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Arrhythmia-Induced Cardiomyopathies: Mechanisms, Recognition, and Management

Journal of the American College of Cardiology, 2015-10, Vol.66 (15), p.1714-1728 [Peer Reviewed Journal]

Copyright © 2015 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2015.08.038 ;PMID: 26449143

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  • Title:
    Arrhythmia-Induced Cardiomyopathies: Mechanisms, Recognition, and Management
  • Author: Gopinathannair, Rakesh ; Etheridge, Susan P ; Marchlinski, Francis E ; Spinale, Francis G ; Lakkireddy, Dhanunjaya ; Olshansky, Brian
  • Subjects: Arrhythmias, Cardiac - complications ; Arrhythmias, Cardiac - therapy ; Cardiomyopathies - diagnosis ; Cardiomyopathies - therapy ; Diagnostic Techniques, Cardiovascular ; Disease Management ; Humans
  • Is Part Of: Journal of the American College of Cardiology, 2015-10, Vol.66 (15), p.1714-1728
  • Description: Arrhythmia-induced cardiomyopathy (AIC) is a potentially reversible condition in which left ventricular dysfunction is induced or mediated by atrial or ventricular arrhythmias. Cellular and extracellular changes in response to the culprit arrhythmia have been identified, but specific pathophysiological mechanisms remain unclear. Early recognition of AIC and prompt treatment of the culprit arrhythmia using pharmacological or ablative techniques result in symptom resolution and recovery of ventricular function. Although cardiomyopathy in response to an arrhythmia may take months to years to develop, recurrent arrhythmia can result in rapid decline in ventricular function with development of heart failure, suggesting residual ultrastructural abnormalities. Reports of sudden death in patients with normalized left ventricular ejection fraction cast doubt on the complete reversibility of this condition. Several aspects of AIC, including specific pathophysiological mechanisms, predisposing factors, optimal therapeutic strategies to prevent ultrastructural changes, and long-term risk of sudden death remain unresolved and need further research.
  • Publisher: United States
  • Language: English
  • Identifier: ISSN: 0735-1097
    EISSN: 1558-3597
    DOI: 10.1016/j.jacc.2015.08.038
    PMID: 26449143
  • Source: GFMER Free Medical Journals
    MEDLINE
    Alma/SFX Local Collection
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