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HIV-1 accessory proteins--ensuring viral survival in a hostile environment
Cell host & microbe, 2008-06, Vol.3 (6), p.388-398
[Peer Reviewed Journal]
ISSN: 1931-3128 ;EISSN: 1934-6069 ;DOI: 10.1016/j.chom.2008.04.008 ;PMID: 18541215
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Title:
HIV-1 accessory proteins--ensuring viral survival in a hostile environment
Author:
Malim, Michael H
;
Emerman, Michael
Subjects:
Animals
;
HIV Infections - immunology
;
HIV Infections - virology
;
HIV-1 - genetics
;
HIV-1 - physiology
;
Host-Pathogen Interactions
;
Human immunodeficiency virus 1
;
Human Immunodeficiency Virus Proteins - genetics
;
Human Immunodeficiency Virus Proteins - metabolism
;
Humans
;
nef Gene Products, Human Immunodeficiency Virus - genetics
;
nef Gene Products, Human Immunodeficiency Virus - metabolism
;
Primates
;
Retrovirus
;
Simian immunodeficiency virus
;
vif Gene Products, Human Immunodeficiency Virus - genetics
;
vif Gene Products, Human Immunodeficiency Virus - metabolism
;
Viral Regulatory and Accessory Proteins - genetics
;
Viral Regulatory and Accessory Proteins - metabolism
;
vpr Gene Products, Human Immunodeficiency Virus - genetics
;
vpr Gene Products, Human Immunodeficiency Virus - metabolism
Is Part Of:
Cell host & microbe, 2008-06, Vol.3 (6), p.388-398
Description:
One of the features of primate immunodeficiency viruses (HIVs and SIVs) that distinguishes them from other retroviruses is the array of "accessory" proteins they encode. Here, we discuss recent advances in understanding the interactions of the HIV-1 Nef, Vif, Vpu, and Vpr proteins with factors and pathways expressed in cells of the immune system. In at least three instances, the principal activity of the accessory proteins appears to be evasion from various forms of cell-mediated (or intrinsic), antiviral resistance. Broadly speaking, the HIV-1 accessory proteins modify the local environment within infected cells to ensure viral persistence, replication, dissemination, and transmission.
Publisher:
United States
Language:
English
Identifier:
ISSN: 1931-3128
EISSN: 1934-6069
DOI: 10.1016/j.chom.2008.04.008
PMID: 18541215
Source:
MEDLINE
Cell Press Archives (Open access)
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