi et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0208100 ;PMID: 30485353

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18
Implications of multiple late gadolinium enhancement lesions on the frequency of left ventricular reverse remodeling and prognosis in patients with non-ischemic cardiomyopathy
Implications of multiple late gadolinium enhancement lesions on the frequency of left ventricular reverse remodeling and prognosis in patients with non-ischemic cardiomyopathy
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Implications of multiple late gadolinium enhancement lesions on the frequency of left ventricular reverse remodeling and prognosis in patients with non-ischemic cardiomyopathy

Journal of cardiovascular magnetic resonance, 2021-03, Vol.23 (1), p.32-32, Article 32 [Peer Reviewed Journal]

COPYRIGHT 2021 BioMed Central Ltd. ;COPYRIGHT 2021 BioMed Central Ltd. ;2021. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 1532-429X ;ISSN: 1097-6647 ;EISSN: 1532-429X ;DOI: 10.1186/s12968-021-00734-3 ;PMID: 33761955

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19
The Role of Cardiac Fibrosis in Diabetic Cardiomyopathy: From Pathophysiology to Clinical Diagnostic Tools
The Role of Cardiac Fibrosis in Diabetic Cardiomyopathy: From Pathophysiology to Clinical Diagnostic Tools
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The Role of Cardiac Fibrosis in Diabetic Cardiomyopathy: From Pathophysiology to Clinical Diagnostic Tools

International journal of molecular sciences, 2023-05, Vol.24 (10), p.8604 [Peer Reviewed Journal]

COPYRIGHT 2023 MDPI AG ;2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms24108604 ;PMID: 37239956

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20
A New Mouse Model of Chronic Myocarditis Induced by Recombinant Bacille Calmette-Guèrin Expressing a T-Cell Epitope of Cardiac Myosin Heavy Chain-α
A New Mouse Model of Chronic Myocarditis Induced by Recombinant Bacille Calmette-Guèrin Expressing a T-Cell Epitope of Cardiac Myosin Heavy Chain-α
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A New Mouse Model of Chronic Myocarditis Induced by Recombinant Bacille Calmette-Guèrin Expressing a T-Cell Epitope of Cardiac Myosin Heavy Chain-α

International journal of molecular sciences, 2021-01, Vol.22 (2), p.794 [Peer Reviewed Journal]

2021. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms22020794 ;PMID: 33466825

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